The cytosolic pattern recognition receptor NOD1 induces inflammatory interleukin-8 during Chlamydia trachomatis infection.
about
cPLA2 regulates the expression of type I interferons and intracellular immunity to Chlamydia trachomatisFunction of Nod-like receptors in microbial recognition and host defenseHost Organelle Hijackers: a similar modus operandi for Toxoplasma gondii and Chlamydia trachomatis: co-infection model as a tool to investigate pathogenesisTherapeutic targeting of NOD1 receptorsTarp regulates early Chlamydia-induced host cell survival through interactions with the human adaptor protein SHC1A Zebrafish Model for Chlamydia Infection with the Obligate Intracellular Pathogen Waddlia chondrophilaChlamydia trachomatis co-opts the FGF2 signaling pathway to enhance infectionThe NOD/RIP2 pathway is essential for host defenses against Chlamydophila pneumoniae lung infection.Induction of interferon-stimulated genes by Chlamydia pneumoniae in fibroblasts is mediated by intracellular nucleotide-sensing receptors.Host chemokine and cytokine response in the endocervix within the first developmental cycle of Chlamydia muridarum.Chlamydial infection of monocytes stimulates IL-1beta secretion through activation of the NLRP3 inflammasomeStimulator of IFN gene is critical for induction of IFN-beta during Chlamydia muridarum infectionWaddlia chondrophila infects and multiplies in ovine trophoblast cells stimulating an inflammatory immune response.Rac1 regulates the NLRP3 inflammasome which mediates IL-1beta production in Chlamydophila pneumoniae infected human mononuclear cells.Enhancement of reactive oxygen species production and chlamydial infection by the mitochondrial Nod-like family member NLRX1.MAPK Activation Is Essential for Waddlia chondrophila Induced CXCL8 Expression in Human Epithelial Cells.Structural characterization of muropeptides from Chlamydia trachomatis peptidoglycan by mass spectrometry resolves "chlamydial anomaly".Nod1, but not the ASC inflammasome, contributes to induction of IL-1β secretion in human trophoblasts after sensing of Chlamydia trachomatisReceptor interacting protein-2 contributes to host defense against Anaplasma phagocytophilum infection.Prevention of Chlamydia-induced infertility by inhibition of local caspase activity.Chlamydial intracellular survival strategies.Severity of allergic airway disease due to house dust mite allergen is not increased after clinical recovery of lung infection with Chlamydia pneumoniae in mice.Killing me softly: chlamydial use of proteolysis for evading host defenses.Pathogen recognition in the innate immune response.Lactobacillus plantarum lipoteichoic acid alleviates TNF-α-induced inflammation in the HT-29 intestinal epithelial cell line.IL-8 promotes HNSCC progression on CXCR1/2-meidated NOD1/RIP2 signaling pathway.The complement C3a receptor is critical in defense against Chlamydia psittaci in mouse lung infection and required for antibody and optimal T cell response.Host immune responses after hypoxic reactivation of IFN-γ induced persistent Chlamydia trachomatis infection.Chlamydiales and the innate immune response: friend or foe?Infection of epithelial cells with Chlamydia trachomatis inhibits TNF-induced apoptosis at the level of receptor internalization while leaving non-apoptotic TNF-signalling intact.The role of NOD1 and NOD2 in host defense against chlamydial infection.Immunopathogenesis of Chlamydial Infections.Progesterone activates multiple innate immune pathways in Chlamydia trachomatis-infected endocervical cells.The multifaceted role of oestrogen in enhancing Chlamydia trachomatis infection in polarized human endometrial epithelial cells.Production of reactive oxygen species is turned on and rapidly shut down in epithelial cells infected with Chlamydia trachomatisActin and intermediate filaments stabilize the Chlamydia trachomatis vacuole by forming dynamic structural scaffolds.Peptidoglycan: a critical activator of the mammalian immune system during infection and homeostasis.Recognition of bacterial infection by innate immune sensors.The Interplay between NLRs and Autophagy in Immunity and Inflammation.Glycemic Status and Infection Risk in Nondiabetic Autologous Hematopoietic Cell Transplantation Recipients.
