Simulation and mechanistic investigation of the arrhythmogenic role of the late sodium current in human heart failure - Wikidata - wikimedia - TriplyDB

Simulation and mechanistic investigation of the arrhythmogenic role of the late sodium current in human heart failure

Simulation and mechanistic investigation of the arrhythmogenic role of the late sodium current in human heart failure

http://www.wikidata.org/entity/Q28731387

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Electrophysiological and structural remodeling in heart failure modulate arrhythmogenesis. 2D simulation study Post-translational modifications of the cardiac Na channel: contribution of CaMKII-dependent phosphorylation to acquired arrhythmias mRNA expression levels in failing human hearts predict cellular electrophysiological remodeling: a population-based simulation study.Electrophysiological and structural remodeling in heart failure modulate arrhythmogenesis. 1D simulation study.Changes in Intracellular Na+ following Enhancement of Late Na+ Current in Virtual Human Ventricular Myocytes.A mathematical model for active contraction in healthy and failing myocytes and left ventricles.Reduced response to IKr blockade and altered hERG1a/1b stoichiometry in human heart failure The arrhythmogenic consequences of increasing late INa in the cardiomyocyte.Dynamics of the late Na(+) current during cardiac action potential and its contribution to afterdepolarizations.In silico assessment of drug safety in human heart applied to late sodium current blockers Na/K pump regulation of cardiac repolarization: insights from a systems biology approach.The late sodium current in heart failure: pathophysiology and clinical relevance.Carbon monoxide effects on human ventricle action potential assessed by mathematical simulations.Illuminating Myocyte-Fibroblast Homotypic and Heterotypic Gap Junction Dynamics Using Dynamic Clamp.An electromechanical left ventricular wedge model to study the effects of deformation on repolarization during heart failure.Abnormal calcium homeostasis in heart failure with preserved ejection fraction is related to both reduced contractile function and incomplete relaxation: an electromechanically detailed biophysical modeling study.Characterization of 2 genetic variants of Na(v) 1.5-arginine 689 found in patients with cardiac arrhythmias.Sensitivity analysis revealing the effect of modulating ionic mechanisms on calcium dynamics in simulated human heart failure.Sensitivity analysis of ventricular activation and electrocardiogram in tailored models of heart-failure patients.Ca Cycling Impairment in Heart Failure Is Exacerbated by Fibrosis: Insights Gained From Mechanistic Simulations

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Simulation and mechanistic investigation of the arrhythmogenic role of the late sodium current in human heart failure

http://www.wikidata.org/entity/Q28731387

description

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Simulation and mechanistic inv ...... current in human heart failure

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Simulation and mechanistic inv ...... current in human heart failure

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Simulation and mechanistic inv ...... current in human heart failure

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Simulation and mechanistic inv ...... current in human heart failure

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Simulation and mechanistic inv ...... current in human heart failure

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Simulation and mechanistic inv ...... current in human heart failure

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Juan F Gomez

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Karen Cardona

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Luiz Belardinelli

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Sridharan Rajamani

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P2860

Mechanistic insight into the functional and toxic effects of Strophanthidin in the failing human myocardium

Novel, ultraslow inactivating sodium current in human ventricular cardiomyocytes

Simulation of the undiseased human cardiac ventricular action potential: model formulation and experimental validation

A β(IV)-spectrin/CaMKII signaling complex is essential for membrane excitability in mice

Late sodium current contributes to the reverse rate-dependent effect of IKr inhibition on ventricular repolarization.

Transmural dispersion of repolarization in failing and nonfailing human ventricle

Electrophysiological remodeling in hypertrophy and heart failure.

Late sodium current contributes to diastolic cell Ca2+ accumulation in chronic heart failure.

Rate dependence of the effect of antiarrhythmic drugs delaying cardiac repolarization: an overview.

Action potential and contractility changes in [Na(+)](i) overloaded cardiac myocytes: a simulation study.

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Jose Maria Ferrero

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