Epstein-Barr virus-derived plasmids replicate only once per cell cycle and are not amplified after entry into cells.
about
Mre11 complex and DNA replication: linkage to E2F and sites of DNA synthesisEBP2, a human protein that interacts with sequences of the Epstein-Barr virus nuclear antigen 1 important for plasmid maintenance.Inhibition of Epstein-Barr virus OriP function by tankyrase, a telomere-associated poly-ADP ribose polymerase that binds and modifies EBNA1Interplay of Murine Gammaherpesvirus 68 with NF-kappaB Signaling of the HostEpstein-Barr virus latent genesEfficient feeder-free episomal reprogramming with small moleculesHuman induced pluripotent stem cells free of vector and transgene sequencesTransmission of human T-cell lymphotropic virus type 1 tax to rabbits by tax-only-positive human cells.Human RPA (hSSB) interacts with EBNA1, the latent origin binding protein of Epstein-Barr virusA role for MRE11, NBS1, and recombination junctions in replication and stable maintenance of EBV episomes.Homologous recombinational repair factors are recruited and loaded onto the viral DNA genome in Epstein-Barr virus replication compartments.Gene-delivery systems for iPS cell generation.A role for DNA polymerase θ in promoting replication through oxidative DNA lesion, thymine glycol, in human cells.Architecture of replication compartments formed during Epstein-Barr virus lytic replication.Functional analyses of the EBNA1 origin DNA binding protein of Epstein-Barr virus.The minimal replicator of Epstein-Barr virus oriP.Repression of Epstein-Barr virus EBNA-1 gene transcription by pRb during restricted latencyCell cycle regulation of chromatin at an origin of DNA replicationThe replicator of the Epstein-Barr virus latent cycle origin of DNA replication, oriP, is composed of multiple functional elements.Protein-DNA binding and CpG methylation at nucleotide resolution of latency-associated promoters Qp, Cp, and LMP1p of Epstein-Barr virusThe dyad symmetry element of Epstein-Barr virus is a dominant but dispensable replication origin.Human DNA replication initiation factors, ORC and MCM, associate with oriP of Epstein-Barr virus.Constitutive binding of EBNA1 protein to the Epstein-Barr virus replication origin, oriP, with distortion of DNA structure during latent infection.The spacing between adjacent binding sites in the family of repeats affects the functions of Epstein-Barr nuclear antigen 1 in transcription activation and stable plasmid maintenanceHuman origin recognition complex binds to the region of the latent origin of DNA replication of Epstein-Barr virus.Separation of the DNA replication, segregation, and transcriptional activation functions of Epstein-Barr nuclear antigen 1.Functional dissection of latency-associated nuclear antigen 1 of Kaposi's sarcoma-associated herpesvirus involved in latent DNA replication and transcription of terminal repeats of the viral genome.Episomal maintenance of plasmids with hybrid origins in mouse cellsInhibition of S-phase cyclin-dependent kinase activity blocks expression of Epstein-Barr virus immediate-early and early genes, preventing viral lytic replicationInterpreting the Epstein-Barr Virus (EBV) epigenome using high-throughput dataDifferent modes of human papillomavirus DNA replication during maintenanceEpstein-Barr virus (EBV) nuclear antigen 1 colocalizes with cellular replication foci in the absence of EBV plasmids.The Epstein-Barr virus encoded BART miRNAs potentiate tumor growth in vivo.The BAH domain facilitates the ability of human Orc1 protein to activate replication origins in vivo.Replication and partitioning of papillomavirus genomes.Identification of an insulator in AAVS1, a preferred region for integration of adeno-associated virus DNA.Telomerase activity impacts on Epstein-Barr virus infection of AGS cells.Chromatin remodeling directly activates V(D)J recombination.The Cell Cycle Timing of Human Papillomavirus DNA Replication.Affinity proteomics to study endogenous protein complexes: pointers, pitfalls, preferences and perspectives.
P2860
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P2860
Epstein-Barr virus-derived plasmids replicate only once per cell cycle and are not amplified after entry into cells.
description
1991 nî lūn-bûn
@nan
1991年の論文
@ja
1991年論文
@yue
1991年論文
@zh-hant
1991年論文
@zh-hk
1991年論文
@zh-mo
1991年論文
@zh-tw
1991年论文
@wuu
1991年论文
@zh
1991年论文
@zh-cn
name
Epstein-Barr virus-derived pla ...... lified after entry into cells.
@ast
Epstein-Barr virus-derived pla ...... lified after entry into cells.
@en
type
label
Epstein-Barr virus-derived pla ...... lified after entry into cells.
@ast
Epstein-Barr virus-derived pla ...... lified after entry into cells.
@en
prefLabel
Epstein-Barr virus-derived pla ...... lified after entry into cells.
@ast
Epstein-Barr virus-derived pla ...... lified after entry into cells.
@en
P2860
P1433
P1476
Epstein-Barr virus-derived pla ...... lified after entry into cells.
@en
P2093
P2860
P304
P407
P577
1991-01-01T00:00:00Z