P2860
Q24618182-8E3BCA93-E870-492A-A84A-08E26AC7AD5CQ24647448-752F40B3-5E0F-415D-9613-5B8DE4422315Q26825487-D3F6E63B-8586-45DD-AB8E-7ED54D55249AQ28676201-460D87FD-B7DF-49BE-A5CE-5AE9BF31A300Q28902456-6848D576-F7F9-40D3-96A6-8A163ABC02E6Q30829026-ABE335C4-D326-4022-8A64-B294789EA6BAQ31036531-64DE0337-8203-43EC-B38E-CE85E337AC6DQ33429029-D53FFE8A-933C-4571-8B54-069A5D816B98Q33553163-B9D8797A-440C-403C-BC13-3AA801D99107Q33557924-00A1A80A-3A81-4538-8D05-CB6820CB037AQ33817314-B446A065-E09E-4E56-89F9-D6B778034B43Q33824328-9174B884-AFC7-4CE7-B2CF-992139135D07Q33882781-F9B819A5-44AF-457A-96FD-71769A74F294Q34140548-F8E0F707-DBBF-46C1-9009-415967D5F263Q34144581-1A8260A4-788A-4BE8-AB9D-86B95F3227B4Q35965894-219D9474-3E6A-4D41-A2B7-D53F4416BCD3Q36079175-6E25DFFF-4804-44A2-94C1-2C322A9A4A67Q36301439-207143C2-EEA2-45DB-BAE3-CFCC3F8F3E96Q36488241-C1A3EC5C-7F94-47EE-8EAA-ED780F2B354BQ36641653-D1804297-244C-4ECA-AB0A-F0D6270E45B2Q36786613-452CBDCA-C59B-44F5-83D1-6FE31D622253Q37123769-19886F49-6A5E-47CA-BD9F-9A5EC3370990Q37371394-9DE1EFE4-0C1E-4515-A680-B8B25578BDCAQ37453504-C70EA0EC-112F-4E13-B0CD-0F90FDF17A3CQ37461581-EB114354-EC3E-4D52-B24A-EA37F1B886D6Q37641690-5AF4B075-E5AC-4F82-A3A3-AEAB3B477961Q37671928-FD5B38F0-E359-44CE-AB8B-CD174FB8CA3DQ37718968-076B07DE-E0DE-4A7B-A79D-EFB9142BA6FEQ37825933-56531307-4FE6-4ED3-9D34-6F981A02F25FQ37835512-831F6241-0C46-4577-B7E3-B20B29A09BB9Q37835976-DEA736D0-1997-49D0-9893-36333E915AE2Q37836040-8F0C2639-BA8C-418A-A9E3-E15CA3972ADAQ37845585-5340ADE0-C727-4F9D-AE13-4A7DA9F6E8AEQ37850835-1BF5D979-1977-4FD5-9EF8-AA3B2CBFD7DAQ37854112-9292666F-BB8A-4780-84DF-5B1031CD01EDQ37856853-7EEFA5E5-096C-478A-B86A-B663EB43F7A4Q37925062-E6BE9BCE-AFB6-434A-84D7-C10302FC25C2Q38032312-4AB66EC7-951B-46CF-8623-03166BEE445AQ38165934-606BC179-E0E0-456D-BC30-B00B59B28D1AQ40823795-848DAA61-20C3-4B55-AA1C-6D2AF376DBB0
P2860
The cytosolic pattern recognition receptor NOD1 induces inflammatory interleukin-8 during Chlamydia trachomatis infection.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
2008年论文
@zh
2008年论文
@zh-cn
name
The cytosolic pattern recognit ...... lamydia trachomatis infection.
@en
type
label
The cytosolic pattern recognit ...... lamydia trachomatis infection.
@en
prefLabel
The cytosolic pattern recognit ...... lamydia trachomatis infection.
@en
P2860
P356
P1476
The cytosolic pattern recognit ...... lamydia trachomatis infection.
@en
P2093
Kerry R Buchholz
Richard S Stephens
P2860
P304
P356
10.1128/IAI.00104-08
P407
P577
2008-04-21T00:00:00Z