IRF-3 activation by Sendai virus infection is required for cellular apoptosis and avoidance of persistence.
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Viral apoptosis is induced by IRF-3-mediated activation of BaxPKC alpha regulates Sendai virus-mediated interferon induction through HDAC6 and β-catenindsRNA-activation of TLR3 and RLR signaling: gene induction-dependent and independent effectsNegative regulation of cytoplasmic RNA-mediated antiviral signalingThe RNA-activated Protein Kinase Enhances the Induction of Interferon- and Apoptosis Mediated by Cytoplasmic RNA SensorsIdentification of Caspase-mediated Decay of Interferon Regulatory Factor-3, Exploited by a Kaposi Sarcoma-associated Herpesvirus Immunoregulatory ProteinMelanoma Differentiation-Associated Gene 5 (MDA5) Is Involved in the Innate Immune Response to Paramyxoviridae Infection In VivoNegative regulation of interferon-β gene expression during acute and persistent virus infectionsInduction of interferon-stimulated genes by IRF3 promotes replication of Toxoplasma gondiiCytopathogenesis of Sendai virus in well-differentiated primary pediatric bronchial epithelial cells.High-throughput screening for TLR3-IFN regulatory factor 3 signaling pathway modulators identifies several antipsychotic drugs as TLR inhibitors.Transient activation of the PI3K/Akt pathway promotes Newcastle disease virus replication and enhances anti-apoptotic signaling responsesHuman parainfluenza virus type 2 V protein inhibits interferon production and signaling and is required for replication in non-human primates.RIG-I enhanced interferon independent apoptosis upon Junin virus infection.Host specificity of the anti-interferon and anti-apoptosis activities of parainfluenza virus P/C gene products.Sendai virus pathogenesis in mice is prevented by Ifit2 and exacerbated by interferon.Dose dependent activation of retinoic acid-inducible gene-I promotes both proliferation and apoptosis signals in human head and neck squamous cell carcinoma.Enterovirus 71 protease 2Apro targets MAVS to inhibit anti-viral type I interferon responses.The IRF-3/Bax-mediated apoptotic pathway, activated by viral cytoplasmic RNA and DNA, inhibits virus replication.Phosphatidylinositol 3-kinase signaling delays sendai virus-induced apoptosis by preventing XIAP degradation.Transfected poly(I:C) activates different dsRNA receptors, leading to apoptosis or immunoadjuvant response in androgen-independent prostate cancer cellsIRF3 polymorphisms induce different innate anti-Theiler's virus immune responses in RAW264.7 macrophages.Functions of the cytoplasmic RNA sensors RIG-I and MDA-5: key regulators of innate immunity.Interferon regulatory transcription factor 3 protects mice from uterine horn pathology during Chlamydia muridarum genital infectionCaspase-8-mediated cleavage inhibits IRF-3 protein by facilitating its proteasome-mediated degradationTom70 mediates Sendai virus-induced apoptosis on mitochondria.TLR3 engagement induces IRF-3-dependent apoptosis in androgen-sensitive prostate cancer cells and inhibits tumour growth in vivo.Lymphocytic Choriomeningitis Virus Differentially Affects the Virus-Induced Type I Interferon Response and Mitochondrial Apoptosis Mediated by RIG-I/MAVS.Sendai Virus Induces Persistent Olfactory Dysfunction in a Murine Model of PVOD via Effects on Apoptosis, Cell Proliferation, and Response to Odorants.Role of interferon regulatory factor 3-mediated apoptosis in the establishment and maintenance of persistent infection by Sendai virusInhibition of viral pathogenesis and promotion of the septic shock response to bacterial infection by IRF-3 are regulated by the acetylation and phosphorylation of its coactivatorsHuman parainfluenza virus type 1 C proteins are nonessential proteins that inhibit the host interferon and apoptotic responses and are required for efficient replication in nonhuman primates.Induction of protein kinase PKR-dependent activation of interferon regulatory factor 3 by vaccinia virus occurs through adapter IPS-1 signalingThe C proteins of human parainfluenza virus type 1 (HPIV1) control the transcription of a broad array of cellular genes that would otherwise respond to HPIV1 infectionUbiquitination of the Transcription Factor IRF-3 Activates RIPA, the Apoptotic Pathway that Protects Mice from Viral PathogenesisAkt inhibitor Akt-IV blocks virus replication through an Akt-independent mechanism.Transcriptional re-programming of primary macrophages reveals distinct apoptotic and anti-tumoral functions of IRF-3 and IRF-7.A novel human parainfluenza virus type 1 (HPIV1) with separated P and C genes is useful for generating C gene mutants for evaluation as live-attenuated virus vaccine candidates.RIG-I-like receptor-induced IRF3 mediated pathway of apoptosis (RIPA): a new antiviral pathway.Anti-viral and anti-inflammatory mechanisms of the innate immune transcription factor interferon regulatory factor 3: relevance to human CNS diseases.
P2860
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P2860
IRF-3 activation by Sendai virus infection is required for cellular apoptosis and avoidance of persistence.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年学术文章
@wuu
2008年学术文章
@zh-cn
2008年学术文章
@zh-hans
2008年学术文章
@zh-my
2008年学术文章
@zh-sg
2008年學術文章
@yue
2008年學術文章
@zh
2008年學術文章
@zh-hant
name
IRF-3 activation by Sendai vir ...... and avoidance of persistence.
@ast
IRF-3 activation by Sendai vir ...... and avoidance of persistence.
@en
type
label
IRF-3 activation by Sendai vir ...... and avoidance of persistence.
@ast
IRF-3 activation by Sendai vir ...... and avoidance of persistence.
@en
prefLabel
IRF-3 activation by Sendai vir ...... and avoidance of persistence.
@ast
IRF-3 activation by Sendai vir ...... and avoidance of persistence.
@en
P2093
P2860
P921
P356
P1433
P1476
IRF-3 activation by Sendai vir ...... and avoidance of persistence.
@en
P2093
Ganes C Sen
Kristi Peters
Saurabh Chattopadhyay
P2860
P304
P356
10.1128/JVI.02536-07
P407
P577
2008-01-23T00:00:00Z