Minocycline attenuates lipopolysaccharide (LPS)-induced neuroinflammation, sickness behavior, and anhedonia. - Wikidata - wikimedia - TriplyDB

Minocycline attenuates lipopolysaccharide (LPS)-induced neuroinflammation, sickness behavior, and anhedonia.

Minocycline attenuates lipopolysaccharide (LPS)-induced neuroinflammation, sickness behavior, and anhedonia.

http://www.wikidata.org/entity/Q36699392

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Neuroinflammation and psychiatric illness The role of immune dysfunction in the pathophysiology of autism Corticosterone primes the neuroinflammatory response to DFP in mice: potential animal model of Gulf War Illness Role of inflammation and infection in the pathogenesis of human acute liver failure: Clinical implications for monitoring and therapy Fundamental role of pan-inflammation and oxidative-nitrosative pathways in neuropathogenesis of Alzheimer's disease in focal cerebral ischemic rats Fundamental role of pan-inflammation and oxidative-nitrosative pathways in neuropathogenesis of Alzheimer's disease Inflammation Models of Depression in Rodents: Relevance to Psychotropic Drug Discovery Microglial Cells as a Link between Cannabinoids and the Immune Hypothesis of Psychiatric Disorders Stress and neuroinflammation: a systematic review of the effects of stress on microglia and the implications for mental illness Neuroinflammation and Depression: Microglia Activation, Extracellular Microvesicles and microRNA Dysregulation Kynurenine 3-Monooxygenase: An Influential Mediator of Neuropathology Glia in the cytokine-mediated onset of depression: fine tuning the immune response Microglial priming and enhanced reactivity to secondary insult in aging, and traumatic CNS injury, and neurodegenerative disease Neuroimmune and Neuropathic Responses of Spinal Cord and Dorsal Root Ganglia in Middle Age The antidepressant-like effects of pioglitazone in a chronic mild stress mouse model are associated with PPARγ-mediated alteration of microglial activation phenotypes.Tumor growth increases neuroinflammation, fatigue and depressive-like behavior prior to alterations in muscle function.Psychoneuroimmunology meets neuropsychopharmacology: translational implications of the impact of inflammation on behavior Rodent models of depression: neurotrophic and neuroinflammatory biomarkers.Neuroinflammation in the normal aging hippocampus Blood-borne metabolic factors in obesity exacerbate injury-induced gliosis The pharmacokinetic-pharmacodynamic model of azithromycin for lipopolysaccharide-induced depressive-like behavior in mice Inflammation and its discontents: the role of cytokines in the pathophysiology of major depression Preventive effects of minocycline in a neurodevelopmental two-hit model with relevance to schizophrenia.Role of interleukin-1beta in postoperative cognitive dysfunction.PKR deficiency alters E. coli-induced sickness behaviors but does not exacerbate neuroimmune responses or bacterial load Oligodendroglial alterations and the role of microglia in white matter injury: relevance to schizophrenia.The immune system and developmental programming of brain and behavior.Post-traumatic anxiety associates with failure of the innate immune receptor TLR9 to evade the pro-inflammatory NFκB pathway.P2X7 signaling promotes microsphere embolism-triggered microglia activation by maintaining elevation of Fas ligand Behavioral and monoamine perturbations in adult male mice with chronic inflammation induced by repeated peripheral lipopolysaccharide administration.Pro-epileptogenic effects of viral-like inflammation in both mature and immature brains.Neurotoxic kynurenine metabolism is increased in the dorsal hippocampus and drives distinct depressive behaviors during inflammation.Differential sensitivity to endotoxin exposure in young and middle-age mice Interferon-gamma and tumor necrosis factor-alpha mediate the upregulation of indoleamine 2,3-dioxygenase and the induction of depressive-like behavior in mice in response to bacillus Calmette-Guerin Immune-mediated animal models of Tourette syndrome.Inflammation: The Common Pathway of Stress-Related Diseases.Anti-inflammatory Actions of Adjunctive Tetracyclines and Other Agents in Periodontitis and Associated Comorbidities.Electroacupuncture Alleviates Postoperative Cognitive Dysfunction in Aged Rats by Inhibiting Hippocampal Neuroinflammation Activated via Microglia/TLRs Pathway.Central administration of lipopolysaccharide induces depressive-like behavior in vivo and activates brain indoleamine 2,3 dioxygenase in murine organotypic hippocampal slice cultures Activated microglia contribute to neuronal apoptosis in Toxoplasmic encephalitis.

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P2860

Minocycline attenuates lipopolysaccharide (LPS)-induced neuroinflammation, sickness behavior, and anhedonia.

http://www.wikidata.org/entity/Q36699392

description

2008 nî lūn-bûn

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2008年の論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年论文

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2008年论文

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2008年论文

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name

Minocycline attenuates lipopol ...... kness behavior, and anhedonia.

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Minocycline attenuates lipopol ...... kness behavior, and anhedonia.

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Minocycline attenuates lipopol ...... kness behavior, and anhedonia.

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Minocycline attenuates lipopol ...... kness behavior, and anhedonia.

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Minocycline attenuates lipopol ...... kness behavior, and anhedonia.

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Minocycline attenuates lipopol ...... kness behavior, and anhedonia.

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Minocycline attenuates lipopol ...... ckness behavior, and anhedonia

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Angela Wynne

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Christopher J Henry

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Jonathan P Godbout

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Justin Himler

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Mark Hanke

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Yan Huang

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P2860

Microglia initiate central nervous system innate and adaptive immune responses through multiple TLRs

Cytokines sing the blues: inflammation and the pathogenesis of depression

From inflammation to sickness and depression: when the immune system subjugates the brain

A novel mechanism of action of tetracyclines: effects on nitric oxide synthases

Resting microglial cells are highly dynamic surveillants of brain parenchyma in vivo

Minocycline down-regulates MHC II expression in microglia and macrophages through inhibition of IRF-1 and protein kinase C (PKC)alpha/betaII

mRNA up-regulation of MHC II and pivotal pro-inflammatory genes in normal brain aging

ATP mediates rapid microglial response to local brain injury in vivo

Inflammation is detrimental for neurogenesis in adult brain

Enhanced neuronal activation in central autonomic network nuclei in aged mice following acute peripheral immune challenge.

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Michael T Bailey

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lipopolysaccharide

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2412862

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