Alveolar macrophages are a major determinant of early responses to viral lung infection but do not influence subsequent disease development
about
Depletion of Alveolar Macrophages Does Not Prevent Hantavirus Disease Pathogenesis in Golden Syrian HamstersImmune Response to Human Metapneumovirus Infection: What We Have Learned from the Mouse ModelDendritic cells in human Pneumovirus and Metapneumovirus infectionsNatural killer cells in infection and inflammation of the lungAlveolar Macrophages Prevent Lethal Influenza Pneumonia By Inhibiting Infection Of Type-1 Alveolar Epithelial CellsAlveolar macrophages are essential for protection from respiratory failure and associated morbidity following influenza virus infection.The chemokine MIP1alpha/CCL3 determines pathology in primary RSV infection by regulating the balance of T cell populations in the murine lung.Effect of recombinant Panton-Valentine leukocidin in vitro on apoptosis and cytokine production of human alveolar macrophages.INHIBITORY EFFECT OF EMODIN ON RAW 264.7 ACTIVATED WITH DOUBLE STRANDED RNA ANALOGUE POLY I:CInterleukin 18 coexpression during respiratory syncytial virus infection results in enhanced disease mediated by natural killer cells.Early local immune defences in the respiratory tractDelivery of cytokines by recombinant virus in early life alters the immune response to adult lung infection.Subversion of pulmonary dendritic cell function by paramyxovirus infectionsAlpha/beta interferon receptor signaling amplifies early proinflammatory cytokine production in the lung during respiratory syncytial virus infection.Critical role of airway macrophages in modulating disease severity during influenza virus infection of mice.CD25+ natural regulatory T cells are critical in limiting innate and adaptive immunity and resolving disease following respiratory syncytial virus infection.Early activation of MAP kinases by influenza A virus X-31 in murine macrophage cell linesGene expression differences in lungs of mice during secondary immune responses to respiratory syncytial virus infection.IL-10 regulates viral lung immunopathology during acute respiratory syncytial virus infection in miceDecreased Toll-like receptor 8 expression and lower TNF-α synthesis in infants with acute RSV infectionAlveolar macrophages contribute to the pathogenesis of human metapneumovirus infection while protecting against respiratory syncytial virus infectionInfection-induced regulation of natural killer cells by macrophages and collagen at the lymph node subcapsular sinus.Characterization of the resistance of SJL/J mice to pneumonia virus of mice, a model for infantile bronchiolitis due to a respiratory syncytial virusProtection and mechanism of action of a novel human respiratory syncytial virus vaccine candidate based on the extracellular domain of small hydrophobic protein.A systems-based approach to analyse the host response in murine lung macrophages challenged with respiratory syncytial virus.Depletion of alveolar macrophages ameliorates virus-induced disease following a pulmonary coronavirus infection.Progress in understanding and controlling respiratory syncytial virus: still crazy after all these yearsAlveolar macrophage-derived type I interferons orchestrate innate immunity to RSV through recruitment of antiviral monocytesDepletion of alveolar macrophages prolongs survival in response to acute pneumovirus infection.An essential role for TH2-type responses in limiting acute tissue damage during experimental helminth infectionRegulatory T cells expressing granzyme B play a critical role in controlling lung inflammation during acute viral infection.Mice with asthma are more resistant to influenza virus infection and NK cells activated by the induction of asthma have potentially protective effects.Type I Interferon Induced Epigenetic Regulation of Macrophages Suppresses Innate and Adaptive Immunity in Acute Respiratory Viral InfectionMacrophage Polarization in Virus-Host InteractionsPartial Attenuation of Respiratory Syncytial Virus with a Deletion of a Small Hydrophobic Gene Is Associated with Elevated Interleukin-1β Responses.Cytokine-like factor 1 gene expression is enriched in idiopathic pulmonary fibrosis and drives the accumulation of CD4+ T cells in murine lungs: evidence for an antifibrotic role in bleomycin injury.The Helminth-Derived Immunomodulator AvCystatin Reduces Virus Enhanced Inflammation by Induction of Regulatory IL-10+ T Cells.Innate and adaptive cellular phenotypes contributing to pulmonary disease in mice after respiratory syncytial virus immunization and infection.The secreted G protein of human respiratory syncytial virus antagonizes antibody-mediated restriction of replication involving macrophages and complement.T cell responses are elicited against Respiratory Syncytial Virus in the absence of signalling through TLRs, RLRs and IL-1R/IL-18R.
