Conditional deletions refine the embryonic requirement for Dlk1. - Wikidata - wikimedia - TriplyDB

Conditional deletions refine the embryonic requirement for Dlk1.

Conditional deletions refine the embryonic requirement for Dlk1.

http://www.wikidata.org/entity/Q36605372

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MicroRNA-126-5p promotes endothelial proliferation and limits atherosclerosis by suppressing Dlk1 The Missing lnc(RNA) between the pancreatic β-cell and diabetes DLK1/PREF1 regulates nutrient metabolism and protects from steatosis.Antagonistic roles in fetal development and adult physiology for the oppositely imprinted Grb10 and Dlk1 genes.Maternal and zygotic Zfp57 modulate NOTCH signaling in cardiac development.Preadipocyte factor 1 induces pancreatic ductal cell differentiation into insulin-producing cells.Pituitary phenotypes of mice lacking the notch signalling ligand delta-like 1 homologue.Pref-1, a gatekeeper of adipogenesis The pancreatic β cell and type 1 diabetes: innocent bystander or active participant?Fetus-derived DLK1 is required for maternal metabolic adaptations to pregnancy and is associated with fetal growth restriction What does genetics tell us about imprinting and the placenta connection?Reduced salivary gland size and increased presence of epithelial progenitor cells in DLK1-deficient mice.Gene expression and epigenetic aberrations in F1-placentas fathered by obese males.Dlk1 promotes a fast motor neuron biophysical signature required for peak force execution.Intrauterine hyperglycemia induces intergenerational Dlk1-Gtl2 methylation changes in mouse placenta.

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Conditional deletions refine the embryonic requirement for Dlk1.

http://www.wikidata.org/entity/Q36605372

description

2012 nî lūn-bûn

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2012年の論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年论文

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2012年论文

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2012年论文

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name

Conditional deletions refine the embryonic requirement for Dlk1.

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Conditional deletions refine the embryonic requirement for Dlk1.

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Conditional deletions refine the embryonic requirement for Dlk1.

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Conditional deletions refine the embryonic requirement for Dlk1.

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Conditional deletions refine the embryonic requirement for Dlk1.

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Conditional deletions refine the embryonic requirement for Dlk1.

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J.MOD.2012.09.010

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Mechanisms of Development

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Conditional deletions refine the embryonic requirement for Dlk1.

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Aleksey Yevtodiyenko

http://www.w3.org/2001/XMLSchema#string

Hilmarie Muniz-Talavera

http://www.w3.org/2001/XMLSchema#string

Jennifer V Schmidt

http://www.w3.org/2001/XMLSchema#string

Oliver K Appelbe

http://www.w3.org/2001/XMLSchema#string

P2860

Nonsense mutation in the human growth hormone-releasing hormone receptor causes growth failure analogous to the little (lit) mouse

Protein structure of fetal antigen 1 (FA1). A novel circulating human epidermal-growth-factor-like protein expressed in neuroendocrine tumors and its relation to the gene products of dlk and pG2

A mammalian model for Laron syndrome produced by targeted disruption of the mouse growth hormone receptor/binding protein gene (the Laron mouse)

Gene dosage effects of the imprinted delta-like homologue 1 (dlk1/pref1) in development: implications for the evolution of imprinting

Generalized lacZ expression with the ROSA26 Cre reporter strain

Regulation of human skeletal stem cells differentiation by Dlk1/Pref-1

Pref-1, a protein containing EGF-like repeats, inhibits adipocyte differentiation

Studies on the isolation, structural analysis and tissue localization of fetal antigen 1 and its relation to a human adrenal-specific cDNA, pG2

FA1 immunoreactivity in endocrine tumours and during development of the human fetal pancreas; negative correlation with glucagon expression

Dual roles for glucokinase in glucose homeostasis as determined by liver and pancreatic beta cell-specific gene knock-outs using Cre recombinase

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143-159

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scholarly article

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10.1016/J.MOD.2012.09.010

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2-3

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130

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2012-10-08T00:00:00Z

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232235460

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23059197

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3570732

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