MyD88 signaling pathway is involved in renal fibrosis by favoring a TH2 immune response and activating alternative M2 macrophages. - Wikidata - wikimedia - TriplyDB

MyD88 signaling pathway is involved in renal fibrosis by favoring a TH2 immune response and activating alternative M2 macrophages.

MyD88 signaling pathway is involved in renal fibrosis by favoring a TH2 immune response and activating alternative M2 macrophages.

http://www.wikidata.org/entity/Q36434363

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The Macrophage Switch in Obesity Development Macrophages During the Fibrotic Process: M2 as Friend and Foe Macrophage heterogeneity, phenotypes, and roles in renal fibrosis Hepatitis B virus infection and immunopathogenesis in a humanized mouse model: induction of human-specific liver fibrosis and M2-like macrophages Macrophage in chronic kidney disease The microbiota and chronic kidney diseases: a double-edged sword Vasohibin-1 deficiency enhances renal fibrosis and inflammation after unilateral ureteral obstruction Transcriptome analysis of renal ischemia/reperfusion injury and its modulation by ischemic pre-conditioning or hemin treatment.Role of TREM1-DAP12 in renal inflammation during obstructive nephropathy.The Role of M2 Macrophages in the Progression of Chronic Kidney Disease following Acute Kidney Injury.TLR4 mutant mice are protected from renal fibrosis and chronic kidney disease progression Inhibition of Toll-Like Receptor 4 Signaling Mitigates Microvascular Loss but Not Fibrosis in a Model of Ischemic Acute Kidney Injury Alternatively activated macrophages determine repair of the infarcted adult murine heart.Toll-like receptor and accessory molecule mRNA expression in humans and mice as well as in murine autoimmunity, transient inflammation, and progressive fibrosis Early infiltration of p40IL12(+)CCR7(+)CD11b(+) cells is critical for fibrosis development.MMP28 promotes macrophage polarization toward M2 cells and augments pulmonary fibrosis.Chronic hepatitis C infection-induced liver fibrogenesis is associated with M2 macrophage activation.Leptospira Interrogans induces fibrosis in the mouse kidney through Inos-dependent, TLR- and NLR-independent signaling pathways.Regulation of wound healing and organ fibrosis by toll-like receptors.Macrophages in kidney injury, inflammation, and fibrosis.IL-4 as a Repurposed Biological Drug for Myocardial Infarction through Augmentation of Reparative Cardiac Macrophages: Proof-of-Concept Data in Mice.Links between coagulation, inflammation, regeneration, and fibrosis in kidney pathology.Soluble Uric Acid Activates the NLRP3 Inflammasome.Gut Bacteria Products Prevent AKI Induced by Ischemia-Reperfusion.The significance of macrophage polarization subtypes for animal models of tissue fibrosis and human fibrotic diseases Type 2 immunity in tissue repair and fibrosis.Protective Effects of Paricalcitol on Peritoneal Remodeling during Peritoneal Dialysis.TNF/Ang-II synergy is obligate for fibroinflammatory pathology, but not for changes in cardiorenal function.Macrophage trafficking as key mediator of adenine-induced kidney injury.Myeloid differentiation primary response gene (MyD) 88 signalling is not essential for intestinal fibrosis development.Renal Macrophages and Dendritic Cells in SLE Nephritis.Immunomodulatory Molecule IRAK-M Balances Macrophage Polarization and Determines Macrophage Responses during Renal Fibrosis.Rictor/mammalian target of rapamycin complex 2 promotes macrophage activation and kidney fibrosis.Recognition of Candida albicans by gingival fibroblasts: The role of TLR2, TLR4/CD14, and MyD88.Renal fibrosis: Primacy of the proximal tubule.CCR2 contributes to the recruitment of monocytes and leads to kidney inflammation and fibrosis development.Functional macrophages and gastrointestinal disorders.Systemic depletion of macrophages in the subacute phase of wound healing reduces hypertrophic scar formation.Possible involvement of Toll-like receptor 7 in the development of type 1 autoimmune pancreatitis.Effective Amelioration of Liver Fibrosis Through Lentiviral Vector Carrying in Murine Model

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P2860

MyD88 signaling pathway is involved in renal fibrosis by favoring a TH2 immune response and activating alternative M2 macrophages.

http://www.wikidata.org/entity/Q36434363

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MOLMED.2012.00131

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Molecular Medicine

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Alvaro Pacheco-Silva

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Angela Castoldi

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Cassiano Donizetti de Oliveira

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Giselle Martins Gonçalves

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Iolanda Midea Cuccovia

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Marcos Antonio Cenedeze

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Matheus Correa-Costa

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Meire Ioshie Hyane

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Simone Aparecida Teixeira

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Yuri Felipe Souza Guise

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P2860

Exploring the full spectrum of macrophage activation

Gout-associated uric acid crystals activate the NALP3 inflammasome

IL-13 effector functions

Pulmonary fibrosis: pathogenesis, etiology and regulation

The role of Toll-like receptor 2 in inflammation and fibrosis during progressive renal injury.

Mechanistic connection between inflammation and fibrosis

An IL-13 inhibitor blocks the development of hepatic fibrosis during a T-helper type 2-dominated inflammatory response.

Toll-like receptor 4 and hepatic fibrogenesis

Sensing the outside world: TSLP regulates barrier immunity.

TLR4 promotes fibrosis but attenuates tubular damage in progressive renal injury.

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18

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Tarcio Teodoro Braga

Marcelo N. Muscara

Katia Regina Perez

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2012-10-24T00:00:00Z

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229013471

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