Increased B-lymphopoiesis by interleukin 7 induces bone loss in mice with intact ovarian function: similarity to estrogen deficiency. - Wikidata - wikimedia - TriplyDB

Increased B-lymphopoiesis by interleukin 7 induces bone loss in mice with intact ovarian function: similarity to estrogen deficiency.

Increased B-lymphopoiesis by interleukin 7 induces bone loss in mice with intact ovarian function: similarity to estrogen deficiency.

http://www.wikidata.org/entity/Q36566505

about

Osteoimmunology: interactions of the bone and immune system Osteoporosis in experimental postmenopausal polyarthritis: the relative contributions of estrogen deficiency and inflammation Interleukin-7 and interleukin-15 for cancer The role of estrogen and androgen receptors in bone health and disease Estrogen regulation of immune cell bone interactions Bone marrow stromal cells mediate androgenic suppression of B lymphocyte development IL-7 induces bone loss in vivo by induction of receptor activator of nuclear factor kappa B ligand and tumor necrosis factor alpha from T cells IL-7 up-regulates TNF-alpha-dependent osteoclastogenesis in patients affected by solid tumor.Lymphocytes and the Dap12 adaptor are key regulators of osteoclast activation associated with gonadal failure.Multiple myeloma disrupts the TRANCE/ osteoprotegerin cytokine axis to trigger bone destruction and promote tumor progression.Daidzein prevents the increase in CD4+CD28null T cells and B lymphopoesis in ovariectomized mice: a key mechanism for anti-osteoclastogenic effect.T cells: critical bone regulators in health and disease.How B cells influence bone biology in health and disease The immune system and bone.Estrogens and Androgens in Skeletal Physiology and Pathophysiology.Estrogen deficiency and bone loss: an inflammatory tale.Increased production of IL-7 uncouples bone formation from bone resorption during estrogen deficiency T-cells and B-cells in osteoporosis Disruption of the estrogen receptor beta gene in mice causes myeloproliferative disease resembling chronic myeloid leukemia with lymphoid blast crisis B cells and T cells are critical for the preservation of bone homeostasis and attainment of peak bone mass in vivo T cells and post menopausal osteoporosis in murine models Osteoblast-specific overexpression of human interleukin-7 rescues the bone mass phenotype of interleukin-7-deficient female mice Absence of sclerostin adversely affects B-cell survival.Receptor activator of nuclear factor κB ligand (RANKL) protein expression by B lymphocytes contributes to ovariectomy-induced bone loss B cells and osteoblast and osteoclast development.The role of T lymphocytes in bone metabolism.Role of interleukin-7 in bone and T-cell homeostasis.Immune responses and bone loss: the estrogen connection.The role of the immune system in the pathophysiology of osteoporosis.An essential role of cytosolic phospholipase A2alpha in prostaglandin E2-mediated bone resorption associated with inflammation Interleukin-7 induced immunopathology in arthritis.A Special Ingredient (VtR) Containing Oligostilbenes Isolated from Vitis thunbergii Prevents Bone Loss in Ovariectomized Mice: In Vitro and In Vivo Study.Interaction among cells of bone, immune system, and solid tumors leads to bone metastases.Possible role of S-equol on bone loss via amelioration of inflammatory indices in ovariectomized mice.The Interplay between the bone and the immune system Role of Bcl2 in osteoclastogenesis and PTH anabolic actions in bone Hormonal regulation of B-cell function and systemic lupus erythematosus.The Role of Inflammatory Cytokines, the RANKL/OPG Axis, and the Immunoskeletal Interface in Physiological Bone Turnover and Osteoporosis.Role of RANKL-induced osteoclast formation and MMP-dependent matrix degradation in bone destruction by breast cancer metastasis.Aromatase-deficient mice spontaneously develop a lymphoproliferative autoimmune disease resembling Sjogren's syndrome.

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P2860

Increased B-lymphopoiesis by interleukin 7 induces bone loss in mice with intact ovarian function: similarity to estrogen deficiency.

http://www.wikidata.org/entity/Q36566505

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Increased B-lymphopoiesis by i ...... larity to estrogen deficiency.

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P2860

Soluble interleukin-6 receptor triggers osteoclast formation by interleukin 6

Stimulation of B-cell progenitors by cloned murine interleukin-7

Interleukin-11: a new cytokine critical for osteoclast development

Interleukin-1 receptor antagonist decreases bone loss and bone resorption in ovariectomized rats.

Estrogen deficiency stimulates B lymphopoiesis in mouse bone marrow.

Pregnancy-related steroids are potential negative regulators of B lymphopoiesis.

17 beta-estradiol inhibits interleukin-6 production by bone marrow-derived stromal cells and osteoblasts in vitro: a potential mechanism for the antiosteoporotic effect of estrogens.

Interleukin-1 receptor antagonist and tumor necrosis factor binding protein decrease osteoclast formation and bone resorption in ovariectomized mice

Resolution and characterization of pro-B and pre-pro-B cell stages in normal mouse bone marrow

Suppression of B lymphopoiesis during normal pregnancy.

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