The no-reflow phenomenon: A basic mechanism of myocardial ischemia and reperfusion.
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Myocardial protection with mild hypothermiaAll-Trans Retinoic Acid Ameliorates Myocardial Ischemia/Reperfusion Injury by Reducing Cardiomyocyte Apoptosis.Effect of microvascular obstruction and intramyocardial hemorrhage by CMR on LV remodeling and outcomes after myocardial infarction: a systematic review and meta-analysis.Limitations and potential clinical application on contrast echocardiographyMeasuring Water Distribution in the Heart: Preventing Edema Reduces Ischemia-Reperfusion Injury.Rapid short-duration hypothermia with cold saline and endovascular cooling before reperfusion reduces microvascular obstruction and myocardial infarct size.Magnetic resonance imaging delineates the ischemic area at risk and myocardial salvage in patients with acute myocardial infarction.Telmisartan protects against microvascular dysfunction during myocardial ischemia/reperfusion injury by activation of peroxisome proliferator-activated receptor γ.Effect of statin pretreatment on myocardial perfusion in patients undergoing primary percutaneous coronary intervention: a systematic review and meta-analysis.Comparison of no-reflow phenomenon after percutaneous coronary intervention for acute myocardial infarction between smokers and nonsmokersNon-invasive evaluation of myocardial reperfusion by transthoracic Doppler echocardiography and single-photon emission computed tomography in patients with anterior acute myocardial infarction.The role of von Willebrand factor and ADAMTS13 in the no-reflow phenomenon: after primary percutaneous coronary interventionSecretome of apoptotic peripheral blood cells (APOSEC) attenuates microvascular obstruction in a porcine closed chest reperfused acute myocardial infarction model: role of platelet aggregation and vasodilation.CMR of microvascular obstruction and hemorrhage in myocardial infarction.Temporal course of microvascular obstruction after myocardial infarction assessed by MRI.Cardiovascular MR T2-STIR imaging does not discriminate between intramyocardial haemorrhage and microvascular obstruction during the subacute phase of a reperfused myocardial infarction.Sphingolipid therapy in myocardial ischemia-reperfusion injury.New horizons in cardioprotection: recommendations from the 2010 National Heart, Lung, and Blood Institute WorkshopDelivery of Nox2-NADPH oxidase siRNA with polyketal nanoparticles for improving cardiac function following myocardial infarctionHSPA12B Attenuated Acute Myocardial Ischemia/reperfusion Injury via Maintaining Endothelial Integrity in a PI3K/Akt/mTOR-dependent Mechanism.Phosphomimetic modulation of eNOS improves myocardial reperfusion and mimics cardiac postconditioning in mice.Ischemia/Reperfusion Injury following Acute Myocardial Infarction: A Critical Issue for Clinicians and Forensic Pathologists.Endothelial mitochondria and heart disease.No-reflow: again prevention is better than treatment.How and when do myocytes die during ischemia and reperfusion: the late phase.No-reflow phenomenon: maintaining vascular integrity.Translating cardioprotection for patient benefit: position paper from the Working Group of Cellular Biology of the Heart of the European Society of Cardiology.Pharmacological and physical prevention and treatment of no-reflow after primary percutaneous coronary intervention in ST-segment elevation myocardial infarction.Coronary microvascular obstruction in acute myocardial infarction.Prospects for improving neovascularization of the ischemic heart: Lessons from development.Ischemic postconditioning: mechanisms, comorbidities, and clinical application.Magnetic resonance imaging-defined areas of microvascular obstruction after acute myocardial infarction represent microvascular destruction and haemorrhage.No-reflow phenomenon and endothelial glycocalyx of microcirculation.Comparing the effect of clopidogrel versus ticagrelor on coronary microvascular dysfunction in acute coronary syndrome patients (TIME trial): study protocol for a randomized controlled trial.The influence of microvascular injury on native T1 and T2* relaxation values after acute myocardial infarction: implications for non-contrast-enhanced infarct assessment.Exogenous nitric oxide requires an endothelial glycocalyx to prevent postischemic coronary vascular leak in guinea pig hearts.MRI manifestations of persistent microvascular obstruction and acute left ventricular remodeling in an experimental reperfused myocardial infarctionThe relationship of coronary flow to neutrophil/lymphocyte ratio in patients undergoing primary percutaneous coronary interventionGrowth differentiation factor 15 may protect the myocardium from no‑reflow by inhibiting the inflammatory‑like response that predominantly involves neutrophil infiltration.Ischaemia-reperfusion injury impairs tissue plasminogen activator release in man.
P2860
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P2860
The no-reflow phenomenon: A basic mechanism of myocardial ischemia and reperfusion.
description
2006 nî lūn-bûn
@nan
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
2006年论文
@zh
2006年论文
@zh-cn
name
The no-reflow phenomenon: A basic mechanism of myocardial ischemia and reperfusion.
@ast
The no-reflow phenomenon: A basic mechanism of myocardial ischemia and reperfusion.
@en
type
label
The no-reflow phenomenon: A basic mechanism of myocardial ischemia and reperfusion.
@ast
The no-reflow phenomenon: A basic mechanism of myocardial ischemia and reperfusion.
@en
prefLabel
The no-reflow phenomenon: A basic mechanism of myocardial ischemia and reperfusion.
@ast
The no-reflow phenomenon: A basic mechanism of myocardial ischemia and reperfusion.
@en
P2860
P1476
The no-reflow phenomenon: A basic mechanism of myocardial ischemia and reperfusion.
@en
P2093
Robert A Kloner
Thorsten Reffelmann
P2860
P2888
P304
P356
10.1007/S00395-006-0615-2
P577
2006-09-01T00:00:00Z