A three-state model of telomere control over human proliferative boundaries.
about
Implications of telomeres and telomerase in endometrial pathologyGenetic and molecular identification of three human TPP1 functions in telomerase action: recruitment, activation, and homeostasis set point regulationTelomerase Cajal body protein 1 depletion inhibits telomerase trafficking to telomeres and induces G1 cell cycle arrest in A549 cells.A basal level of DNA damage and telomere deprotection increases the sensitivity of cancer cells to G-quadruplex interactive compounds.GSE4, a Small Dyskerin- and GSE24.2-Related Peptide, Induces Telomerase Activity, Cell Proliferation and Reduces DNA Damage, Oxidative Stress and Cell Senescence in Dyskerin Mutant CellsDynamics of Human Telomerase Holoenzyme Assembly and Subunit Exchange across the Cell Cycle.Telomeres and viruses: common themes of genome maintenance.Complex interactions between the DNA-damage response and mammalian telomeres.Chromatin structure in telomere dynamicsTRF2-Mediated Control of Telomere DNA Topology as a Mechanism for Chromosome-End ProtectionCellular senescence and its effector programs.G-Quadruplex binding enantiomers show chiral selective interactions with human telomere.Beyond Telomerase: Telomere Instability as a Novel Target for Cancer Therapy.Telomere uncapping at the crossroad between cell cycle arrest and carcinogenesisInterchromosomal homology searches drive directional ALT telomere movement and synapsis.Telomere-driven diseases and telomere-targeting therapies.Basic Biology of Oxidative Stress and the Cardiovascular System: Part 1 of a 3-Part Series.TRF1 and TRF2 binding to telomeres is modulated by nucleosomal organization.Retrotransposon-derived p53 binding sites enhance telomere maintenance and genome protection.Mitosis, double strand break repair, and telomeres: a view from the end: how telomeres and the DNA damage response cooperate during mitosis to maintain genome stability.The telomere deprotection response is functionally distinct from the genomic DNA damage response.HoxC5 and miR-615-3p target newly evolved genomic regions to repress hTERT and inhibit tumorigenesis.Distinct Responses of Stem Cells to Telomere Uncapping-A Potential Strategy to Improve the Safety of Cell Therapy.Telomeres: Implications for Cancer Development.In medio stat virtus: unanticipated consequences of telomere dysequilibrium.The BUB3-BUB1 Complex Promotes Telomere DNA Replication.Histone depletion prevents telomere fusions in pre-senescent cells.Local enrichment of HP1alpha at telomeres alters their structure and regulation of telomere protection
P2860
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P2860
A three-state model of telomere control over human proliferative boundaries.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年学术文章
@wuu
2012年学术文章
@zh-cn
2012年学术文章
@zh-hans
2012年学术文章
@zh-my
2012年学术文章
@zh-sg
2012年學術文章
@yue
2012年學術文章
@zh
2012年學術文章
@zh-hant
name
A three-state model of telomere control over human proliferative boundaries.
@ast
A three-state model of telomere control over human proliferative boundaries.
@en
type
label
A three-state model of telomere control over human proliferative boundaries.
@ast
A three-state model of telomere control over human proliferative boundaries.
@en
prefLabel
A three-state model of telomere control over human proliferative boundaries.
@ast
A three-state model of telomere control over human proliferative boundaries.
@en
P2860
P1476
A three-state model of telomere control over human proliferative boundaries.
@en
P2093
Anthony J Cesare
Jan Karlseder
P2860
P304
P356
10.1016/J.CEB.2012.08.007
P577
2012-09-02T00:00:00Z