Dormant cancer cells contribute to residual disease in a model of reversible pancreatic cancer. - Wikidata - wikimedia - TriplyDB

Dormant cancer cells contribute to residual disease in a model of reversible pancreatic cancer.

Dormant cancer cells contribute to residual disease in a model of reversible pancreatic cancer.

http://www.wikidata.org/entity/Q36698162

about

Galectin-1 drives pancreatic carcinogenesis through stroma remodeling and Hedgehog signaling activation.Autocrine IGF1 Signaling Mediates Pancreatic Tumor Cell Dormancy in the Absence of Oncogenic Drivers.Cancer stem cells as a target population for drug discovery.Whole-exome sequencing of pancreatic cancer defines genetic diversity and therapeutic targets.ERK kinase phosphorylates and destabilizes the tumor suppressor FBW7 in pancreatic cancer Overexpression of PD2 leads to increased tumorigenicity and metastasis in pancreatic ductal adenocarcinoma.Pancreatic cancer stem cells in patient pancreatic xenografts are sensitive to drozitumab, an agonistic antibody against DR5.Cancer cell dormancy in novel mouse models for reversible pancreatic cancer: a lingering challenge in the development of targeted therapies.Gene expression profiling of patient-derived pancreatic cancer xenografts predicts sensitivity to the BET bromodomain inhibitor JQ1: implications for individualized medicine efforts MYC in pancreatic cancer: novel mechanistic insights and their translation into therapeutic strategies.SIRT1 and circadian gene expression in pancreatic ductal adenocarcinoma: Effect of starvation.Longikaurin E induces apoptosis of pancreatic cancer cells via modulation of the p38 and PI3K/AKT pathways by ROS.Kras(G12D) induces EGFR-MYC cross signaling in murine primary pancreatic ductal epithelial cells.The BET bromodomain inhibitor JQ1 suppresses growth of pancreatic ductal adenocarcinoma in patient-derived xenograft models.MYC regulates ductal-neuroendocrine lineage plasticity in pancreatic ductal adenocarcinoma associated with poor outcome and chemoresistance.KRAS: The Critical Driver and Therapeutic Target for Pancreatic Cancer.Inherited pancreatic cancer.Loss of KDM6A Activates Super-Enhancers to Induce Gender-Specific Squamous-like Pancreatic Cancer and Confers Sensitivity to BET Inhibitors.Pancreatic Cancer: Molecular Characterization, Clonal Evolution and Cancer Stem Cells.E47 Governs the MYC-CDKN1B/p27KIP1-RB Network to Growth Arrest PDA Cells Independent of CDKN2A/p16INK4A and Wild-Type p53.

P2860

Q30620697-BEB65354-F2DF-4610-9208-225A1E21DBAF Q30843173-AAF4DC28-50C0-47EE-A33B-BDEC82DC7925 Q35390334-ED8C3D0C-2DFC-4DCD-B024-1CC4E212A7F7 Q35487646-9F5EF511-1280-403D-96F7-2F204AFA7035 Q35578938-4CF48377-00B9-4272-BC2B-97299D4598DC Q36772125-D2273DE1-49B1-42B8-A713-C3217D6B9A57 Q37024543-1E13CF52-BE41-420C-A054-ED0539E7C5D2 Q37712209-352129BA-E517-4AEF-A588-C4EED8A2C80E Q37733990-5F3BEB6C-66A9-4F24-BE2C-C4C448B6925E Q38539670-C2DCCC03-3D61-4605-BAA8-9649D0EAD7D2 Q38896426-2ED50062-152D-4713-A159-8392A98C8439 Q38903188-F2411331-E228-437E-BCBF-38C861270A11 Q40291371-9181906E-8709-49FF-A7B4-2CD5EFCAEC14 Q42701804-46F167BA-4FC4-48E8-8667-623868A145A0 Q47111931-7B4C3A40-2F62-4A81-9939-1E80B35BBFAC Q47563012-D895E0C2-F16D-4FD9-963E-F945628D928A Q50146622-4AD49755-9E0B-4411-8030-011316D6248E Q52357490-44DD3759-B98E-4DE0-9ED4-8AA829DC3232 Q55104265-75860DDF-DFA1-48BF-9091-9BB628CFEAC5 Q55616605-474E63F2-BE68-4D76-9E8F-6225BB593C06

P2860

Dormant cancer cells contribute to residual disease in a model of reversible pancreatic cancer.

http://www.wikidata.org/entity/Q36698162

description

2013 nî lūn-bûn

@nan

2013年の論文

@ja

2013年論文

@yue

2013年論文

@zh-hant

2013年論文

@zh-hk

2013年論文

@zh-mo

2013年論文

@zh-tw

2013年论文

@wuu

2013年论文

@zh

2013年论文

@zh-cn

name

Dormant cancer cells contribut ...... reversible pancreatic cancer.

@ast

Dormant cancer cells contribut ...... reversible pancreatic cancer.

@en

type

label

Dormant cancer cells contribut ...... reversible pancreatic cancer.

@ast

Dormant cancer cells contribut ...... reversible pancreatic cancer.

@en

prefLabel

Dormant cancer cells contribut ...... reversible pancreatic cancer.

@ast

Dormant cancer cells contribut ...... reversible pancreatic cancer.

