IL-17 disrupts corneal barrier following desiccating stress. - Wikidata - wikimedia - TriplyDB

IL-17 disrupts corneal barrier following desiccating stress.

IL-17 disrupts corneal barrier following desiccating stress.

http://www.wikidata.org/entity/Q36676135

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sPLA2-IIa amplifies ocular surface inflammation in the experimental dry eye (DE) BALB/c mouse model Tear fluid biomarkers in ocular and systemic disease: potential use for predictive, preventive and personalised medicine Tear cytokine profile as a noninvasive biomarker of inflammation for ocular surface diseases: standard operating procedures Modulation of ocular surface glycocalyx barrier function by a galectin-3 N-terminal deletion mutant and membrane-anchored synthetic glycopolymers NK cells promote Th-17 mediated corneal barrier disruption in dry eye Effect of desiccating stress on mouse meibomian gland function.Dendritic cell-derived thrombospondin-1 is critical for the generation of the ocular surface Th17 response to desiccating stress.Goblet Cells Contribute to Ocular Surface Immune Tolerance-Implications for Dry Eye Disease.Subnormal cytokine profile in the tear fluid of keratoconus patients.Tear cytokine and chemokine analysis and clinical correlations in evaporative-type dry eye disease Lacrimal gland-derived IL-22 regulates IL-17-mediated ocular mucosal inflammation.Hyperosmolarity-induced AQP5 upregulation promotes inflammation and cell death via JNK1/2 Activation in human corneal epithelial cells Effect of pro-inflammatory mediators on membrane-associated mucins expressed by human ocular surface epithelial cells.Desiccating stress promotion of Th17 differentiation by ocular surface tissues through a dendritic cell-mediated pathway.Age-related T-cell cytokine profile parallels corneal disease severity in Sjogren's syndrome-like keratoconjunctivitis sicca in CD25KO mice Managing Sjögren's Syndrome and non-Sjögren Syndrome dry eye with anti-inflammatory therapy.Spontaneous autoimmune dacryoadenitis in aged CD25KO mice Disruption of TGF-β signaling improves ocular surface epithelial disease in experimental autoimmune keratoconjunctivitis sicca Topical interleukin 1 receptor antagonist for treatment of dry eye disease: a randomized clinical trial.CCR7 is critical for the induction and maintenance of Th17 immunity in dry eye disease.Corneal transplantation and immune privilege.Desiccating stress-induced chemokine expression in the epithelium is dependent on upregulation of NKG2D/RAE-1 and release of IFN-γ in experimental dry eye.The core mechanism of dry eye disease is inflammation.Dry eye as a mucosal autoimmune disease.Two different regulatory T cell populations that promote corneal allograft survival.Investigating the relationship between serum interleukin-17 levels and systemic immune-mediated disease in patients with dry eye syndrome Surfactant protein D contributes to ocular defense against Pseudomonas aeruginosa in a murine model of dry eye disease.Loss of NHE8 expression impairs ocular surface function in mice Decreased central corneal thickness in ankylosing spondylitis.Dynamic ocular surface and lacrimal gland changes induced in experimental murine dry eye.Interferon-γ-secreting NK cells promote induction of dry eye disease IL-17A-dependent CD4+CD25+ regulatory T cells promote immune privilege of corneal allografts Chemokine receptors CCR6 and CXCR3 are necessary for CD4(+) T cell mediated ocular surface disease in experimental dry eye disease.Interleukin-17 in various ocular surface inflammatory diseases IL-17 promotes immune privilege of corneal allografts Interferon-γ exacerbates dry eye-induced apoptosis in conjunctiva through dual apoptotic pathways.Desiccating stress induces CD4+ T-cell-mediated Sjögren's syndrome-like corneal epithelial apoptosis via activation of the extrinsic apoptotic pathway by interferon-γ Altered balance of interleukin-13/interferon-gamma contributes to lacrimal gland destruction and secretory dysfunction in CD25 knockout model of Sjögren's syndrome.A novel pro-lymphangiogenic function for Th17/IL-17.Cyclosporine ophthalmic emulsions for the treatment of dry eye: a review of the clinical evidence.

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IL-17 disrupts corneal barrier following desiccating stress.

http://www.wikidata.org/entity/Q36676135

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2009 nî lūn-bûn

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2009年の論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年论文

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2009年论文

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2009年论文

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IL-17 disrupts corneal barrier following desiccating stress.

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IL-17 disrupts corneal barrier following desiccating stress.

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IL-17 disrupts corneal barrier following desiccating stress.

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IL-17 disrupts corneal barrier following desiccating stress.

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IL-17 disrupts corneal barrier following desiccating stress.

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IL-17 disrupts corneal barrier following desiccating stress.

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Mucosal Immunology

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IL-17 disrupts corneal barrier following desiccating stress

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B Fang

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D-Q Li

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J D Pitcher

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J Y Niederkorn

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K F Siemasko

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M E Stern

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P Ma

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S B Pangelinan

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S C Pflugfelder

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S Chotikavanich

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P2860

A receptor for the heterodimeric cytokine IL-23 is composed of IL-12Rbeta1 and a novel cytokine receptor subunit, IL-23R

Interleukin (IL)-22 and IL-17 are coexpressed by Th17 cells and cooperatively enhance expression of antimicrobial peptides

Reciprocal developmental pathways for the generation of pathogenic effector TH17 and regulatory T cells

The orphan nuclear receptor RORgammat directs the differentiation program of proinflammatory IL-17+ T helper cells

Transforming growth factor-beta induces development of the T(H)17 lineage

IL-23 promotes tumour incidence and growth

TGFbeta in the context of an inflammatory cytokine milieu supports de novo differentiation of IL-17-producing T cells

A distinct lineage of CD4 T cells regulates tissue inflammation by producing interleukin 17

Development, cytokine profile and function of human interleukin 17-producing helper T cells

Interleukins 1beta and 6 but not transforming growth factor-beta are essential for the differentiation of interleukin 17-producing human T helper cells

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243-253

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10.1038/MI.2009.5

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3

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2

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Cintia Sade de Paiva

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2009-02-25T00:00:00Z

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24039733

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19242409

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3594767

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