T-bet deficiency attenuates renal injury in experimental crescentic glomerulonephritis. - Wikidata - wikimedia - TriplyDB

T-bet deficiency attenuates renal injury in experimental crescentic glomerulonephritis.

T-bet deficiency attenuates renal injury in experimental crescentic glomerulonephritis.

http://www.wikidata.org/entity/Q36667205

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CD4+ T Cell Fate in Glomerulonephritis: A Tale of Th1, Th17, and Novel Treg Subtypes The role of the Th1 transcription factor T-bet in a mouse model of immune-mediated bone-marrow failure Toll-like receptor 9 enhances nephritogenic immunity and glomerular leukocyte recruitment, exacerbating experimental crescentic glomerulonephritis.The Th17-defining transcription factor RORγt promotes glomerulonephritis The IL-27 receptor has biphasic effects in crescentic glomerulonephritis mediated through Th1 responses.Interleukin-17A promotes early but attenuates established disease in crescentic glomerulonephritis in mice.Local IL-17 Production Exerts a Protective Role in Murine Experimental Glomerulonephritis.IL-17 Expression in the Time Course of Acute Anti-Thy1 Glomerulonephritis.A novel role for type 1 angiotensin receptors on T lymphocytes to limit target organ damage in hypertension.AKAP9 regulates activation-induced retention of T lymphocytes at sites of inflammation CXCR3+ Regulatory T Cells Control TH1 Responses in Crescentic GN.Regulation of pathogenic Th17 cell differentiation by IL-10 in the development of glomerulonephritis.IL-23, not IL-12, directs autoimmunity to the Goodpasture antigen.Inhibition of zymosan-induced kidney dysfunction by tyrphostin AG-490.T-bet is essential for encephalitogenicity of both Th1 and Th17 cells MicroRNA-155 drives TH17 immune response and tissue injury in experimental crescentic GN.Th1 and Th17 cells induce proliferative glomerulonephritis.Advances in human antiglomerular basement membrane disease.Update on crescentic glomerulonephritis.Gene polymorphisms of interleukin-17 and interleukin-17 receptor are associated with end-stage kidney disease.Cytokines: Names and Numbers You Should Care About.T-Bet Enhances Regulatory T Cell Fitness and Directs Control of Th1 Responses in Crescentic GN.Tim3 is upregulated and protective in nephrotoxic serum nephritis.Galectin-9 ameliorates anti-GBM glomerulonephritis by inhibiting Th1 and Th17 immune responses in mice.Signal transducer and activation of transcription 6 (STAT6) regulates T helper type 1 (Th1) and Th17 nephritogenic immunity in experimental crescentic glomerulonephritis.Early life stress induces immune priming in kidneys of adult male rats.The pathological role of IL-18Rα in renal ischemia/reperfusion injury.

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P2860

T-bet deficiency attenuates renal injury in experimental crescentic glomerulonephritis.

http://www.wikidata.org/entity/Q36667205

description

2008 nî lūn-bûn

@nan

2008年の論文

@ja

2008年論文

@yue

2008年論文

@zh-hant

2008年論文

@zh-hk

2008年論文

@zh-mo

2008年論文

@zh-tw

2008年论文

@wuu

2008年论文

@zh

2008年论文

@zh-cn

name

T-bet deficiency attenuates renal injury in experimental crescentic glomerulonephritis.

@ast

T-bet deficiency attenuates renal injury in experimental crescentic glomerulonephritis.

@en

type

label

T-bet deficiency attenuates renal injury in experimental crescentic glomerulonephritis.

@ast

T-bet deficiency attenuates renal injury in experimental crescentic glomerulonephritis.

@en

prefLabel

T-bet deficiency attenuates renal injury in experimental crescentic glomerulonephritis.

@ast

T-bet deficiency attenuates renal injury in experimental crescentic glomerulonephritis.

@en

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P1433

Journal of the American Society of Nephrology

P1476

T-bet deficiency attenuates renal injury in experimental crescentic glomerulonephritis

@en

P2093

Dragana Odobasic

http://www.w3.org/2001/XMLSchema#string

Lynelle K Jones

http://www.w3.org/2001/XMLSchema#string

Richard K S Phoon

http://www.w3.org/2001/XMLSchema#string

Stephen R Holdsworth

http://www.w3.org/2001/XMLSchema#string

Timothy J Semple

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P2860

A novel transcription factor, T-bet, directs Th1 lineage commitment

Cutting edge: role of IL-27/WSX-1 signaling for induction of T-bet through activation of STAT1 during initial Th1 commitment

IL-21 up-regulates the expression of genes associated with innate immunity and Th1 response

The role of T-cell interleukin-17 in conducting destructive arthritis: lessons from animal models.

Primer3 on the WWW for general users and for biologist programmers

T-bet regulates IgG class switching and pathogenic autoantibody production

IL-23 drives a pathogenic T cell population that induces autoimmune inflammation

Two types of murine helper T cell clone. I. Definition according to profiles of lymphokine activities and secreted proteins

Interleukin-23 rather than interleukin-12 is the critical cytokine for autoimmune inflammation of the brain

The lineage decisions of helper T cells

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477-485

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scholarly article

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10.1681/ASN.2007030392

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3

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19

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A Richard Kitching

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2008-01-30T00:00:00Z

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18235099

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glomerulonephritis

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2391047

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