Genetic deletion or pharmacological inhibition of cyclooxygenase-1 attenuate lipopolysaccharide-induced inflammatory response and brain injury.
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Enzymes of the cyclooxygenase pathways of prostanoid biosynthesisNon-steroidal anti-inflammatory drugs and cognitive function: are prostaglandins at the heart of cognitive impairment in dementia and delirium?Reciprocal regulation of the nitric oxide and cyclooxygenase pathway in pathophysiology: relevance and clinical implicationsChemical dampening of Ly6C(hi) monocytes in the periphery produces anti-depressant effects in mice.Astrocytic expression of Parkinson's disease-related A53T alpha-synuclein causes neurodegeneration in mice.Characterization of inflamin, the first member of a new family of snake venom proteins that induces inflammation.Effects of genetic deficiency of cyclooxygenase-1 or cyclooxygenase-2 on functional and histological outcomes following traumatic brain injury in mice.Cyclooxygenase-1 null mice show reduced neuroinflammation in response to beta-amyloid.A role for cyclooxygenase-1 in beta-amyloid-induced neuroinflammation.Cyclooxygenase-1 and -2 differentially modulate lipopolysaccharide-induced blood-brain barrier disruption through matrix metalloproteinase activityUnesterified docosahexaenoic acid is protective in neuroinflammation.Cyclooxygenase-1 orchestrates germinal center formation and antibody class-switch via regulation of IL-17Neuronal overexpression of cyclooxygenase-2 does not alter the neuroinflammatory response during brain innate immune activation.Cyclooxygenases: structural and functional insights.Purinergic signaling induces cyclooxygenase-1-dependent prostanoid synthesis in microglia: roles in the outcome of excitotoxic brain injury.Induction of COX-2 enzyme and down-regulation of COX-1 expression by lipopolysaccharide (LPS) control prostaglandin E2 production in astrocytesEffectiveness and tolerance of anti-inflammatory drugs' add-on therapy in major mental disorders: a systematic qualitative review.Targeting cyclooxygenases-1 and -2 in neuroinflammation: Therapeutic implications.Role of the cyclooxygenase pathway in chemotherapy-induced oral mucositis: a pilot studyCOX1 and COX2 expression in non-neuronal cellular compartments of the rhesus macaque brain during lentiviral infection.NMDA-induced seizure intensity is enhanced in COX-2 deficient mice.The effect of non-steroidal anti-inflammatory agents on behavioural changes and cytokine production following systemic inflammation: Implications for a role of COX-1Time-dependent changes in the brain arachidonic acid cascade during cuprizone-induced demyelination and remyelination.Inhibition of 5-lipoxygenase activity in mice during cuprizone-induced demyelination attenuates neuroinflammation, motor dysfunction and axonal damageThe role of oxidative stress and NADPH oxidase in cerebrovascular disease.Piroxicam reverses endotoxin-induced hypotension in rats: contribution of vasoactive eicosanoids and nitric oxideCyclooxygenase-1 is involved in the inhibition of hippocampal neurogenesis after lipopolysaccharide-induced neuroinflammationThe role of nitric oxide in prostaglandin biology; update.Endocannabinoid hydrolysis generates brain prostaglandins that promote neuroinflammation.Effects of betaine on lipopolysaccharide-induced memory impairment in mice and the involvement of GABA transporter 2.Cerebrovascular cyclooxygenase-1 expression, regulation, and role in hypothalamic-pituitary-adrenal axis activation by inflammatory stimuli.The distinct roles of cyclooxygenase-1 and -2 in neuroinflammation: implications for translational research.Cyclooxygenases-1 and -2 differentially modulate leukocyte recruitment into the inflamed brain.Inhibition of NADPH oxidase promotes alternative and anti-inflammatory microglial activation during neuroinflammationDeletion of Monoglyceride Lipase in Astrocytes Attenuates Lipopolysaccharide-induced NeuroinflammationImmune-neural connections: how the immune system's response to infectious agents influences behaviorRapid and profound rewiring of brain lipid signaling networks by acute diacylglycerol lipase inhibition.Neuroinflammatory response to lipopolysaccharide is exacerbated in mice genetically deficient in cyclooxygenase-2.Cyclooxygenase-1 inhibition reduces amyloid pathology and improves memory deficits in a mouse model of Alzheimer's disease.Ginsenoside rg3 alleviates lipopolysaccharide-induced learning and memory impairments by anti-inflammatory activity in rats
P2860
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P2860
Genetic deletion or pharmacological inhibition of cyclooxygenase-1 attenuate lipopolysaccharide-induced inflammatory response and brain injury.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
Genetic deletion or pharmacolo ...... ory response and brain injury.
@ast
Genetic deletion or pharmacolo ...... ory response and brain injury.
@en
type
label
Genetic deletion or pharmacolo ...... ory response and brain injury.
@ast
Genetic deletion or pharmacolo ...... ory response and brain injury.
@en
prefLabel
Genetic deletion or pharmacolo ...... ory response and brain injury.
@ast
Genetic deletion or pharmacolo ...... ory response and brain injury.
@en
P2093
P2860
P356
P1433
P1476
Genetic deletion or pharmacolo ...... ory response and brain injury.
@en
P2093
Francesca Bosetti
Robert Langenbach
Sang-Ho Choi
P2860
P304
P356
10.1096/FJ.07-9411COM
P407
P577
2007-12-27T00:00:00Z