@en

P1433

Q36698162-B16D94EF-4055-4617-B401-00D580B49CB5

P1476

Q36698162-D33BCBCC-BAEB-46AD-8511-CE782AE4186F

P2093

Q36698162-36A6487E-0F2E-4B59-94E3-3ECF78ED725E

Q36698162-3A527AA2-C5E1-49F6-9758-02D0DB3F4D3F

Q36698162-3D085E10-3BA4-48BA-8A3E-17B39C21BE75

Q36698162-4D094C72-FF5A-4574-9EE7-F54AB84A5EB7

Q36698162-5943E174-6F4D-40B1-94A9-252B9E0FB129

Q36698162-6911FA37-6988-43FD-8C22-9E789D438063

Q36698162-7DDAA354-9FFA-4B3A-90BB-A10734DA2AA8

Q36698162-8E250141-EFB2-452C-B2D1-9D6CB9BD5D4C

Q36698162-A3B6FE57-C21A-4A54-B103-5708A7FCA5E4

Q36698162-A725094C-622A-4FE4-A85F-AEF6FC2B5AD8

P2860

Q36698162-0985DA7D-DA81-42C2-8149-F6FE4D4D7953

Q36698162-0E3A4504-3B88-461F-8165-679FA85D8086

Q36698162-0EF96E3D-EE63-4A3A-A044-BF9447147C52

Q36698162-1EDE1683-717B-42AE-89F7-6F8E440DE7FF

Q36698162-2CFBD001-0CAB-4C5F-BF26-76BA1CD6B14F

Q36698162-304492B9-0D8D-43B7-AB46-4598A7FB9C40

Q36698162-43A6AD5E-FBBA-49C1-B07B-AA58A67EC06E

Q36698162-4A5D1456-6E91-4975-B54A-F1C0F4576679

Q36698162-51584ECF-F6B4-49D4-9900-558220ED2163

Q36698162-58BDA4EA-6F7F-42B4-AF44-97D91DEF9E52

P304

Q36698162-1ACCB484-547A-4170-AE0D-B029CDD18406

P31

Q36698162-A518A141-C6E6-4519-A7B9-B4F9C036E254

P356

Q36698162-D3BECE97-D459-4C85-9158-50CA0CCCC56D

P407

Q36698162-F0AE676F-4571-4811-BDC2-14022763DEA5

P433

Q36698162-A772F70E-D966-4A53-8D55-92022D520D7E

P478

Q36698162-A1E8D945-4B8A-4610-AE09-0EDA46619230

P50

Q36698162-AA232C39-BE3C-4162-9350-49E46B0FB7D8

P577

Q36698162-F92C3F86-2D0E-4974-B16F-215FFFF786FA

P5875

Q36698162-9D538E54-EAD7-4F81-8129-3AA8A9171C43

P698

Q36698162-3D3557B4-DFAB-424D-9408-EBF47CE153D5

P932

Q36698162-54130E40-1637-4D50-9BFF-41D063FC63AD

P356

0008-5472.CAN-12-2067

P1433

Cancer Research

P1476

Dormant cancer cells contribut ...... f reversible pancreatic cancer

@en

P2093

Agnieszka K Witkiewicz

http://www.w3.org/2001/XMLSchema#string

Aleata A Triplett

http://www.w3.org/2001/XMLSchema#string

Chengbao Liu

http://www.w3.org/2001/XMLSchema#string

Dean W Felsher

http://www.w3.org/2001/XMLSchema#string

Dominick J DiMaio

http://www.w3.org/2001/XMLSchema#string

Hallgeir Rui

http://www.w3.org/2001/XMLSchema#string

John P Morris

http://www.w3.org/2001/XMLSchema#string

Jonathan R Brody

http://www.w3.org/2001/XMLSchema#string

Kazuhito Sakamoto

http://www.w3.org/2001/XMLSchema#string

Matthias Hebrok

http://www.w3.org/2001/XMLSchema#string

P2860

Core signaling pathways in human pancreatic cancers revealed by global genomic analyses

Distinct populations of cancer stem cells determine tumor growth and metastatic activity in human pancreatic cancer

Identification of pancreatic cancer stem cells

Defining the epithelial stem cell niche in skin

Autophagy in cell death: an innocent convict?

Preinvasive and invasive ductal pancreatic cancer and its early detection in the mouse

Isolation and characterization of centroacinar/terminal ductal progenitor cells in adult mouse pancreas

Mutant p53 drives metastasis and overcomes growth arrest/senescence in pancreatic cancer

Prognostic significance of tumorigenic cells with mesenchymal features in pancreatic adenocarcinoma

Temporally and spatially controlled expression of transgenes in embryonic and adult tissues.

P304

1821-1830

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1158/0008-5472.CAN-12-2067

http://www.w3.org/2001/XMLSchema#string

P407

P433

6

http://www.w3.org/2001/XMLSchema#string

P478

73

http://www.w3.org/2001/XMLSchema#string

P50

P577

2013-03-06T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P5875

235881695

http://www.w3.org/2001/XMLSchema#string

P698

23467612

http://www.w3.org/2001/XMLSchema#string

P932

3602120

http://www.w3.org/2001/XMLSchema#string