Regulation of Ca2+ and Na+ in normal and failing cardiac myocytes. - Wikidata - wikimedia - TriplyDB

Regulation of Ca2+ and Na+ in normal and failing cardiac myocytes.

Regulation of Ca2+ and Na+ in normal and failing cardiac myocytes.

http://www.wikidata.org/entity/Q36667318

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The late Na+ current--origin and pathophysiological relevance EH-myomesin splice isoform is a novel marker for dilated cardiomyopathy The Effect of a Novel Highly Selective Inhibitor of the Sodium/Calcium Exchanger (NCX) on Cardiac Arrhythmias in In Vitro and In Vivo Experiments Perinatal loss of Nkx2-5 results in rapid conduction and contraction defects Simulation and mechanistic investigation of the arrhythmogenic role of the late sodium current in human heart failure Phospholemman is a negative feed-forward regulator of Ca2+ in β-adrenergic signaling, accelerating β-adrenergic inotropy.Inhibitory effect of ETB receptor on Na(+)-K(+) ATPase activity by extracellular Ca(2+) entry and Ca(2+) release from the endoplasmic reticulum in renal proximal tubule cells.Basal and β-adrenergic regulation of the cardiac calcium channel CaV1.2 requires phosphorylation of serine 1700 Artificial selection for whole animal low intrinsic aerobic capacity co-segregates with hypoxia-induced cardiac pump failure.Role and possible mechanisms of clenbuterol in enhancing reverse remodelling during mechanical unloading in murine heart failure Targeting device therapy: genomics of sudden death.Tight coupling of Na+/K+-ATPase with glycolysis demonstrated in permeabilized rat cardiomyocytes.There goes the neighborhood: pathological alterations in T-tubule morphology and consequences for cardiomyocyte Ca2+ handling.Control of Ca2+ release by action potential configuration in normal and failing murine cardiomyocytes Depolarization of cardiac membrane potential synchronizes calcium sparks and waves in tissue Two close, too close: sarcoplasmic reticulum-mitochondrial crosstalk and cardiomyocyte fate Disease-related cardiac troponins alter thin filament Ca2+ association and dissociation rates.Succinate causes pathological cardiomyocyte hypertrophy through GPR91 activation.Deciphering Arrhythmia Mechanisms - Tools of the Trade Electrophysiological and structural remodeling in heart failure modulate arrhythmogenesis. 1D simulation study.Phospholemman expression is high in the newborn rabbit heart and declines with postnatal maturation Targeting cardiomyocyte Ca2+ homeostasis in heart failure.Instabilities of the resting state in a mathematical model of calcium handling in cardiac myocytes.Dantrolene prevents arrhythmogenic Ca2+ release in heart failure Enzyme localization, crowding, and buffers collectively modulate diffusion-influenced signal transduction: Insights from continuum diffusion modeling.Cardiac arrhythmia mechanisms in rats with heart failure induced by pulmonary hypertension.Na+/Ca2+ exchanger-1 protects against systolic failure in the Akitains2 model of diabetic cardiomyopathy via a CXCR4/NF-κB pathway.Reduced response to IKr blockade and altered hERG1a/1b stoichiometry in human heart failure Late sodium current in failing heart: friend or foe?Synchrony of cardiomyocyte Ca(2+) release is controlled by T-tubule organization, SR Ca(2+) content, and ryanodine receptor Ca(2+) sensitivity.Late sodium current is a new therapeutic target to improve contractility and rhythm in failing heart.Exercise training reverses myocardial dysfunction induced by CaMKIIδC overexpression by restoring Ca2+ homeostasis.Bendavia restores mitochondrial energy metabolism gene expression and suppresses cardiac fibrosis in the border zone of the infarcted heart.Asymmetric methyl group labeling as a probe of membrane protein homo-oligomers by NMR spectroscopy Designing heart performance by gene transfer.High inborn aerobic capacity does not protect the heart following myocardial infarction.Reduced aerobic capacity causes leaky ryanodine receptors that trigger arrhythmia in a rat strain artificially selected and bred for low aerobic running capacity.Optical imaging of voltage and calcium in cardiac cells & tissues Redox regulation of sodium and calcium handling No rest for the weary: diastolic calcium homeostasis in the normal and failing myocardium.

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P2860

Regulation of Ca2+ and Na+ in normal and failing cardiac myocytes.

http://www.wikidata.org/entity/Q36667318

description

2006 nî lūn-bûn

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2006年の論文

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2006年論文

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2006年論文

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2006年論文

@zh-hk

2006年論文

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2006年論文

@zh-tw

2006年论文

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2006年论文

@zh

2006年论文

@zh-cn

name

Regulation of Ca2+ and Na+ in normal and failing cardiac myocytes.

@ast

Regulation of Ca2+ and Na+ in normal and failing cardiac myocytes.

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type

label

Regulation of Ca2+ and Na+ in normal and failing cardiac myocytes.

@ast

Regulation of Ca2+ and Na+ in normal and failing cardiac myocytes.

@en

prefLabel

Regulation of Ca2+ and Na+ in normal and failing cardiac myocytes.

@ast

Regulation of Ca2+ and Na+ in normal and failing cardiac myocytes.

@en

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P356

ANNALS.1380.015

P1433

Annals of the New York Academy of Sciences

P1476

Regulation of Ca2+ and Na+ in normal and failing cardiac myocytes.

@en

P2093

Donald M Bers

http://www.w3.org/2001/XMLSchema#string

Julie Bossuyt

http://www.w3.org/2001/XMLSchema#string

Sanda Despa

http://www.w3.org/2001/XMLSchema#string

P2860

PKA phosphorylation dissociates FKBP12.6 from the calcium release channel (ryanodine receptor): defective regulation in failing hearts

Transgenic CaMKIIdeltaC overexpression uniquely alters cardiac myocyte Ca2+ handling: reduced SR Ca2+ load and activated SR Ca2+ release

The deltaC isoform of CaMKII is activated in cardiac hypertrophy and induces dilated cardiomyopathy and heart failure

Phosphodiesterase 4D deficiency in the ryanodine-receptor complex promotes heart failure and arrhythmias

The FXYD gene family of small ion transport regulators or channels: cDNA sequence, protein signature sequence, and expression

FXYD3 (Mat-8), a new regulator of Na,K-ATPase

Differential regulation of renal Na,K-ATPase by splice variants of the gamma subunit

Phospholemman overexpression inhibits Na+-K+-ATPase in adult rat cardiac myocytes: relevance to decreased Na+ pump activity in postinfarction myocytes.

Phospholemman (FXYD1) associates with Na,K-ATPase and regulates its transport properties.

Ryanodine receptor/calcium release channel PKA phosphorylation: a critical mediator of heart failure progression.

P304

165-177

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scholarly article

P356

10.1196/ANNALS.1380.015

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P407

P478

1080

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P577

2006-10-01T00:00:00Z

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P698

17132783

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