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The Temporal Evolution of Airways Hyperresponsiveness and Inflammation

The Temporal Evolution of Airways Hyperresponsiveness and Inflammation

http://www.wikidata.org/entity/Q36737202

about

The immune profile associated with acute allergic asthma accelerates clearance of influenza virus Absence of c-Jun NH2-terminal kinase 1 protects against house dust mite-induced pulmonary remodeling but not airway hyperresponsiveness and inflammation.IL-25 induces airways angiogenesis and expression of multiple angiogenic factors in a murine asthma model.Mouse Invariant Monoclonal Antibody NKT14: A Novel Tool to Manipulate iNKT Cell Function In Vivo.Dissecting the inflammatory twitch in allergically inflamed mice Epithelial NF-κB orchestrates house dust mite-induced airway inflammation, hyperresponsiveness, and fibrotic remodeling Antigen-induced mast cell expansion and bronchoconstriction in a mouse model of asthma.The inflammatory twitch as a general strategy for controlling the host response.Ablation of Glutaredoxin-1 Modulates House Dust Mite-Induced Allergic Airways Disease in Mice.Issues determining direct airways hyperresponsiveness in mice.Comprehensive attenuation of IL-25-induced airway hyperresponsiveness, inflammation and remodelling by the PI3K inhibitor LY294002.Characteristics of IL-25 and allergen-induced airway fibrosis in a murine model of asthma.Distinct sustained structural and functional effects of interleukin-33 and interleukin-25 on the airways in a murine asthma surrogate.

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The Temporal Evolution of Airways Hyperresponsiveness and Inflammation

http://www.wikidata.org/entity/Q36737202

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2012 nî lūn-bûn

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2012年の論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年论文

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2012年论文

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2012年论文

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The Temporal Evolution of Airways Hyperresponsiveness and Inflammation

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The Temporal Evolution of Airways Hyperresponsiveness and Inflammation

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The Temporal Evolution of Airways Hyperresponsiveness and Inflammation

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The Temporal Evolution of Airways Hyperresponsiveness and Inflammation

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Journal of allergy & therapy

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The Temporal Evolution of Airways Hyperresponsiveness and Inflammation

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P2093

Erik Riesenfeld

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Gilman B Allen

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Jason Ht Bates

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Lennart Ka Lundblad

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Min Wu

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Steven Aimiand

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P2860

Asthma: clinical expression and molecular mechanisms

Non-eosinophilic asthma: importance and possible mechanisms

Rapid activation of nuclear factor-kappaB in airway epithelium in a murine model of allergic airway inflammation

Eosinophilic Inflammation in Asthma

Inhaled salmeterol and/or fluticasone alters structure/function in a murine model of allergic airways disease.

The late, but not early, asthmatic response is dependent on IL-5 and correlates with eosinophil infiltration.

Eosinophils in asthma and other allergic diseases.

Detrimental effects of albuterol on airway responsiveness requires airway inflammation and is independent of β-receptor affinity in murine models of asthma

Serum amyloid A activates the NLRP3 inflammasome and promotes Th17 allergic asthma in mice.

Chronic inhaled ovalbumin exposure induces antigen-dependent but not antigen-specific inhalational tolerance in a murine model of allergic airway disease.

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1-7

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scholarly article

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10.4172/2155-6121.S1-005

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5

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1

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Matthew E Poynter

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2012-01-01T00:00:00Z

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23565340

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3615437

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