Opposing regulation of T cell function by Egr-1/NAB2 and Egr-2/Egr-3. - Wikidata - wikimedia - TriplyDB

Opposing regulation of T cell function by Egr-1/NAB2 and Egr-2/Egr-3.

Opposing regulation of T cell function by Egr-1/NAB2 and Egr-2/Egr-3.

http://www.wikidata.org/entity/Q36685758

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Emerging roles of Egr2 and Egr3 in the control of systemic autoimmunity Feeding an army: The metabolism of T cells in activation, anergy, and exhaustion Does antigen masking by ubiquitin chains protect from the development of autoimmune diseases?Distinctive features of Egr transcription factor regulation and DNA binding activity in CA1 of the hippocampus in synaptic plasticity and consolidation and reconsolidation of fear memory Behavioral and Transcriptomic Fingerprints of an Enriched Environment in Horses (Equus caballus).Avidity-dependent programming of autoreactive T cells in T1D EGR-2 is not required for in vivo CD4 T cell mediated immune responses.A transcription factor map as revealed by a genome-wide gene expression analysis of whole-blood mRNA transcriptome in multiple sclerosis.Methamphetamine-induced dopamine-independent alterations in striatal gene expression in the 6-hydroxydopamine hemiparkinsonian rats.Regulation of CD4⁺ and CD8⁺ effector responses by Sprouty-1 Egr1 mediates p53-independent c-Myc-induced apoptosis via a noncanonical ARF-dependent transcriptional mechanism.The immediate early gene product EGR1 and polycomb group proteins interact in epigenetic programming during chondrogenesis.The early growth response gene Egr2 (Alias Krox20) is a novel transcriptional target of transforming growth factor-β that is up-regulated in systemic sclerosis and mediates profibrotic responses.Early growth response transcription factors: key mediators of fibrosis and novel targets for anti-fibrotic therapy.Early growth response-2 expression in uterine leiomyoma cells: regulation and function Apoptotic effects of two COX-2 inhibitors on breast adenocarcinoma cells through COX-2 independent pathway.Functional characterization of human T cell hyporesponsiveness induced by CTLA4-Ig.Genetic association of the EGR2 gene with bipolar disorder in Korea EGR1 and the ERK-ERF axis drive mammary cell migration in response to EGF.EGR1 controls divergent cellular responses of distinctive nucleus pulposus cell types T cell-specific notch inhibition blocks graft-versus-host disease by inducing a hyporesponsive program in alloreactive CD4+ and CD8+ T cells.Distinct functions of egr gene family members in cognitive processes.Egr2 is required for Bcl-2 induction during positive selection.IL-2 signaling prevents T cell anergy by inhibiting the expression of anergy-inducing genes The molecular signature of CD8+ T cells undergoing deletional tolerance Early growth response 3 (Egr-3) is induced by transforming growth factor-β and regulates fibrogenic responses.Egr3 induces a Th17 response by promoting the development of γδ T cells.Egr2 and Egr3 in regulatory T cells cooperatively control systemic autoimmunity through Ltbp3-mediated TGF-β3 production.E3 ubiquitin ligases in T-cell tolerance.Egr-1: new conductor for the tissue repair orchestra directs harmony (regeneration) or cacophony (fibrosis).Genetic and biochemical regulation of CD4 T cell effector differentiation: insights from examination of T cell clonal anergy.Blood cytokine, chemokine and gene expression in cholestasis patients with intractable pruritis treated with a molecular adsorbent recirculating system: a case series.Identification of Novel Fibrosis Modifiers by In Vivo siRNA Silencing.The transcriptional regulator NAB2 reveals a two-step induction of TRAIL in activated plasmacytoid DCs.The Myc-ARF-Egr1 pathway: unleashing the apoptotic power of c-Myc.EGR1, EGR2, and EGR3 activate the expression of their coregulator NAB2 establishing a negative feedback loop in cells of neuroectodermal and epithelial origin.The transcription factors Egr1 and Egr2 have opposing influences on adipocyte differentiation.Early growth response 2 and Egr3 are unique regulators in immune system Elevated and sustained expression of the transcription factors Egr1 and Egr2 controls NKT lineage differentiation in response to TCR signaling.Immature dendritic cells convert anergic nonregulatory T cells into Foxp3- IL-10+ regulatory T cells by engaging CD28 and CTLA-4.

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Opposing regulation of T cell function by Egr-1/NAB2 and Egr-2/Egr-3.

http://www.wikidata.org/entity/Q36685758

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2008 nî lūn-bûn

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2008年の論文

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2008年論文

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2008年論文

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2008年論文

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2008年论文

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name

Opposing regulation of T cell function by Egr-1/NAB2 and Egr-2/Egr-3.

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Opposing regulation of T cell function by Egr-1/NAB2 and Egr-2/Egr-3.

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Opposing regulation of T cell function by Egr-1/NAB2 and Egr-2/Egr-3.

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Opposing regulation of T cell function by Egr-1/NAB2 and Egr-2/Egr-3.

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European Journal of Immunology

P1476

Opposing regulation of T cell function by Egr-1/NAB2 and Egr-2/Egr-3.

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Gilbert J Kersh

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Jonathan D Powell

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Michael A Lutz

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Paul E Zarek

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Robert A Anders

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Sam Collins

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P2860

Calcineurin imposes T cell unresponsiveness through targeted proteolysis of signaling proteins

Sensory ataxia and muscle spindle agenesis in mice lacking the transcription factor Egr3

NAB2, a corepressor of NGFI-A (Egr-1) and Krox20, is induced by proliferative and differentiative stimuli

Early growth response proteins (EGR) and nuclear factors of activated T cells (NFAT) form heterodimers and regulate proinflammatory cytokine gene expression

Identification of NAB1, a repressor of NGFI-A- and Krox20-mediated transcription

Sequential involvement of NFAT and Egr transcription factors in FasL regulation

Nab proteins are essential for peripheral nervous system myelination

The EGR family of transcription-regulatory factors: progress at the interface of molecular and systems neuroscience.

Cyclosporin A-sensitive transcription factor Egr-3 regulates Fas ligand expression.

Transcriptional mechanisms underlying lymphocyte tolerance.

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528-536

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10.1002/EJI.200737157

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2

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38

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5648691

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18203138

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3598016

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