Integrative molecular profiling reveals asparagine synthetase is a target in castration-resistant prostate cancer.
about
The structure and function of NKAIN2-a candidate tumor suppressorAn Integrative Genomics Approach for Associating Genome-Wide Association Studies Information With Localized and Metastatic Prostate Cancer PhenotypesPolymorphism in asparagine synthetase is associated with overall survival of hepatocellular carcinoma patients.Targeting poly(ADP-ribose) polymerase and the c-Myb-regulated DNA damage response pathway in castration-resistant prostate cancer.Polymorphisms of asparaginase pathway and asparaginase-related complications in children with acute lymphoblastic leukemia.The Emerging Hallmarks of Cancer MetabolismAsparagine synthetase is an independent predictor of surgical survival and a potential therapeutic target in hepatocellular carcinomaFilamentation of Metabolic Enzymes in Saccharomyces cerevisiae.Metabolic Alterations Caused by KRAS Mutations in Colorectal Cancer Contribute to Cell Adaptation to Glutamine Depletion by Upregulation of Asparagine Synthetase.Optimized CGenFF force-field parameters for acylphosphate and N-phosphonosulfonimidoyl functional groups.NKAIN2 functions as a novel tumor suppressor in prostate cancer.Elucidation of a novel phenformin derivative on glucose-deprived stress responses in HT-29 cells.Knockdown of asparagine synthetase (ASNS) suppresses cell proliferation and inhibits tumor growth in gastric cancer cells.Pharmacological inhibition of fatty-acid oxidation synergistically enhances the effect of l-asparaginase in childhood ALL cells.Expression array analysis of the hepatocyte growth factor invasive program.Down-regulation of asparagine synthetase induces cell cycle arrest and inhibits cell proliferation of breast cancer.Asparagine depletion potentiates the cytotoxic effect of chemotherapy against brain tumors.Similarities and Distinctions of Cancer and Immune Metabolism in Inflammation and Tumors.Association between protocadherin 8 promoter hypermethylation and the pathological status of prostate cancer.Pyruvate kinase is a dosage-dependent regulator of cellular amino acid homeostasis.Loss of asparagine synthetase suppresses the growth of human lung cancer cells by arresting cell cycle at G0/G1 phase.Asparagine synthetase expression is associated with the sensitivity to asparaginase in extranodal natural killer/T-cell lymphoma in vivo and in vitroConstruction of a set of novel and robust gene expression signatures predicting prostate cancer recurrence
P2860
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P2860
Integrative molecular profiling reveals asparagine synthetase is a target in castration-resistant prostate cancer.
description
2012 nî lūn-bûn
@nan
2012年の論文
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2012年学术文章
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2012年学术文章
@zh-cn
2012年学术文章
@zh-hans
2012年学术文章
@zh-my
2012年学术文章
@zh-sg
2012年學術文章
@yue
2012年學術文章
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2012年學術文章
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name
Integrative molecular profilin ...... ion-resistant prostate cancer.
@ast
Integrative molecular profilin ...... ion-resistant prostate cancer.
@en
type
label
Integrative molecular profilin ...... ion-resistant prostate cancer.
@ast
Integrative molecular profilin ...... ion-resistant prostate cancer.
@en
prefLabel
Integrative molecular profilin ...... ion-resistant prostate cancer.
@ast
Integrative molecular profilin ...... ion-resistant prostate cancer.
@en
P2093
P2860
P1476
Integrative molecular profilin ...... ion-resistant prostate cancer.
@en
P2093
Ana Aparicio
David Cogdell
Eleni Efstathiou
Heng Huang
Ismaël H Koumakpayi
Jasreman Dhillon
Kanishka Sircar
P2860
P304
P356
10.1016/J.AJPATH.2011.11.030
P407
P577
2012-01-11T00:00:00Z