P2860
Q26314485-478EB0D6-586A-484A-97D5-35C93F350676Q26782209-DE39BF53-2C6C-4AE3-947B-38F41A28AFB6Q26865337-24570A82-D62E-475A-98F5-0FFB76B0A8E0Q28391714-BD9A9BF8-D96D-482D-8713-C237FC2E6295Q30275003-68EAF10C-323D-4503-A98D-1B229F021D59Q31157006-5828DFD5-2AC9-4D4B-AA54-C46B0A0B9B82Q33535780-5C80E560-2654-4F87-ACB0-1DD20D0942F3Q33572786-1C174126-0314-470A-8231-05EC599CB1D0Q33624026-A3957252-DFF7-4114-8D18-6372CA8E31DAQ33769399-F277CF97-87A9-408B-9603-3057253182CEQ33824093-FA2AEE2E-2B98-4FFC-B73A-10C3C91EAADEQ33826900-A1BAEA4B-169B-47FB-B16F-02578C2983A4Q33832308-F1C8BA7F-64CB-46D5-9ABA-7516F68283EFQ33887844-DAFEEFA8-1C06-4710-9750-23CF228185FAQ33964051-224F36E8-B3F9-4687-8EE9-C884112D657DQ34055489-C2AD5B71-0ABF-4901-876A-49C4583D99F0Q34104767-98642D38-6BAA-491E-8841-16A57B52D690Q34120191-975A0064-6DF1-481D-B87C-CA3F49FBFB7AQ34185860-6FCF02CB-7688-4943-879E-D61DEDA4B03FQ34282702-70B6E85A-408B-4E99-81F4-8DD282550F77Q34305520-CDD84D13-F1B5-4FBE-B327-A685553FA1B1Q34354896-9B6710F2-28BE-401F-89C3-2CF8254EF35BQ34450231-666B9D01-5028-443F-8366-57E945ED7B09Q34542687-5DBF55AF-8F81-4CC8-B148-57ABB012D275Q34626385-3A050E5D-3462-463C-93B3-EA4B408C76F9Q35114458-F834A416-1E24-41BD-9F26-956C30C671C3Q35566601-88154332-4192-4672-9432-891F42606E15Q35568416-BC3F2D14-0D80-425C-A0F2-074D4DFE0431Q35668961-E0F0DBD5-B643-4152-8915-FE46D188E41CQ35742768-E4389384-A07D-44F6-92D2-218D913C5185Q35765491-E3EA5700-50CC-4DED-9579-E61FCF93142EQ35834816-49FBD628-1144-48E5-9C96-CD52667CDBC9Q35879429-0846611E-27FB-49B7-B0E7-AD0FDA9F5805Q35882034-50977504-A9CD-4F02-BF23-45A4E0E1D7F0Q35913930-71644714-4621-4280-A2A6-899244537D50Q35967027-C93AE6D9-A52C-4A66-94B6-4DAD8124FA54Q36113037-B9FD02DB-23CB-44F7-8D56-FDA4C5141831Q36202706-2D1333F7-55D7-4CDC-BCC5-A236C3F9723CQ36276609-FEA3E188-16FD-466F-BDE5-D2DF98D918F5Q36387954-8EB41C10-E4A3-443C-BD96-899A5D8C8C35
P2860
Alveolar macrophages are a major determinant of early responses to viral lung infection but do not influence subsequent disease development
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
2008年论文
@zh
2008年论文
@zh-cn
name
Alveolar macrophages are a maj ...... subsequent disease development
@ast
Alveolar macrophages are a maj ...... subsequent disease development
@en
type
label
Alveolar macrophages are a maj ...... subsequent disease development
@ast
Alveolar macrophages are a maj ...... subsequent disease development
@en
prefLabel
Alveolar macrophages are a maj ...... subsequent disease development
@ast
Alveolar macrophages are a maj ...... subsequent disease development
@en
P2093
P2860
P50
P356
P1433
P1476
Alveolar macrophages are a maj ...... subsequent disease development
@en
P2093
Belinda Wang
Hongwei Wang
Philippa K Pribul
P2860
P304
P356
10.1128/JVI.02541-07
P407
P577
2008-02-20T00:00:00Z