Apoptosis and regeneration of hepatocytes during recovery from transient hepadnavirus infections
about
Hepatitis B virus regulatory HBx protein binds to adaptor protein IPS-1 and inhibits the activation of beta interferonTargeted DNA mutagenesis for the cure of chronic viral infectionsTherapeutic vaccination and immunomodulation in the treatment of chronic hepatitis B: preclinical studies in the woodchuckCytopathic and Noncytopathic Interferon Responses in Cells Expressing Hepatitis C Virus Subgenomic RepliconsImmune-Regulatory Events in the Clearance of HBsAg in Chronic Hepatitis B: Focuses on HLA-DPAntiviral activity of beta-L-2',3'-dideoxy-2',3'-didehydro-5-fluorocytidine in woodchucks chronically infected with woodchuck hepatitis virusHepatitis B virus molecular biology and pathogenesisThe woodchuck as an animal model for pathogenesis and therapy of chronic hepatitis B virus infectionCombination therapy with lamivudine and adenovirus causes transient suppression of chronic woodchuck hepatitis virus infections.Role of NF-kappaB and myc proteins in apoptosis induced by hepatitis B virus HBx protein.Kinetics of hepadnavirus loss from the liver during inhibition of viral DNA synthesis.Hepatitis B virus biology.Antibody responses during hepatitis B viral infection.Inhibitory effect of adefovir on viral DNA synthesis and covalently closed circular DNA formation in duck hepatitis B virus-infected hepatocytes in vivo and in vitro.Interferons accelerate decay of replication-competent nucleocapsids of hepatitis B virusThe woodchuck model of hepatitis B virus infection.Persistence of hepatitis B virus covalently closed circular DNA in hepatocytes: molecular mechanisms and clinical significance.Dynamics of hepatitis B virus clearance in chimpanzees.Resistance of hepatitis B virus to antiviral drugs: current aspects and directions for future investigation.Deficiencies in the acute-phase cell-mediated immune response to viral antigens are associated with development of chronic woodchuck hepatitis virus infection following neonatal inoculation.Immunization with surface antigen vaccine alone and after treatment with 1-(2-fluoro-5-methyl-beta-L-arabinofuranosyl)-uracil (L-FMAU) breaks humoral and cell-mediated immune tolerance in chronic woodchuck hepatitis virus infection.CD8(+) T cells mediate viral clearance and disease pathogenesis during acute hepatitis B virus infection.Conditional replication of duck hepatitis B virus in hepatoma cells.Mathematical modeling of triphasic viral dynamics in patients with HBeAg-positive chronic hepatitis B showing response to 24-week clevudine therapy.Control or persistence of hepatitis B virus: the critical role of initial host-virus interactions.Quantitative analysis of HBV cccDNA from clinical specimens: correlation with clinical and virological response during antiviral therapy.Targeting Hepatitis B Virus With CRISPR/Cas9.Tumor necrosis factor alpha inhibition of hepatitis B virus replication involves disruption of capsid Integrity through activation of NF-kappaB.Identification and characterization of multiple TRIM proteins that inhibit hepatitis B virus transcription.Alpha-interferon suppresses hepadnavirus transcription by altering epigenetic modification of cccDNA minichromosomesSTING agonists induce an innate antiviral immune response against hepatitis B virus.Emerging drugs for hepatitis B.The role of cells refractory to productive infection in acute hepatitis B viral dynamics.The liver of woodchucks chronically infected with the woodchuck hepatitis virus contains foci of virus core antigen-negative hepatocytes with both altered and normal morphology.Hepadnavirus Genome Replication and Persistence.Tumor necrosis factor activates a conserved innate antiviral response to hepatitis B virus that destabilizes nucleocapsids and reduces nuclear viral DNA.Antiviral therapy of chronic hepatitis B: can we clear the virus and prevent drug resistance?Hepatocyte turnover during resolution of a transient hepadnaviral infection.Kinetics of acute hepatitis B virus infection in humans.Hepatitis B virus X protein targets Bcl-2 proteins to increase intracellular calcium, required for virus replication and cell death induction.
P2860
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P2860
Apoptosis and regeneration of hepatocytes during recovery from transient hepadnavirus infections
description
2000 nî lūn-bûn
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2000 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2000 թվականի փետրվարին հրատարակված գիտական հոդված
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2000年の論文
@ja
2000年論文
@yue
2000年論文
@zh-hant
2000年論文
@zh-hk
2000年論文
@zh-mo
2000年論文
@zh-tw
2000年论文
@wuu
name
Apoptosis and regeneration of ...... nsient hepadnavirus infections
@ast
Apoptosis and regeneration of ...... nsient hepadnavirus infections
@en
type
label
Apoptosis and regeneration of ...... nsient hepadnavirus infections
@ast
Apoptosis and regeneration of ...... nsient hepadnavirus infections
@en
prefLabel
Apoptosis and regeneration of ...... nsient hepadnavirus infections
@ast
Apoptosis and regeneration of ...... nsient hepadnavirus infections
@en
P2093
P2860
P1433
P1476
Apoptosis and regeneration of ...... nsient hepadnavirus infections
@en
P2093
J Saputelli
M I Barrasa
T Whitaker
P2860
P304
P356
10.1128/JVI.74.3.1495-1505.2000
P577
2000-02-01T00:00:00Z