Rapid resolution of duck hepatitis B virus infections occurs after massive hepatocellular involvement
about
Hepatitis B virus regulatory HBx protein binds to adaptor protein IPS-1 and inhibits the activation of beta interferonIdentification and characterization of avihepadnaviruses isolated from exotic anseriformes maintained in captivityHeterologous replacement of the supposed host determining region of avihepadnaviruses: high in vivo infectivity despite low infectivity for hepatocytesMolecular biology of hepatitis B virus infectionAntiviral activity of beta-L-2',3'-dideoxy-2',3'-didehydro-5-fluorocytidine in woodchucks chronically infected with woodchuck hepatitis virusEvidence that hepatocyte turnover is required for rapid clearance of duck hepatitis B virus during antiviral therapy of chronically infected ducksInhibitory effect of 2'-fluoro-5-methyl-beta-L-arabinofuranosyl-uracil on duck hepatitis B virus replication.Immune Tolerant Chronic Hepatitis B: The Unrecognized RisksEffect of antiviral treatment with entecavir on age- and dose-related outcomes of duck hepatitis B virus infection.Protective efficacy of DNA vaccines against duck hepatitis B virus infection.Infection of ducklings with virus particles containing linear double-stranded duck hepatitis B virus DNA: illegitimate replication and reversionApoptosis and regeneration of hepatocytes during recovery from transient hepadnavirus infectionsCombination therapy with lamivudine and adenovirus causes transient suppression of chronic woodchuck hepatitis virus infections.Kinetics of hepadnavirus loss from the liver during inhibition of viral DNA synthesis.Hepatitis B virus biology.Reverse transcription-associated dephosphorylation of hepadnavirus nucleocapsids.Hepadnavirus assembly and reverse transcription require a multi-component chaperone complex which is incorporated into nucleocapsids.Age-related differences in amplification of covalently closed circular DNA at early times after duck hepatitis B virus infection of ducks.Characterization of the antiviral effect of 2',3'-dideoxy-2', 3'-didehydro-beta-L-5-fluorocytidine in the duck hepatitis B virus infection model.Covalently closed circular DNA is the predominant form of duck hepatitis B virus DNA that persists following transient infection.Proteomic analysis of primary duck hepatocytes infected with duck hepatitis B virusInhibitory effect of adefovir on viral DNA synthesis and covalently closed circular DNA formation in duck hepatitis B virus-infected hepatocytes in vivo and in vitro.Phosphorylation state-dependent interactions of hepadnavirus core protein with host factorsInterferons accelerate decay of replication-competent nucleocapsids of hepatitis B virusDynamics of hepatitis B virus clearance in chimpanzees.Half-life of the duck hepatitis B virus covalently closed circular DNA pool in vivo following inhibition of viral replication.CD8(+) T cells mediate viral clearance and disease pathogenesis during acute hepatitis B virus infection.Identification and characterization of multiple TRIM proteins that inhibit hepatitis B virus transcription.Regulation of multiple stages of hepadnavirus replication by the carboxyl-terminal domain of viral core protein in trans.Selective inhibition of the duck hepatitis B virus by a new class of tetraazamacrocyclesEntecavir therapy combined with DNA vaccination for persistent duck hepatitis B virus infection.Animal models and the molecular biology of hepadnavirus infectionRapid production of neutralizing antibody leads to transient hepadnavirus infection.Regulation of hepadnavirus reverse transcription by dynamic nucleocapsid phosphorylation.The liver of woodchucks chronically infected with the woodchuck hepatitis virus contains foci of virus core antigen-negative hepatocytes with both altered and normal morphology.The level of viral antigen presented by hepatocytes influences CD8 T-cell function.Review of cytokine profiles in patients with hepatitisTherapeutic Antiviral Effect of the Nucleic Acid Polymer REP 2055 against Persistent Duck Hepatitis B Virus Infection.Detection of an RNase H activity associated with hepadnaviruses.n-Butyrate, a cell cycle blocker, inhibits early amplification of duck hepatitis B virus covalently closed circular DNA after in vitro infection of duck hepatocytes
P2860
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P2860
Rapid resolution of duck hepatitis B virus infections occurs after massive hepatocellular involvement
description
1992 nî lūn-bûn
@nan
1992年の論文
@ja
1992年論文
@yue
1992年論文
@zh-hant
1992年論文
@zh-hk
1992年論文
@zh-mo
1992年論文
@zh-tw
1992年论文
@wuu
1992年论文
@zh
1992年论文
@zh-cn
name
Rapid resolution of duck hepat ...... ive hepatocellular involvement
@ast
Rapid resolution of duck hepat ...... ive hepatocellular involvement
@en
type
label
Rapid resolution of duck hepat ...... ive hepatocellular involvement
@ast
Rapid resolution of duck hepat ...... ive hepatocellular involvement
@en
prefLabel
Rapid resolution of duck hepat ...... ive hepatocellular involvement
@ast
Rapid resolution of duck hepat ...... ive hepatocellular involvement
@en
P2093
P2860
P1433
P1476
Rapid resolution of duck hepat ...... ive hepatocellular involvement
@en
P2093
A P O'Connell
A R Jilbert
J M England
P2860
P304
P407
P577
1992-03-01T00:00:00Z