Microglial infection by a neurovirulent murine retrovirus results in defective processing of envelope protein and intracellular budding of virus particles.
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Endoplasmic Reticulum Stress Is a Determinant of Retrovirus-Induced Spongiform NeurodegenerationPostinhibitory rebound neurons and networks are disrupted in retrovirus-induced spongiform neurodegeneration.Retrotransposition of nonviral RNAs in an avian packaging cell lineNeurovirulence in feline immunodeficiency virus-infected neonatal cats is viral strain specific and dependent on systemic immune suppression.Differential glycosylation of the Cas-Br-E env protein is associated with retrovirus-induced spongiform neurodegenerationGene expression profiling of microglia infected by a highly neurovirulent murine leukemia virus: implications for neuropathogenesisRetrovirus-induced spongiform neurodegeneration is mediated by unique central nervous system viral targeting and expression of env alonePulse labeling and long-term tracing of newborn neurons in the adult subgranular zone.Disparate regions of envelope protein regulate syncytium formation versus spongiform encephalopathy in neurological disease induced by murine leukemia virus TR.Transfer of neuropathogenic simian immunodeficiency virus with naturally infected microglia.Infection of central nervous system cells by ecotropic murine leukemia virus in C58 and AKR mice and in in utero-infected CE/J mice predisposes mice to paralytic infection by lactate dehydrogenase-elevating virus.Induction of focal spongiform neurodegeneration in developmentally resistant mice by implantation of murine retrovirus-infected microglia.Inhibition of murine retrovirus-induced neurodegeneration in the spinal cord by explant culture.A mechanism of restricted human immunodeficiency virus type 1 expression in human glial cells.Late virus replication events in microglia are required for neurovirulent retrovirus-induced spongiform neurodegeneration: evidence from neural progenitor-derived chimeric mouse brainsExpression of murine leukemia virus envelope protein is sufficient for the induction of apoptosisUnique N-linked glycosylation of CasBrE Env influences its stability, processing, and viral infectivity but not its neurotoxicity.Mink epithelial cell killing by pathogenic murine leukemia viruses involves endoplasmic reticulum stress.Abundant defective viral particles budding from microglia in the course of retroviral spongiform encephalopathy.Neural stem cells as engraftable packaging lines can mediate gene delivery to microglia: evidence from studying retroviral env-related neurodegeneration.Misfolding of CasBrE SU is reversed by interactions with 4070A Env: implications for gammaretroviral neuropathogenesis.Rebound from Inhibition: Self-Correction against Neurodegeneration?
P2860
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P2860
Microglial infection by a neurovirulent murine retrovirus results in defective processing of envelope protein and intracellular budding of virus particles.
description
1994 nî lūn-bûn
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1994年の論文
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1994年論文
@yue
1994年論文
@zh-hant
1994年論文
@zh-hk
1994年論文
@zh-mo
1994年論文
@zh-tw
1994年论文
@wuu
1994年论文
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1994年论文
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name
Microglial infection by a neur ...... ar budding of virus particles.
@ast
Microglial infection by a neur ...... ar budding of virus particles.
@en
type
label
Microglial infection by a neur ...... ar budding of virus particles.
@ast
Microglial infection by a neur ...... ar budding of virus particles.
@en
prefLabel
Microglial infection by a neur ...... ar budding of virus particles.
@ast
Microglial infection by a neur ...... ar budding of virus particles.
@en
P2093
P2860
P1433
P1476
Microglial infection by a neur ...... lar budding of virus particles
@en
P2093
P2860
P304
P407
P577
1994-05-01T00:00:00Z