CD19 and CD22 expression reciprocally regulates tyrosine phosphorylation of Vav protein during B lymphocyte signaling.
about
Regulatory and signaling properties of the Vav familyVav-2 controls NFAT-dependent transcription in B- but not T-lymphocytesCD22 and Siglec-G in B cell function and toleranceCD22 forms a quaternary complex with SHIP, Grb2, and Shc. A pathway for regulation of B lymphocyte antigen receptor-induced calcium fluxCD22: an inhibitory enigmaAnalysis of tyrosine phosphorylation-dependent interactions between stimulatory effector proteins and the B cell co-receptor CD22Uncoupling CD21 and CD19 of the B-cell coreceptorCD19, CD21, and CD22: multifaceted response regulators of B lymphocyte signal transduction.Enhancement of humoral immunity in mice by coupling pUCpGs10 and aluminium to the HCV recombinant immunogen.A sensitized genetic system for the analysis of murine B lymphocyte signal transduction pathways dependent on Bruton's tyrosine kinase.A c-Myc and surface CD19 signaling amplification loop promotes B cell lymphoma development and progression in mice.CD19-regulated signaling thresholds control peripheral tolerance and autoantibody production in B lymphocytes.Humoral response to herpes simplex virus is complement-dependent.Sialic acids and autoimmune disease.CD21 signaling via C3 regulates Purkinje cell protein 4 expressionTranslational Mini-Review Series on B cell subsets in disease. Transitional B cells in systemic lupus erythematosus and Sjögren's syndrome: clinical implications and effects of B cell-targeted therapies.CD22 and autoimmune disease.Cytoskeletal control of B cell responses to antigens.IgM and stromal cell-associated heparan sulfate/heparin as complement-independent ligands for CD19.CD24 induces changes to the surface receptors of B cell microvesicles with variable effects on their RNA and protein cargo.Loss of N-glycolylneuraminic acid in human evolution. Implications for sialic acid recognition by siglecs.CD19 amplification of B lymphocyte Ca2+ responses: a role for Lyn sequestration in extinguishing negative regulation.CD22 ligation inhibits downstream B cell receptor signaling and Ca(2+) flux upon activation.Modification of ligand-independent B cell receptor tonic signals activates receptor editing in immature B lymphocytes.CD22 Promotes B-1b Cell Responses to T Cell-Independent Type 2 Antigens.Homeostatic 'bystander' proliferation of human peripheral blood B cells in response to polyclonal T-cell stimulation in vitro.
P2860
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P2860
CD19 and CD22 expression reciprocally regulates tyrosine phosphorylation of Vav protein during B lymphocyte signaling.
description
1997 nî lūn-bûn
@nan
1997年の論文
@ja
1997年論文
@yue
1997年論文
@zh-hant
1997年論文
@zh-hk
1997年論文
@zh-mo
1997年論文
@zh-tw
1997年论文
@wuu
1997年论文
@zh
1997年论文
@zh-cn
name
CD19 and CD22 expression recip ...... during B lymphocyte signaling.
@ast
CD19 and CD22 expression recip ...... during B lymphocyte signaling.
@en
type
label
CD19 and CD22 expression recip ...... during B lymphocyte signaling.
@ast
CD19 and CD22 expression recip ...... during B lymphocyte signaling.
@en
prefLabel
CD19 and CD22 expression recip ...... during B lymphocyte signaling.
@ast
CD19 and CD22 expression recip ...... during B lymphocyte signaling.
@en
P2093
P2860
P356
P1476
CD19 and CD22 expression recip ...... during B lymphocyte signaling.
@en
P2093
P2860
P304
13158-13162
P356
10.1073/PNAS.94.24.13158
P407
P577
1997-11-01T00:00:00Z