CADASIL: a critical look at a Notch disease. - Wikidata - wikimedia - TriplyDB

CADASIL: a critical look at a Notch disease.

CADASIL: a critical look at a Notch disease.

http://www.wikidata.org/entity/Q36409731

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Endothelial cells downregulate apolipoprotein D expression in mural cells through paracrine secretion and Notch signaling The canonical Notch signaling pathway: unfolding the activation mechanism.A novel mechanism for sevoflurane preconditioning-induced neuroprotection.Direct observation of proteolytic cleavage at the S2 site upon forced unfolding of the Notch negative regulatory region Mutational analysis of the Notch2 negative regulatory region identifies key structural elements for mechanical stability.Where will the next generation of stroke treatments come from?Hypomorphic Notch 3 alleles link Notch signaling to ischemic cerebral small-vessel disease.The miR-143/145 cluster is a novel transcriptional target of Jagged-1/Notch signaling in vascular smooth muscle cells.O-glucose trisaccharide is present at high but variable stoichiometry at multiple sites on mouse Notch1.Notch: from neural development to neurological disorders.Notch signalling in vertebrate neural development.Linking Notch signaling to ischemic stroke Is the amyloid hypothesis of Alzheimer's disease therapeutically relevant?ADAM proteases: ligand processing and modulation of the Notch pathway A Notch updated.CADASIL: Ultrastructural insights into the morphology of granular osmiophilic material.Notch regulation of tumor angiogenesis.Dll4-Notch signaling in regulation of tumor angiogenesis.Notch signaling in cerebrovascular diseases (Review)Notch signaling in cardiovascular disease and calcification.Effects of Linker Length and Transient Secondary Structure Elements in the Intrinsically Disordered Notch RAM Region on Notch Signaling.Notch Signaling in Endothelial Cells: Is It the Therapeutic Target for Vascular Neointimal Hyperplasia?BMPs and FGFs target Notch signalling via jagged 2 to regulate tooth morphogenesis and cytodifferentiation.Identification of Domains for Efficient Notch Signaling Activity in Immobilized Notch Ligand Proteins.Antibodies against the extracellular domain of human Notch1 receptor reveal the critical role of epidermal-growth-factor-like repeats 25-26 in ligand binding and receptor activation.Neuroprotection of Botch in experimental intracerebral hemorrhage in rats.

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CADASIL: a critical look at a Notch disease.

http://www.wikidata.org/entity/Q36409731

description

2006 nî lūn-bûn

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2006年の論文

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2006年学术文章

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2006年学术文章

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2006年学术文章

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2006年学术文章

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2006年學術文章

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2006年學術文章

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2006年學術文章

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name

CADASIL: a critical look at a Notch disease.

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CADASIL: a critical look at a Notch disease.

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CADASIL: a critical look at a Notch disease.

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Developmental Neuroscience

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CADASIL: a critical look at a Notch disease.

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Angeliki Louvi

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Joseph F Arboleda-Velasquez

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P2860

Mutations in the human delta homologue, DLL3, cause axial skeletal defects in spondylocostal dysostosis

Alagille syndrome is caused by mutations in human Jagged1, which encodes a ligand for Notch1

Notch signaling: cell fate control and signal integration in development

Splice site mutation causing a seven amino acid Notch3 in-frame deletion in CADASIL

Notch1 and Notch3 instructively restrict bFGF-responsive multipotent neural progenitor cells to an astroglial fate

Pathogenic mutations associated with cerebral autosomal dominant arteriopathy with subcortical infarcts and leukoencephalopathy differently affect Jagged1 binding and Notch3 activity via the RBP/JK signaling Pathway.

Glycosylation regulates Notch signalling.

Differential lesion patterns in CADASIL and sporadic subcortical arteriosclerotic encephalopathy: MR imaging study with statistical parametric group comparison.

An animal model for the molecular genetics of CADASIL. (Cerebral autosomal dominant arteriopathy with subcortical infarcts and leukoencephalopathy).

Transgenic mice expressing mutant Notch3 develop vascular alterations characteristic of cerebral autosomal dominant arteriopathy with subcortical infarcts and leukoencephalopathy.

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10.1159/000090748

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1-2

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Spyros Artavanis-Tsakonas

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2006-01-01T00:00:00Z

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