T-bet deficiency attenuates renal injury in experimental crescentic glomerulonephritis.
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CD4+ T Cell Fate in Glomerulonephritis: A Tale of Th1, Th17, and Novel Treg SubtypesThe role of the Th1 transcription factor T-bet in a mouse model of immune-mediated bone-marrow failureToll-like receptor 9 enhances nephritogenic immunity and glomerular leukocyte recruitment, exacerbating experimental crescentic glomerulonephritis.The Th17-defining transcription factor RORγt promotes glomerulonephritisThe IL-27 receptor has biphasic effects in crescentic glomerulonephritis mediated through Th1 responses.Interleukin-17A promotes early but attenuates established disease in crescentic glomerulonephritis in mice.Local IL-17 Production Exerts a Protective Role in Murine Experimental Glomerulonephritis.IL-17 Expression in the Time Course of Acute Anti-Thy1 Glomerulonephritis.A novel role for type 1 angiotensin receptors on T lymphocytes to limit target organ damage in hypertension.AKAP9 regulates activation-induced retention of T lymphocytes at sites of inflammationCXCR3+ Regulatory T Cells Control TH1 Responses in Crescentic GN.Regulation of pathogenic Th17 cell differentiation by IL-10 in the development of glomerulonephritis.IL-23, not IL-12, directs autoimmunity to the Goodpasture antigen.Inhibition of zymosan-induced kidney dysfunction by tyrphostin AG-490.T-bet is essential for encephalitogenicity of both Th1 and Th17 cellsMicroRNA-155 drives TH17 immune response and tissue injury in experimental crescentic GN.Th1 and Th17 cells induce proliferative glomerulonephritis.Advances in human antiglomerular basement membrane disease.Update on crescentic glomerulonephritis.Gene polymorphisms of interleukin-17 and interleukin-17 receptor are associated with end-stage kidney disease.Cytokines: Names and Numbers You Should Care About.T-Bet Enhances Regulatory T Cell Fitness and Directs Control of Th1 Responses in Crescentic GN.Tim3 is upregulated and protective in nephrotoxic serum nephritis.Galectin-9 ameliorates anti-GBM glomerulonephritis by inhibiting Th1 and Th17 immune responses in mice.Signal transducer and activation of transcription 6 (STAT6) regulates T helper type 1 (Th1) and Th17 nephritogenic immunity in experimental crescentic glomerulonephritis.Early life stress induces immune priming in kidneys of adult male rats.The pathological role of IL-18Rα in renal ischemia/reperfusion injury.
P2860
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P2860
T-bet deficiency attenuates renal injury in experimental crescentic glomerulonephritis.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
2008年论文
@zh
2008年论文
@zh-cn
name
T-bet deficiency attenuates renal injury in experimental crescentic glomerulonephritis.
@ast
T-bet deficiency attenuates renal injury in experimental crescentic glomerulonephritis.
@en
type
label
T-bet deficiency attenuates renal injury in experimental crescentic glomerulonephritis.
@ast
T-bet deficiency attenuates renal injury in experimental crescentic glomerulonephritis.
@en
prefLabel
T-bet deficiency attenuates renal injury in experimental crescentic glomerulonephritis.
@ast
T-bet deficiency attenuates renal injury in experimental crescentic glomerulonephritis.
@en
P2093
P2860
P356
P1476
T-bet deficiency attenuates renal injury in experimental crescentic glomerulonephritis
@en
P2093
Dragana Odobasic
Lynelle K Jones
Richard K S Phoon
Stephen R Holdsworth
Timothy J Semple
P2860
P304
P356
10.1681/ASN.2007030392
P577
2008-01-30T00:00:00Z