Activation induces sensitivity toward APO-1 (CD95)-mediated apoptosis in human B cells.
about
A distinct plasmablast and naïve B-cell phenotype in primary immune thrombocytopenia.Phase I trial of weekly tigatuzumab, an agonistic humanized monoclonal antibody targeting death receptor 5 (DR5)Expression of Fas (CD95/APO-1) ligand by human breast cancers: significance for tumor immune privilegeMolecular mechanisms of irradiation-induced apoptosisExpression of Fas ligand by human gastric adenocarcinomas: a potential mechanism of immune escape in stomach cancerThe Fas/CD95 Receptor Regulates the Death of Autoreactive B Cells and the Selection of Antigen-Specific B Cells.CD40 ligation induces Apo-1/Fas expression on human B lymphocytes and facilitates apoptosis through the Apo-1/Fas pathway.Concurrent engagement of CD40 and the antigen receptor protects naive and memory human B cells from APO-1/Fas-mediated apoptosisThe Fas counterattack: Fas-mediated T cell killing by colon cancer cells expressing Fas ligandA disease-related rheumatoid factor autoantibody is not tolerized in a normal mouse: implications for the origins of autoantibodies in autoimmune diseaseAutoantigen-specific B cell activation in Fas-deficient rheumatoid factor immunoglobulin transgenic mice.Essential lymphocyte function associated 1 (LFA-1): intercellular adhesion molecule interactions for T cell-mediated B cell apoptosis by Fas/APO-1/CD95.The Fas counterattack: a molecular mechanism of tumor immune privilege.Quantitation of Fas and Fas ligand gene expression in human ovarian, cervical and endometrial carcinomas using real-time quantitative RT-PCR.Enhanced and accelerated lymphoproliferation in Fas-null mice.A Recombinant Bispecific CD20×CD95 Antibody With Superior Activity Against Normal and Malignant B-cells.Fas ligation induces apoptosis of CD40-activated human B lymphocytes.IL-7 sensitizes human pre-B cells but not pro-B cells to Fas/APO-1 (CD95)-mediated apoptosis.Paneth cells express high levels of CD95 ligand transcripts: a unique property among gastrointestinal epithelia.In situ studies of the primary immune response to (4-hydroxy-3-nitrophenyl)acetyl. IV. Affinity-dependent, antigen-driven B cell apoptosis in germinal centers as a mechanism for maintaining self-tolerance.
P2860
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P2860
Activation induces sensitivity toward APO-1 (CD95)-mediated apoptosis in human B cells.
description
1994 nî lūn-bûn
@nan
1994年の論文
@ja
1994年論文
@yue
1994年論文
@zh-hant
1994年論文
@zh-hk
1994年論文
@zh-mo
1994年論文
@zh-tw
1994年论文
@wuu
1994年论文
@zh
1994年论文
@zh-cn
name
Activation induces sensitivity toward APO-1 (CD95)-mediated apoptosis in human B cells.
@ast
Activation induces sensitivity toward APO-1 (CD95)-mediated apoptosis in human B cells.
@en
type
label
Activation induces sensitivity toward APO-1 (CD95)-mediated apoptosis in human B cells.
@ast
Activation induces sensitivity toward APO-1 (CD95)-mediated apoptosis in human B cells.
@en
prefLabel
Activation induces sensitivity toward APO-1 (CD95)-mediated apoptosis in human B cells.
@ast
Activation induces sensitivity toward APO-1 (CD95)-mediated apoptosis in human B cells.
@en
P1476
Activation induces sensitivity toward APO-1 (CD95)-mediated apoptosis in human B cells
@en
P2093
P304
P407
P577
1994-06-01T00:00:00Z