The combination of a tumor necrosis factor inhibitor and antibiotic alleviates staphylococcal arthritis and sepsis in mice.
about
Whole-body deletion of LPS-induced TNF-α factor (LITAF) markedly improves experimental endotoxic shock and inflammatory arthritisSepsis: in search of cure.Septic arthritis: immunopathogenesis, experimental models and therapyHMGB1 in health and disease.IL-1 Receptor Antagonist Treatment Aggravates Staphylococcal Septic Arthritis and Sepsis in Mice.RAGE Deficiency Impairs Bacterial Clearance in Murine Staphylococcal Sepsis, but Has No Significant Impact on Staphylococcal Septic ArthritisRadiological features of experimental staphylococcal septic arthritis by micro computed tomography scanBoth anti-TNF and CTLA4 Ig treatments attenuate the disease severity of staphylococcal dermatitis in mice.Sulfatide attenuates experimental Staphylococcus aureus sepsis through a CD1d-dependent pathway.Staphylococcal Protein A Promotes Colonization and Immune Evasion of the Epidemic Healthcare-Associated MRSA ST239Shedding of tumor necrosis factor receptor 1 induced by protein A decreases tumor necrosis factor alpha availability and inflammation during systemic Staphylococcus aureus infection.Novel long-chain compounds with both immunomodulatory and MenA inhibitory activities against Staphylococcus aureus and its biofilm.Anti-inflammatory effects of infliximab in mice are independent of tumour necrosis factor α neutralization.Deficiency of the Complement Component 3 but Not Factor B Aggravates Staphylococcus aureus Septic Arthritis in Mice.Neutralization of MMP-2 and TNFR1 Regulates the Severity of S. aureus-Induced Septic Arthritis by Differential Alteration of Local and Systemic Proinflammatory Cytokines in Mice.Riboflavin along with antibiotics balances reactive oxygen species and inflammatory cytokines and controls Staphylococcus aureus infection by boosting murine macrophage function and regulates inflammation.Cloxacillin control of experimental arthritis induced by SEC(+) Staphylococcus aureus is associated with downmodulation of local and systemic cytokines.CTLA4 Immunoglobulin but Not Anti-Tumor Necrosis Factor Therapy Promotes Staphylococcal Septic Arthritis in Mice.Antibiotic-killed Staphylococcus aureus induces destructive arthritis in mice.Inflammation, myocardial dysfunction, and mortality in children with septic shock: an observational study.CXCR2 is critical for bacterial control and development of joint damage and pain in Staphylococcus aureus-induced septic arthritis in mouse.Dual neutralization of TNFR-2 and MMP-2 regulates the severity of S. aureus induced septic arthritis correlating alteration in the level of interferon gamma and interleukin-10 in terms of TNFR2 blocking.Antibiotics with Interleukin-15 inhibition reduces joint inflammation and bone erosions but not cartilage destruction in Staphylococcus aureus-induced arthritis.Infliximab alleviates the mortality, mesenteric hypoperfusion, aortic dysfunction, and multiple organ damage in septic rats.
P2860
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P2860
The combination of a tumor necrosis factor inhibitor and antibiotic alleviates staphylococcal arthritis and sepsis in mice.
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年学术文章
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2011年学术文章
@zh-cn
2011年学术文章
@zh-hans
2011年学术文章
@zh-my
2011年学术文章
@zh-sg
2011年學術文章
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2011年學術文章
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2011年學術文章
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name
The combination of a tumor nec ...... arthritis and sepsis in mice.
@en
The combination of a tumor nec ...... arthritis and sepsis in mice.
@nl
type
label
The combination of a tumor nec ...... arthritis and sepsis in mice.
@en
The combination of a tumor nec ...... arthritis and sepsis in mice.
@nl
prefLabel
The combination of a tumor nec ...... arthritis and sepsis in mice.
@en
The combination of a tumor nec ...... arthritis and sepsis in mice.
@nl
P2093
P2860
P50
P356
P1476
The combination of a tumor nec ...... arthritis and sepsis in mice.
@en
P2093
Elisabet Josefsson
Ing-Marie Jonsson
Wanzhong Wang
P2860
P304
P356
10.1093/INFDIS/JIR266
P407
P577
2011-08-01T00:00:00Z