about
The role of pro-resolution lipid mediators in infectious diseaseReduced lung function in a chronic asthma model is associated with prolonged inflammation, but independent of peribronchial fibrosis.The chemokine MIP1alpha/CCL3 determines pathology in primary RSV infection by regulating the balance of T cell populations in the murine lung.Respiratory viral infections in infants: causes, clinical symptoms, virology, and immunology.The late, but not early, asthmatic response is dependent on IL-5 and correlates with eosinophil infiltration.The role of viruses in development or exacerbation of atopic asthma.CD25+ natural regulatory T cells are critical in limiting innate and adaptive immunity and resolving disease following respiratory syncytial virus infection.Lymphoid and myeloid cell populations in the non-pregnant human Fallopian tube and in ectopic pregnancyThe human cathelicidin LL-37 has antiviral activity against respiratory syncytial virus.Using bacterial biomarkers to identify early indicators of cystic fibrosis pulmonary exacerbation onsetWogonin induces eosinophil apoptosis and attenuates allergic airway inflammationEnhanced virulence, airway inflammation and impaired lung function induced by respiratory syncytial virus deficient in secreted G proteinOX40 ligand and programmed cell death 1 ligand 2 expression on inflammatory dendritic cells regulates CD4 T cell cytokine production in the lung during viral disease.Mouse models of rhinovirus-induced disease and exacerbation of allergic airway inflammationRespiratory syncytial virus infection provokes airway remodelling in allergen-exposed mice in absence of prior allergen sensitization.Inhalation of stable dust extract prevents allergen induced airway inflammation and hyperresponsiveness.Research needs in allergy: an EAACI position paper, in collaboration with EFA.Alveolar macrophages are a major determinant of early responses to viral lung infection but do not influence subsequent disease developmentCathelicidins Have Direct Antiviral Activity against Respiratory Syncytial Virus In Vitro and Protective Function In Vivo in Mice and Humans.Healthy but not RSV-infected lung epithelial cells profoundly inhibit T cell activationMolecular and cellular mechanisms in the viral exacerbation of asthma.Prostaglandin F2alpha-F-prostanoid receptor signaling promotes neutrophil chemotaxis via chemokine (C-X-C motif) ligand 1 in endometrial adenocarcinomaEffector and central memory T helper 2 cells respond differently to peptide immunotherapy.Function of the intestinal epithelium and its dysregulation in inflammatory bowel disease.Respiratory and gastrointestinal epithelial modulation of the immune response during viral infection.Peptide immunotherapy for childhood allergy - addressing translational challenges.Pediatric pulmonology year in review 2014: Part 2.Combination peptide immunotherapy based on T-cell epitope mapping reduces allergen-specific IgE and eosinophilia in allergic airway inflammation.Chest auscultatory signs in infants presenting to A&E with bronchiolitis.Republished: novel insights into immune and inflammatory responses to respiratory viruses.Respiratory viral infections as promoters of allergic sensitization and asthma in animal models.Local CD11c+ MHC class II- precursors generate lung dendritic cells during respiratory viral infection, but are depleted in the process.Unravelling synergistic immune interactions between respiratory virus infections and allergic airway inflammation.Lung dendritic cells in respiratory syncytial virus bronchiolitis.Plasmacytoid dendritic cells limit viral replication, pulmonary inflammation, and airway hyperresponsiveness in respiratory syncytial virus infection.Latency and persistence of respiratory syncytial virus despite T cell immunity.Sustained increases in numbers of pulmonary dendritic cells after respiratory syncytial virus infection.Critical roles for interleukin-4 and interleukin-5 during respiratory syncytial virus infection in the development of airway hyperresponsiveness after airway sensitization.Type 4 phosphodiesterase inhibitors attenuate respiratory syncytial virus-induced airway hyper-responsiveness and lung eosinophilia.Transfer of the enhancing effect of respiratory syncytial virus infection on subsequent allergic airway sensitization by T lymphocytes.
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Jürgen Schwarze
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Jürgen Schwarze
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Jürgen Schwarze
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Jürgen Schwarze
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Jürgen Schwarze
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Jürgen Schwarze
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Jürgen Schwarze
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Jürgen Schwarze
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Jürgen Schwarze
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Jürgen Schwarze
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Jürgen Schwarze
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Jürgen Schwarze
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Jürgen Schwarze
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Jürgen Schwarze
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Jürgen Schwarze
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P106
P1153
7005823373
P21
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0000-0002-6899-748X