Comparative analysis of four oxidized guanine lesions from reactions of DNA with peroxynitrite, singlet oxygen, and γ-radiation
about
Reconciliation of chemical, enzymatic, spectroscopic and computational data to assign the absolute configuration of the DNA base lesion spiroiminodihydantoin.An automated Fpg-based FADU method for the detection of oxidative DNA lesions and screening of antioxidantsChemistry meets biology in colitis-associated carcinogenesis5-Carboxamido-5-formamido-2-iminohydantoin, in Addition to 8-oxo-7,8-Dihydroguanine, Is the Major Product of the Iron-Fenton or X-ray Radiation-Induced Oxidation of Guanine under Aerobic Reducing Conditions in Nucleoside and DNA ContextsBase and Nucleotide Excision Repair of Oxidatively Generated Guanine Lesions in DNA.An ameliorative protocol for the quantification of purine 5',8-cyclo-2'-deoxynucleosides in oxidized DNA.Interaction of Iron II Complexes with B-DNA. Insights from Molecular Modeling, Spectroscopy, and Cellular Biology.Guanine oxidation product 5-carboxamido-5-formamido-2-iminohydantoin induces mutations when bypassed by DNA polymerases and is a substrate for base excision repair.Klenow Fragment Discriminates against the Incorporation of the Hyperoxidized dGTP Lesion Spiroiminodihydantoin into DNA.Voltammetric microwell array for oxidized guanosine in intact ds-DNAMechanisms of base substitution mutagenesis in cancer genomes.Occurrence, Biological Consequences, and Human Health Relevance of Oxidative Stress-Induced DNA Damage.Time-course and intensity-based classifications of oxidative stresses and their potential application in biomedical, comparative and environmental research.The Nonbulky DNA Lesions Spiroiminodihydantoin and 5-Guanidinohydantoin Significantly Block Human RNA Polymerase II Elongation in Vitro.Excessive Reactive Oxygen Species and Exotic DNA Lesions as an Exploitable LiabilityRemoval of oxidatively generated DNA damage by overlapping repair pathways.Formation and processing of DNA damage substrates for the hNEIL enzymes.Rates of chemical cleavage of DNA and RNA oligomers containing guanine oxidation products.G-quadruplex folds of the human telomere sequence alter the site reactivity and reaction pathway of guanine oxidation compared to duplex DNA.Self-Cyclizing Antioxidants to Prevent DNA Damage Caused by Hydroxyl Radical.Cirrhotic patients with minimal hepatic encephalopathy have increased capacity to eliminate superoxide and peroxynitrite in lymphocytes, associated with cognitive impairment.
P2860
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P2860
Comparative analysis of four oxidized guanine lesions from reactions of DNA with peroxynitrite, singlet oxygen, and γ-radiation
description
2013 nî lūn-bûn
@nan
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
2013年论文
@zh
2013年论文
@zh-cn
name
Comparative analysis of four o ...... inglet oxygen, and γ-radiation
@ast
Comparative analysis of four o ...... inglet oxygen, and γ-radiation
@en
type
label
Comparative analysis of four o ...... inglet oxygen, and γ-radiation
@ast
Comparative analysis of four o ...... inglet oxygen, and γ-radiation
@en
prefLabel
Comparative analysis of four o ...... inglet oxygen, and γ-radiation
@ast
Comparative analysis of four o ...... inglet oxygen, and γ-radiation
@en
P2093
P2860
P356
P1476
Comparative analysis of four o ...... inglet oxygen, and γ-radiation
@en
P2093
John S Wishnok
Koli Taghizadeh
Steven R Tannenbaum
P2860
P304
P356
10.1021/TX300294D
P577
2013-01-24T00:00:00Z