Inhaled ultrafine particulate matter affects CNS inflammatory processes and may act via MAP kinase signaling pathways.
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Physical Activity, Air Pollution and the BrainTraffic-related air pollution and cognitive function in a cohort of older menSocial Isolation-Induced Territorial Aggression in Male Offspring Is Enhanced by Exposure to Diesel Exhaust during PregnancyLung injury after cigarette smoking is particle relatedInhaled nickel nanoparticles alter vascular reactivity in C57BL/6 miceNanoparticles and neurotoxicityNanoparticle Uptake: The Phagocyte ProblemNeurotoxicants are in the air: convergence of human, animal, and in vitro studies on the effects of air pollution on the brainComparative evaluation of the effects of short-term inhalation exposure to diesel engine exhaust on rat lung and brainIntranasal Chromium Induces Acute Brain and Lung Injuries in Rats: Assessment of Different Potential Hazardous Effects of Environmental and Occupational Exposure to Chromium and Introduction of a Novel Pharmacological and Toxicological Animal ModelParticulate matter beyond mass: recent health evidence on the role of fractions, chemical constituents and sources of emissionGlutamatergic neurons in rodent models respond to nanoscale particulate urban air pollutants in vivo and in vitroToxicity of inhaled particulate matter on the central nervous system: neuroinflammation, neuropsychological effects and neurodegenerative disease.Toll-like receptor 4 in glial inflammatory responses to air pollution in vitro and in vivo.Air pollution toxicology--a brief review of the role of the science in shaping the current understanding of air pollution health risks.A national case-crossover analysis of the short-term effect of PM2.5 on hospitalizations and mortality in subjects with diabetes and neurological disorders.Developmental exposure to concentrated ambient ultrafine particulate matter air pollution in mice results in persistent and sex-dependent behavioral neurotoxicity and glial activation.Effect of prolonged exposure to diesel engine exhaust on proinflammatory markers in different regions of the rat brainAssociations of PM10 with sleep and sleep-disordered breathing in adults from seven U.S. urban areasUrban air pollutants reduce synaptic function of CA1 neurons via an NMDA/NȮ pathway in vitroRisks from accidental exposures to engineered nanoparticles and neurological health effects: a critical review.Air pollution: mechanisms of neuroinflammation and CNS disease.Exposure to inhaled particulate matter activates early markers of oxidative stress, inflammation and unfolded protein response in rat striatumProteome changes in the small intestinal mucosa of broilers (Gallus gallus) induced by high concentrations of atmospheric ammonia.High Concentrations of Atmospheric Ammonia Induce Alterations in the Hepatic Proteome of Broilers (Gallus gallus): An iTRAQ-Based Quantitative Proteomic AnalysisAssociations of Ozone and PM2.5 Concentrations With Parkinson's Disease Among Participants in the Agricultural Health Study.The adverse effects of air pollution on the nervous system.Urban traffic-derived nanoparticulate matter reduces neurite outgrowth via TNFα in vitroDevelopmental exposure to concentrated ambient particles and preference for immediate reward in miceModification by hemochromatosis gene polymorphisms of the association between traffic-related air pollution and cognition in older men: a cohort study.Optimizing superparamagnetic iron oxide nanoparticles as drug carriers using an in vitro blood-brain barrier model.Surface area-dependence of gas-particle interactions influences pulmonary and neuroinflammatory outcomes.Health effects of concentrated ambient air particulate matter (CAPs) and its components.Air pollution: An environmental factor contributing to intestinal disease.Nanoparticles and colloids as contributing factors in neurodegenerative disease.Health effects of particulate air pollution: A review of epidemiological evidence.Toxicological and epidemiological studies of cardiovascular effects of ambient air fine particulate matter (PM2.5) and its chemical components: coherence and public health implications.Public health and components of particulate matter: the changing assessment of black carbon.Particulate Matter Facilitates C6 Glioma Cells Activation and the Release of Inflammatory Factors Through MAPK and JAK2/STAT3 Pathways.Ultrafine particles from diesel engines induce vascular oxidative stress via JNK activation.
P2860
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P2860
Inhaled ultrafine particulate matter affects CNS inflammatory processes and may act via MAP kinase signaling pathways.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
2008年论文
@zh
2008年论文
@zh-cn
name
Inhaled ultrafine particulate ...... MAP kinase signaling pathways.
@ast
Inhaled ultrafine particulate ...... MAP kinase signaling pathways.
@en
type
label
Inhaled ultrafine particulate ...... MAP kinase signaling pathways.
@ast
Inhaled ultrafine particulate ...... MAP kinase signaling pathways.
@en
prefLabel
Inhaled ultrafine particulate ...... MAP kinase signaling pathways.
@ast
Inhaled ultrafine particulate ...... MAP kinase signaling pathways.
@en
P2093
P2860
P1433
P1476
Inhaled ultrafine particulate ...... MAP kinase signaling pathways.
@en
P2093
A Campbell
J A Araujo
M T Kleinman
P2860
P304
P356
10.1016/J.TOXLET.2008.03.001
P577
2008-03-18T00:00:00Z