Infliximab and etanercept are equally effective in reducing enterocyte APOPTOSIS in experimental colitis
about
Fc gamma receptor CD64 modulates the inhibitory activity of infliximabBalsalazine decreases intestinal mucosal permeability of dextran sulfate sodium-induced colitis in mice.Etanercept protects rat cardiomyocytes against hypertrophy by regulating inflammatory cytokines secretion and cell apoptosisTumour necrosis factor superfamily members in the pathogenesis of inflammatory bowel diseaseAnti-TNF-alpha therapy does not ameliorate disease in a model of acute virus-endotoxin mediated respiratory disease in pigs.Etanercept biosimilarsAblation of tumor necrosis factor is associated with decreased inflammation and alterations of the microbiota in a mouse model of inflammatory bowel disease.Loss of ADAM17-Mediated Tumor Necrosis Factor Alpha Signaling in Intestinal Cells Attenuates Mucosal Atrophy in a Mouse Model of Parenteral NutritionClinically validated approaches to the treatment of autoimmune diseases.Apoptosis and the FLIP and NF-kappa B proteins as pharmacodynamic criteria for biosimilar TNF-alpha antagonists.Dendritic cells expressing immunoreceptor CD300f are critical for controlling chronic gut inflammationMolecular pathways driving disease-specific alterations of intestinal epithelial cells.The combination of a tumor necrosis factor inhibitor and antibiotic alleviates staphylococcal arthritis and sepsis in mice.Anti-inflammatory effects of infliximab in mice are independent of tumour necrosis factor α neutralization.How bacteria-induced apoptosis of intestinal epithelial cells contributes to mucosal inflammation.Antibiotic-killed Staphylococcus aureus induces destructive arthritis in mice.Myosin light chain kinase inhibitor inhibits dextran sulfate sodium-induced colitis in mice.The Cytosolic Microbial Receptor Nod2 Regulates Small Intestinal Crypt Damage and Epithelial Regeneration following T Cell-Induced Enteropathy.Intrarectal administration of oxygenated perfluorodecalin promotes healing of murine colitis by targeting inflammatory hypoxia.TNFα depleting therapy improves fertility and animal welfare in TNFα-driven transgenic models of polyarthritis when administered in their routine breeding.Tumour necrosis factor--induced loss of intestinal barrier function requires TNFR1 and TNFR2 signalling in a mouse model of total parenteral nutrition.Etanercept attenuates TNBS-induced experimental colitis: role of TNF-α expression.
P2860
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P2860
Infliximab and etanercept are equally effective in reducing enterocyte APOPTOSIS in experimental colitis
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年論文
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2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
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2008年论文
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2008年论文
@zh-cn
name
Infliximab and etanercept are ...... PTOSIS in experimental colitis
@ast
Infliximab and etanercept are ...... PTOSIS in experimental colitis
@en
type
label
Infliximab and etanercept are ...... PTOSIS in experimental colitis
@ast
Infliximab and etanercept are ...... PTOSIS in experimental colitis
@en
prefLabel
Infliximab and etanercept are ...... PTOSIS in experimental colitis
@ast
Infliximab and etanercept are ...... PTOSIS in experimental colitis
@en
P2093
P2860
P356
P1476
Infliximab and etanercept are ...... PTOSIS in experimental colitis
@en
P2093
Carmela Crisafulli
Carmelo Muja
Giuseppe Costantino
Giuseppe Longo
Walter Fries
P2860
P304
P356
10.7150/IJMS.5.169
P407
P577
2008-07-03T00:00:00Z