Ultrafine particles from diesel engines induce vascular oxidative stress via JNK activation. - Wikidata - wikimedia - TriplyDB

Ultrafine particles from diesel engines induce vascular oxidative stress via JNK activation.

Ultrafine particles from diesel engines induce vascular oxidative stress via JNK activation.

http://www.wikidata.org/entity/Q38901889

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Disturbed Flow Induces Autophagy, but Impairs Autophagic Flux to Perturb Mitochondrial Homeostasis.New approach to investigate the cytotoxicity of nanomaterials using single cell mechanics Particulate air pollution, systemic oxidative stress, inflammation, and atherosclerosis Manufactured and airborne nanoparticle cardiopulmonary interactions: a review of mechanisms and the possible contribution of mast cells Wood Smoke Particle Sequesters Cell Iron to Impact a Biological Effect Breathing easier? The known impacts of biodiesel on air quality Mitochondrial genetic background modifies the relationship between traffic-related air pollution exposure and systemic biomarkers of inflammation Oxidative stress and air pollution exposure Particulate matter exposure exacerbates high glucose-induced cardiomyocyte dysfunction through ROS generation Glutamatergic neurons in rodent models respond to nanoscale particulate urban air pollutants in vivo and in vitro A dynamic model of calcific nodule destabilization in response to monocyte- and oxidized lipid-induced matrix metalloproteinases.Mitochondrial haplogroups modify the effect of black carbon on age-related cognitive impairment Angiopoeitin-2 modulates Survivin expression in OxLDL-induced endothelial cell apoptosis.Beyond the Mean: Quantile Regression to Explore the Association of Air Pollution with Gene-Specific Methylation in the Normative Aging Study Stroke Damage Is Exacerbated by Nano-Size Particulate Matter in a Mouse Model.Heme oxygenase-1 protects endothelial cells from the toxicity of air pollutant chemicals Atmospheric ultrafine particles promote vascular calcification via the NF-κB signaling pathway Ambient ultrafine particles reduce endothelial nitric oxide production via S-glutathionylation of eNOS.Role of oxidative damage in toxicity of particulates.Ambient Ultrafine Particle Ingestion Alters Gut Microbiota in Association with Increased Atherogenic Lipid Metabolites.Composition of air pollution particles and oxidative stress in cells, tissues, and living systems.From particles to patients: oxidative stress and the cardiovascular effects of air pollution.Nanoparticles and the cardiovascular system: a critical review.The biological effects upon the cardiovascular system consequent to exposure to particulates of less than 500 nm in size.Organic nanoparticles from different fuel blends: in vitro toxicity and inflammatory potential.Biological effects of desert dust in respiratory epithelial cells and a murine model.Prooxidative effects of ambient pollutant chemicals are inhibited by HDL.Ultrafine particles from diesel vehicle emissions at different driving cycles induce differential vascular pro-inflammatory responses: implication of chemical components and NF-kappaB signaling.Diesel exhaust particles modulate vascular endothelial cell permeability: implication of ZO-1 expression Commentary: Flawed scientific-evidence standards delay diesel regulations.Emissions from commercial-grade charbroiling meat operations induce oxidative stress and inflammatory responses in human bronchial epithelial cells Pulsatile shear stress increased mitochondrial membrane potential: implication of Mn-SOD Sequestration of mitochondrial iron by silica particle initiates a biological effect.Ultrafine Particle Exposure Reveals the Importance of FOXO1/Notch Activation Complex for Vascular Regeneration.Diesel Exhaust Particles and the Induction of Macrophage Activation and Dysfunction.PM2.5 induces embryonic growth retardation: Potential involvement of ROS-MAPKs-apoptosis and G0/G1 arrest pathways.A new method and tool for detection and quantification of PM oxidative potential.Ultrafine Particulate Matter Increases Cardiac Ischemia/Reperfusion Injury via Mitochondrial Permeability Transition Pore.Resonant Raman-based cytochrome C biosensor as a tool for evaluating the oxidative properties of the diesel exhaust particulate matter Nature of Sub-23-nm Particles Downstream of the European Particle Measurement Programme (PMP)-Compliant System: A Real-Time Data Perspective

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P2860

Ultrafine particles from diesel engines induce vascular oxidative stress via JNK activation.

http://www.wikidata.org/entity/Q38901889

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2008 nî lūn-bûn

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2008年の論文

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Ultrafine particles from diese ...... ive stress via JNK activation.

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type

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Ultrafine particles from diese ...... ive stress via JNK activation.

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Ultrafine particles from diese ...... ive stress via JNK activation.

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Free Radical Biology and Medicine

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Ultrafine particles from diese ...... ive stress via JNK activation.

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Constantinos Sioutas

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Jeffery Cui

http://www.w3.org/2001/XMLSchema#string

Lisong Ai

http://www.w3.org/2001/XMLSchema#string

Rohit Majumdar

http://www.w3.org/2001/XMLSchema#string

Rongsong Li

http://www.w3.org/2001/XMLSchema#string

Tyler Beebe

http://www.w3.org/2001/XMLSchema#string

Tzung Hsiai

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Wakako Takabe

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P2860

Ultrafine particulate pollutants induce oxidative stress and mitochondrial damage

Toxic potential of materials at the nanolevel

Diesel exhaust particles induce oxidative stress, proinflammatory signaling, and P-glycoprotein up-regulation at the blood-brain barrier

The role of oxidative stress in ambient particulate matter-induced lung diseases and its implications in the toxicity of engineered nanoparticles

Air pollution and health

Use of proteomics to demonstrate a hierarchical oxidative stress response to diesel exhaust particle chemicals in a macrophage cell line.

Oxygen, oxidative stress, hypoxia, and heart failure.

Air pollution, oxidative stress and dietary supplementation: a review.

Ambient particulate pollutants in the ultrafine range promote early atherosclerosis and systemic oxidative stress.

Oxidative stress and cardiovascular injury: Part II: animal and human studies.

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