Inducible nitric oxide synthase deficiency protects the heart from systolic overload-induced ventricular hypertrophy and congestive heart failure.
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Altered Nitric Oxide System in Cardiovascular and Renal DiseasesNitric oxide synthases, S-nitrosylation and cardiovascular health: from molecular mechanisms to therapeutic opportunities (review)Cardiac-targeted expression of soluble fas attenuates doxorubicin-induced cardiotoxicity in miceExercise training inducibility of MnSOD protein expression and activity is retained while reducing prooxidant signaling in the heart of senescent ratsCross-talk between lipid and protein carbonylation in a dynamic cardiomyocyte model of mild nitroxidative stressSepiapterin decreases acute rejection and apoptosis in cardiac transplants independently of changes in nitric oxide and inducible nitric-oxide synthase dimerization.Traumatic brain injury disrupts cerebrovascular tone through endothelial inducible nitric oxide synthase expression and nitric oxide gain of functionAdenosine A3 receptor deficiency exerts unanticipated protective effects on the pressure-overloaded left ventricleVascular endothelial-specific dimethylarginine dimethylaminohydrolase-1-deficient mice reveal that vascular endothelium plays an important role in removing asymmetric dimethylarginine.Protective effect of resveratrol against pressure overload-induced heart failure.Disruption of sarcolemmal ATP-sensitive potassium channel activity impairs the cardiac response to systolic overload.Ablation of iNOS delays cardiac contractile dysfunction in chronic hypertensionThe role of TLR4 in photoreceptor {alpha}a crystallin upregulation during early experimental autoimmune uveitisHydrogen sulfide anion regulates redox signaling via electrophile sulfhydrationPeroxynitrite Increases Protein Phosphatase Activity and Promotes the Interaction of Phospholamban with Protein Phosphatase 2a in the Myocardium.TRPV1 Activation Attenuates High-Salt Diet-Induced Cardiac Hypertrophy and Fibrosis through PPAR-δ Upregulation.Attenuated cardiovascular hypertrophy and oxidant generation in response to angiotensin II infusion in glutaredoxin-1 knockout mice.Oxidative stress regulates left ventricular PDE5 expression in the failing heartRedox proteomics identification of oxidatively modified myocardial proteins in human heart failure: implications for protein function.mTOR attenuates the inflammatory response in cardiomyocytes and prevents cardiac dysfunction in pathological hypertrophy.Regulation of Inducible Nitric Oxide Synthase (iNOS) and its Potential Role in Insulin Resistance, Diabetes and Heart FailureThe ameliorating effects of long-term electroacupuncture on cardiovascular remodeling in spontaneously hypertensive rats.Reactive oxygen species in cardiovascular disease.Interaction between connexin 43 and nitric oxide synthase in mice heart mitochondria.Nitric oxide signaling and the regulation of myocardial function.Myocardial ischaemia inhibits mitochondrial metabolism of 4-hydroxy-trans-2-nonenal.Modulation of myocardial contraction by peroxynitriteExtracellular superoxide dismutase protects the heart against oxidative stress and hypertrophy after myocardial infarction.Ecto-5'-nucleotidase deficiency exacerbates pressure-overload-induced left ventricular hypertrophy and dysfunction.Extracellular superoxide dismutase deficiency exacerbates pressure overload-induced left ventricular hypertrophy and dysfunctionRegulation of DDAH1 as a Potential Therapeutic Target for Treating Cardiovascular Diseases.Xanthine oxidase inhibition with febuxostat attenuates systolic overload-induced left ventricular hypertrophy and dysfunction in miceDeletion of inducible nitric oxide synthase provides cardioprotection in mice with 2-kidney, 1-clip hypertension.NADPH oxidase contributes to coronary endothelial dysfunction in the failing heartProtein Inhibitor of NOS1 Plays a Central Role in the Regulation of NOS1 Activity in Human Dilated Hearts.Targeting of phospholamban by peroxynitrite decreases beta-adrenergic stimulation in cardiomyocytesEffect of asymmetric dimethylarginine (ADMA) on heart failure development.Adenosine regulation of microtubule dynamics in cardiac hypertrophyPressure overload-induced alterations in fibrillar collagen content and myocardial diastolic function: role of secreted protein acidic and rich in cysteine (SPARC) in post-synthetic procollagen processing.Potential novel pharmacological therapies for myocardial remodelling.
P2860
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P2860
Inducible nitric oxide synthase deficiency protects the heart from systolic overload-induced ventricular hypertrophy and congestive heart failure.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
Inducible nitric oxide synthas ...... and congestive heart failure.
@ast
Inducible nitric oxide synthas ...... and congestive heart failure.
@en
type
label
Inducible nitric oxide synthas ...... and congestive heart failure.
@ast
Inducible nitric oxide synthas ...... and congestive heart failure.
@en
prefLabel
Inducible nitric oxide synthas ...... and congestive heart failure.
@ast
Inducible nitric oxide synthas ...... and congestive heart failure.
@en
P2093
P2860
P1433
P1476
Inducible nitric oxide synthas ...... y and congestive heart failure
@en
P2093
Elza D van Deel
Guangshuo Zhu
Ping Zhang
P2860
P304
P356
10.1161/01.RES.0000264081.78659.45
P50
P577
2007-03-15T00:00:00Z