CIKS (Act1 or TRAF3IP2) mediates high glucose-induced endothelial dysfunction. - Wikidata - wikimedia - TriplyDB

CIKS (Act1 or TRAF3IP2) mediates high glucose-induced endothelial dysfunction.

CIKS (Act1 or TRAF3IP2) mediates high glucose-induced endothelial dysfunction.

http://www.wikidata.org/entity/Q36711861

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The Nox1/4 Dual Inhibitor GKT137831 or Nox4 Knockdown Inhibits Angiotensin-II-Induced Adult Mouse Cardiac Fibroblast Proliferation and Migration. AT1 Physically Associates With Nox4.Dynamic cellular and molecular modulations of diabetes mediated head and neck carcinogenesis.Interleukin-18 enhances IL-18R/Nox1 binding, and mediates TRAF3IP2-dependent smooth muscle cell migration. Inhibition by simvastatin Advanced oxidation protein products induce cardiomyocyte death via Nox2/Rac1/superoxide-dependent TRAF3IP2/JNK signaling.Angiotensin II stimulates cardiac fibroblast migration via the differential regulation of matrixins and RECK OxLDL induces endothelial dysfunction and death via TRAF3IP2: inhibition by HDL3 and AMPK activators Stevens-Johnson syndrome and toxic epidermal necrolysis: an update on pharmacogenetics studies in drug-induced severe skin reaction.C/EBPβ regulates P2X7 receptor expression in response to glucose challenge in intestinal epithelial cells.Targeting TRAF3IP2 by Genetic and Interventional Approaches Inhibits Ischemia/Reperfusion-induced Myocardial Injury and Adverse Remodeling.Acetylsalicylic acid inhibits IL-18-induced cardiac fibroblast migration through the induction of RECK A pharmacogenetics study in Mozambican patients treated with nevirapine: full resequencing of TRAF3IP2 gene shows a novel association with SJS/TEN susceptibility Impact of glutathione transferases genes polymorphisms in nevirapine adverse reactions: a possible role for GSTM1 in SJS/TEN susceptibility.TRAF3IP2 mediates atherosclerotic plaque development and vulnerability in ApoE(-/-) mice.TRAF3IP2 mediates interleukin-18-induced cardiac fibroblast migration and differentiation TRAF3IP2 mediates high glucose-induced endothelin-1 production as well as endothelin-1-induced inflammation in endothelial cells.Dipeptidyl Peptidase-4 Inhibition With Saxagliptin Ameliorates Angiotensin II-Induced Cardiac Diastolic Dysfunction in Male Mice.Anti-inflammatory Effects of Atorvastatin by Suppressing TRAF3IP2 and IL-17RA in Human Glioblastoma Spheroids Cultured in a Three-dimensional Model: Possible Relevance to Glioblastoma Treatment.Too much TRAFfic at the crossroads of diabetes and endothelial dysfunction.

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CIKS (Act1 or TRAF3IP2) mediates high glucose-induced endothelial dysfunction.

http://www.wikidata.org/entity/Q36711861

description

2012 nî lūn-bûn

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2012年の論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年论文

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2012年论文

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2012年论文

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name

CIKS (Act1 or TRAF3IP2) mediates high glucose-induced endothelial dysfunction.

@ast

CIKS (Act1 or TRAF3IP2) mediates high glucose-induced endothelial dysfunction.

@en

type

label

CIKS (Act1 or TRAF3IP2) mediates high glucose-induced endothelial dysfunction.

@ast

CIKS (Act1 or TRAF3IP2) mediates high glucose-induced endothelial dysfunction.

@en

prefLabel

CIKS (Act1 or TRAF3IP2) mediates high glucose-induced endothelial dysfunction.

@ast

CIKS (Act1 or TRAF3IP2) mediates high glucose-induced endothelial dysfunction.

@en

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J.CELLSIG.2012.10.009

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Cellular Signalling

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CIKS (Act1 or TRAF3IP2) mediates high glucose-induced endothelial dysfunction

@en

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Andrea J Carpenter

http://www.w3.org/2001/XMLSchema#string

Anthony J Valente

http://www.w3.org/2001/XMLSchema#string

Balachandar Venkatesan

http://www.w3.org/2001/XMLSchema#string

Bysani Chandrasekar

http://www.w3.org/2001/XMLSchema#string

Jean-Luc Delafontaine

http://www.w3.org/2001/XMLSchema#string

Nitin A Das

http://www.w3.org/2001/XMLSchema#string

Ulrich Siebenlist

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P2860

Osteopontin selectively regulates p70S6K/mTOR phosphorylation leading to NF-kappaB dependent AP-1-mediated ICAM-1 expression in breast cancer cells

CIKS, a connection to Ikappa B kinase and stress-activated protein kinase

A novel role for interleukin-18 in adhesion molecule induction through NF kappa B and phosphatidylinositol (PI) 3-kinase-dependent signal transduction pathways

JNK1: a protein kinase stimulated by UV light and Ha-Ras that binds and phosphorylates the c-Jun activation domain

An endothelial receptor for oxidized low-density lipoprotein

Act1, an NF-kappa B-activating protein

Functional analysis of Nox4 reveals unique characteristics compared to other NADPH oxidases

Nitric oxide inhibits endothelial IL-1[beta]-induced ICAM-1 gene expression at the transcriptional level decreasing Sp1 and AP-1 activity

Angiotensin II enhances AT1-Nox1 binding and stimulates arterial smooth muscle cell migration and proliferation through AT1, Nox1, and interleukin-18.

Identification of Novel Type 1 Diabetes Candidate Genes by Integrating Genome-Wide Association Data, Protein-Protein Interactions, and Human Pancreatic Islet Gene Expression

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359-371

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10.1016/J.CELLSIG.2012.10.009

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1

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25

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Tadashi Yoshida

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2012-10-17T00:00:00Z

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23085260

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3606809

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