B7-1 Is Not Induced in Podocytes of Human and Experimental Diabetic Nephropathy.
about
FSGS: Diagnosis and Diagnostic Work-UpAbatacept Treatment Does Not Preserve Renal Function in the Streptozocin-Induced Model of Diabetic NephropathyThe costimulatory receptor B7-1 is not induced in injured podocytes.Tubular B7-1 expression parallels proteinuria levels, but not clinical outcomes in adult minimal change disease patients.Targeting the podocyte cytoskeleton: from pathogenesis to therapy in proteinuric kidney disease.B7-1 Blockade Does Not Improve Post-Transplant Nephrotic Syndrome Caused by Recurrent FSGS.Podocyte Expression of B7-1/CD80: Is it a Reliable Biomarker for the Treatment of Proteinuric Kidney Diseases with Abatacept?Interaction of CD80 with Neph1: a potential mechanism of podocyte injury.Urinary CD80: a biomarker for a favorable response to corticosteroids in minimal change disease.Understanding Podocyte Biology to Develop Novel Kidney Therapeutics
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P2860
B7-1 Is Not Induced in Podocytes of Human and Experimental Diabetic Nephropathy.
description
2015 nî lūn-bûn
@nan
2015年の論文
@ja
2015年論文
@yue
2015年論文
@zh-hant
2015年論文
@zh-hk
2015年論文
@zh-mo
2015年論文
@zh-tw
2015年论文
@wuu
2015年论文
@zh
2015年论文
@zh-cn
name
B7-1 Is Not Induced in Podocytes of Human and Experimental Diabetic Nephropathy.
@ast
B7-1 Is Not Induced in Podocytes of Human and Experimental Diabetic Nephropathy.
@en
type
label
B7-1 Is Not Induced in Podocytes of Human and Experimental Diabetic Nephropathy.
@ast
B7-1 Is Not Induced in Podocytes of Human and Experimental Diabetic Nephropathy.
@en
prefLabel
B7-1 Is Not Induced in Podocytes of Human and Experimental Diabetic Nephropathy.
@ast
B7-1 Is Not Induced in Podocytes of Human and Experimental Diabetic Nephropathy.
@en
P2093
P2860
P356
P1476
B7-1 Is Not Induced in Podocytes of Human and Experimental Diabetic Nephropathy.
@en
P2093
Barbara Ruggiero
Carlamaria Zoja
Daniela Corna
Elena Gagliardini
Rubina Novelli
P2860
P304
P356
10.1681/ASN.2015030266
P577
2015-08-28T00:00:00Z