Proteomic analysis of lithium-induced nephrogenic diabetes insipidus: mechanisms for aquaporin 2 down-regulation and cellular proliferation - Wikidata - wikimedia - TriplyDB

Proteomic analysis of lithium-induced nephrogenic diabetes insipidus: mechanisms for aquaporin 2 down-regulation and cellular proliferation

Proteomic analysis of lithium-induced nephrogenic diabetes insipidus: mechanisms for aquaporin 2 down-regulation and cellular proliferation

http://www.wikidata.org/entity/Q36491091

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Current and future treatment options in SIADH The Trafficking of the Water Channel Aquaporin-2 in Renal Principal Cells-a Potential Target for Pharmacological Intervention in Cardiovascular Diseases Molecular mechanisms of urea transport in health and disease A novel therapeutic effect of statins on nephrogenic diabetes insipidus Proteomic analysis of rat hippocampus exposed to the antidepressant paroxetine Identification of phosphorylation-dependent binding partners of aquaporin-2 using protein mass spectrometry Akt and ERK1/2 pathways are components of the vasopressin signaling network in rat native IMCD Mice lacking mPGES-1 are resistant to lithium-induced polyuria.GSK3beta mediates renal response to vasopressin by modulating adenylate cyclase activity.Phosphoproteomic profiling reveals vasopressin-regulated phosphorylation sites in collecting duct Expression of transporters involved in urine concentration recovers differently after cessation of lithium treatment.alphaENaC-mediated lithium absorption promotes nephrogenic diabetes insipidus.Management of hyponatraemia in older people: old threats and new opportunities Aquaporin-2 regulation in health and disease.Urea transport in the kidney.Interaction networks of lithium and valproate molecular targets reveal a striking enrichment of apoptosis functional clusters and neurotrophin signaling Glycogen synthase kinase 3α regulates urine concentrating mechanism in mice.Absence of PKC-alpha attenuates lithium-induced nephrogenic diabetes insipidus.Quantitative proteomics identifies vasopressin-responsive nuclear proteins in collecting duct cells.New insights into the dynamic regulation of water and acid-base balance by renal epithelial cells.Dual effect of lithium on NFAT5 activity in kidney cells.Phosphoproteomics of vasopressin signaling in the kidney.Nephrogenic diabetes insipidus: essential insights into the molecular background and potential therapies for treatment.Lithium: a versatile tool for understanding renal physiology.Role of aquaporins in cell proliferation: What else beyond water permeability?Glycogen synthase kinase-3 regulation of urinary concentrating ability Vasopressin and the regulation of aquaporin-2 Rapamycin inhibition of mTORC1 reverses lithium-induced proliferation of renal collecting duct cells.Lithium causes G2 arrest of renal principal cells Role of adenylyl cyclase 6 in the development of lithium-induced nephrogenic diabetes insipidus.Hyponatremia and the syndrome of inappropriate secretion of antidiuretic hormone (SIADH).Dynamic regulation and dysregulation of the water channel aquaporin-2: a common cause of and promising therapeutic target for water balance disorders.Lithium in the Kidney: Friend and Foe?Molecular mechanisms in lithium-associated renal disease: a systematic review.Tankyrase-mediated β-catenin activity regulates vasopressin-induced AQP2 expression in kidney collecting duct mpkCCDc14 cells.Molecular mechanisms regulating aquaporin-2 in kidney collecting duct.Lithium enhances TRAIL-induced apoptosis in human lung carcinoma A549 cells.Aquaporins: The renal water channels.Solid renal tumours of collecting duct origin in patients on chronic lithium therapy EGF Receptor Inhibition by Erlotinib Increases Aquaporin 2-Mediated Renal Water Reabsorption.

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Proteomic analysis of lithium-induced nephrogenic diabetes insipidus: mechanisms for aquaporin 2 down-regulation and cellular proliferation

http://www.wikidata.org/entity/Q36491091

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2008 nî lūn-bûn

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Proceedings of the National Academy of Sciences of the United States of America

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Proteomic analysis of lithium- ...... ion and cellular proliferation

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Jakob Nielsen

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Jason D Hoffert

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Mark A Knepper

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Robert A Fenton

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Søren Nielsen

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P2860

A molecular mechanism for the effect of lithium on development

AKT/PKB signaling: navigating downstream

Requirement for glycogen synthase kinase-3beta in cell survival and NF-kappaB activation

Lithium nephrotoxicity: a progressive combined glomerular and tubulointerstitial nephropathy

Lithium-induced downregulation of aquaporin-2 water channel expression in rat kidney medulla

Lithium inhibits glycogen synthase kinase-3 activity and mimics wingless signalling in intact cells

Segment-specific ENaC downregulation in kidney of rats with lithium-induced NDI.

Application of difference gel electrophoresis to the identification of inner medullary collecting duct proteins.

Lithium intoxication.

Aquaporins in the kidney: from molecules to medicine.

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