"Reperfusion injury" by oxygen-derived free radicals? Effect of superoxide dismutase plus catalase, given at the time of reperfusion, on myocardial infarct size, contractile function, coronary microvasculature, and regional myocardial blood flow.
about
Congestive heart failure. New frontiersTransvalvular left ventricular assistance in acute myocardial infarction with cardiogenic shock and high risk angioplasty: experimental and clinical results with the Hemopump."No-reflow" phenomenon following percutaneous coronary intervention: an uncommon complication.Reperfusion injury.Cardiac protection by pharmacological modulation of inflammation.The "no-reflow" phenomenon: basic science and clinical correlates.A hypothesis for the cause of complex regional pain syndrome-type I (reflex sympathetic dystrophy): pain due to deep-tissue microvascular pathologyRole of platelet-activating factor in the reperfusion injury of rabbit ischemic heartRole of platelet-activating factor in polymorphonuclear neutrophil recruitment in reperfused ischemic rabbit heart.Allopurinol and amlodipine improve coronary vasodilatation after myocardial ischaemia and reperfusion in anaesthetized dogs.Impaired endothelium-dependent relaxation of dog coronary arteries after myocardial ischaemia and reperfusion: prevention by amlodipine, propranolol and allopurinolBeneficial effects of N-acetylcysteine and cysteine in stunned myocardium in perfused rat heart.Microvascular alterations after temporary coronary artery occlusion: the no-reflow phenomenon.Reduction of ischemia/reperfusion injury with bendavia, a mitochondria-targeting cytoprotective Peptide.The no-reflow phenomenon: A basic mechanism of myocardial ischemia and reperfusion.The effects of allopurinol on the ultrastructure of ischaemic and reperfused large intestine of sheep.Reduction of oxidative stress does not affect recovery of myocardial function: warm continuous versus cold intermittent blood cardioplegia.Stunning: a radical re-view.Intermittent hypoxia has organ-specific effects on oxidative stressReperfusion-induced injury: a possible role for oxidant stress and its manipulation.Oxygen-derived free radicals and myocardial reperfusion injury: an overview.The diagnosis and treatment of the no-reflow phenomenon in patients with myocardial infarction undergoing percutaneous coronary interventionDelineating the relationships among the formation of reactive oxygen species, cell membrane instability and innate autoimmunity in intestinal reperfusion injury.There is more to life than revascularization: therapeutic targeting of myocardial ischemia/reperfusion injury.No-reflow phenomenon: maintaining vascular integrity.Oxygen-derived free radicals and postischemic myocardial reperfusion: therapeutic implications.Pharmacological approaches to the treatment of oxidative stress-induced cardiovascular dysfunctions.Relationships between structure and effects of ACE inhibitors: comparative effects in myocardial ischaemic/reperfusion injury.Future cardioprotective considerations.Cytochrome c oxidase and cardiolipin alterations in response to skeletal muscle ischaemia and reperfusion.Loss of plasma membrane integrity, complement response and formation of reactive oxygen species during early myocardial ischemia/reperfusion.Timing of treatment with oxygen radical scavengers and its influence on reperfusion injury.Combination antioxidant effect of α-tocoferol and erdosteine in ischemia-reperfusion injury in rat model.Post-resuscitative hypothermic bypass reduces ischemic brain injury in swine.Moderation of myocardial ischemia reperfusion injury by calcium channel and calmodulin receptor inhibition.Relationship between no reflow and infarct size as influenced by the duration of ischemia and reperfusion.Microvascular reperfusion injury: rapid expansion of anatomic no reflow during reperfusion in the rabbit.Is microvascular protection by cariporide and ischemic preconditioning causally linked to myocardial salvage?Lack of significant effects of superoxide dismutase and catalase on development of reperfusion arrhythmias.Post-conditioning induced cardioprotection requires signaling through a redox-sensitive mechanism, mitochondrial ATP-sensitive K+ channel and protein kinase C activation.
P2860
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P2860
"Reperfusion injury" by oxygen-derived free radicals? Effect of superoxide dismutase plus catalase, given at the time of reperfusion, on myocardial infarct size, contractile function, coronary microvasculature, and regional myocardial blood flow.
description
1989 nî lūn-bûn
@nan
1989年の論文
@ja
1989年学术文章
@wuu
1989年学术文章
@zh-cn
1989年学术文章
@zh-hans
1989年学术文章
@zh-my
1989年学术文章
@zh-sg
1989年學術文章
@yue
1989年學術文章
@zh
1989年學術文章
@zh-hant
name
"Reperfusion injury" by oxygen ...... egional myocardial blood flow.
@en
"Reperfusion injury" by oxygen ...... egional myocardial blood flow.
@nl
type
label
"Reperfusion injury" by oxygen ...... egional myocardial blood flow.
@en
"Reperfusion injury" by oxygen ...... egional myocardial blood flow.
@nl
prefLabel
"Reperfusion injury" by oxygen ...... egional myocardial blood flow.
@en
"Reperfusion injury" by oxygen ...... egional myocardial blood flow.
@nl
P356
P1433
P1476
"Reperfusion injury" by oxygen ...... egional myocardial blood flow.
@en
P2093
Przyklenk K
P356
10.1161/01.RES.64.1.86
P577
1989-01-01T00:00:00Z