Regulatory mode shift of Tbc1d1 is required for acquisition of insulin-responsive GLUT4-trafficking activity.
about
Single quantum dot tracking reveals that an individual multivalent HIV-1 Tat protein transduction domain can activate machinery for lateral transport and endocytosis.Enhanced fasting glucose turnover in mice with disrupted action of TUG protein in skeletal muscle.AMP-activated protein kinase: a key regulator of energy balance with many roles in human disease.Live-cell single-molecule labeling and analysis of myosin motors with quantum dots.Expression, phosphorylation and function of the Rab-GTPase activating protein TBC1D1 in pancreatic beta-cells.Quercetin oppositely regulates insulin-mediated glucose disposal in skeletal muscle under normal and inflammatory conditions: The dual roles of AMPK activation.Heterotypic endosomal fusion as an initial trigger for insulin-induced glucose transporter 4 (GLUT4) translocation in skeletal muscle.Isoform-specific AMPK association with TBC1D1 is reduced by a mutation associated with severe obesity
P2860
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P2860
Regulatory mode shift of Tbc1d1 is required for acquisition of insulin-responsive GLUT4-trafficking activity.
description
2013 nî lūn-bûn
@nan
2013年の論文
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2013年論文
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2013年論文
@zh-hant
2013年論文
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2013年論文
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2013年論文
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2013年论文
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2013年论文
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2013年论文
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name
Regulatory mode shift of Tbc1d ...... ve GLUT4-trafficking activity.
@ast
Regulatory mode shift of Tbc1d ...... ve GLUT4-trafficking activity.
@en
type
label
Regulatory mode shift of Tbc1d ...... ve GLUT4-trafficking activity.
@ast
Regulatory mode shift of Tbc1d ...... ve GLUT4-trafficking activity.
@en
prefLabel
Regulatory mode shift of Tbc1d ...... ve GLUT4-trafficking activity.
@ast
Regulatory mode shift of Tbc1d ...... ve GLUT4-trafficking activity.
@en
P2860
P356
P1476
Regulatory mode shift of Tbc1d ...... ive GLUT4-trafficking activity
@en
P2093
Hiroyasu Hatakeyama
P2860
P304
P356
10.1091/MBC.E12-10-0725
P577
2013-01-16T00:00:00Z