Strictly co-isogenic C57BL/6J-Prnp-/- mice: A rigorous resource for prion science. - Wikidata - wikimedia - TriplyDB

Strictly co-isogenic C57BL/6J-Prnp-/- mice: A rigorous resource for prion science.

Strictly co-isogenic C57BL/6J-Prnp-/- mice: A rigorous resource for prion science.

http://www.wikidata.org/entity/Q36745030

about

NCAM1 Polysialylation: The Prion Protein's Elusive Reason for Being?The prion protein is an agonistic ligand of the G protein-coupled receptor Adgrg6 The biological function of the cellular prion protein: an update Loss of prion protein induces a primed state of type I interferon-responsive genes.Cystatin F is a biomarker of prion pathogenesis in mice.Prion pathogenesis is unaltered in the absence of SIRPα-mediated "don't-eat-me" signaling.Prion disease: experimental models and reality.Physiological Functions of the Cellular Prion Protein.The cellular prion protein (PrPC) as neuronal receptor for α-synuclein.Structural and mechanistic aspects influencing the ADAM10-mediated shedding of the prion protein.Extended characterization of the novel co-isogenic C57BL/6J Prnp-/- mouse line.Strictly co-isogenic C57BL/6J-Prnp −/− mice: A rigorous resource for prion science.Lymphocyte activation gene 3 (Lag3) expression is increased in prion infections but does not modify disease progression

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Strictly co-isogenic C57BL/6J-Prnp-/- mice: A rigorous resource for prion science.

http://www.wikidata.org/entity/Q36745030

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2016 nî lūn-bûn

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Strictly co-isogenic C57BL/6J-Prnp-/- mice: A rigorous resource for prion science.

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Strictly co-isogenic C57BL/6J-Prnp-/- mice: A rigorous resource for prion science.

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Strictly co-isogenic C57BL/6J-Prnp-/- mice: A rigorous resource for prion science.

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Strictly co-isogenic C57BL/6J-Prnp-/- mice: A rigorous resource for prion science.

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Strictly co-isogenic C57BL/6J-Prnp-/- mice: A rigorous resource for prion science.

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Strictly co-isogenic C57BL/6J-Prnp-/- mice: A rigorous resource for prion science.

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Strictly co-isogenic C57BL/6J-Prnp-/- mice: A rigorous resource for prion science.

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Cinzia Tiberi

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A Mouse for All Reasons

Accurate normalization of real-time quantitative RT-PCR data by geometric averaging of multiple internal control genes

Involvement of PrP(C) in kainate-induced excitotoxicity in several mouse strains.

edgeR: a Bioconductor package for differential expression analysis of digital gene expression data

Cmr1/WDR76 defines a nuclear genotoxic stress body linking genome integrity and protein quality control.

Ataxia in prion protein (PrP)-deficient mice is associated with upregulation of the novel PrP-like protein doppel

RNA editing in brain controls a determinant of ion flow in glutamate-gated channels

A flanking gene problem leads to the discovery of a Gprc5b splice variant predominantly expressed in C57Bl/6J mouse brain and in maturing neurons

A framework for variation discovery and genotyping using next-generation DNA sequencing data

Integrative genomics viewer

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10.1084/JEM.20151610

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3

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Prion protein family

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