RAGE and modulation of ischemic injury in the diabetic myocardium - Wikidata - wikimedia - TriplyDB

RAGE and modulation of ischemic injury in the diabetic myocardium

RAGE and modulation of ischemic injury in the diabetic myocardium

http://www.wikidata.org/entity/Q36756482

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RAGE modulates hypoxia/reoxygenation injury in adult murine cardiomyocytes via JNK and GSK-3beta signaling pathways.Molecular Pathways Involved in the Amelioration of Myocardial Injury in Diabetic Rats by Kaempferol.The RAGE axis: a fundamental mechanism signaling danger to the vulnerable vasculature S100/Calgranulin-mediated inflammation accelerates left ventricular hypertrophy and aortic valve sclerosis in chronic kidney disease in a receptor for advanced glycation end products-dependent manner Advanced glycation end product (AGE)-receptor for AGE (RAGE) signaling and up-regulation of Egr-1 in hypoxic macrophages Soluble RAGE: therapy and biomarker in unraveling the RAGE axis in chronic disease and aging Exendin-4, a glucagon-like peptide-1 receptor agonist, inhibits hyperglycemia-induced apoptosis in myocytes by suppressing receptor for advanced glycation end products expression Nuclear DAMP complex-mediated RAGE-dependent macrophage cell death.Small Molecule Inhibition of Ligand-Stimulated RAGE-DIAPH1 Signal Transduction Mitochondrial ROS Induces Cardiac Inflammation via a Pathway through mtDNA Damage in a Pneumonia-Related Sepsis Model.Oxidative stress contributes to the impaired sonic hedgehog pathway in type 1 diabetic mice with myocardial infarction.Imaging of receptors for advanced glycation end products in experimental myocardial ischemia and reperfusion injury Insulin resistance: metabolic mechanisms and consequences in the heart.Radical roles for RAGE in the pathogenesis of oxidative stress in cardiovascular diseases and beyond.Losartan protects liver against ischaemia/reperfusion injury through PPAR-γ activation and receptor for advanced glycation end-products down-regulation.Receptor for AGE (RAGE) and its ligands-cast into leading roles in diabetes and the inflammatory response The receptor for advanced glycation endproducts (RAGE) and cardiovascular disease.Tempering the wrath of RAGE: an emerging therapeutic strategy against diabetic complications, neurodegeneration, and inflammation Non-histone nuclear factor HMGB1 as a therapeutic target in colorectal cancer.Receptor for AGE (RAGE): signaling mechanisms in the pathogenesis of diabetes and its complications.Receptor for advanced glycation end products (RAGE) and implications for the pathophysiology of heart failure.The diverse ligand repertoire of the receptor for advanced glycation endproducts and pathways to the complications of diabetes.Advanced glycation end products: role in pathology of diabetic cardiomyopathy.Deoxycholic acid-modified polyethylenimine based nanocarriers for RAGE siRNA therapy in acute myocardial infarction.Damaging effects of hyperglycemia on cardiovascular function: spotlight on glucose metabolic pathways.Maillard reaction in food allergy: Pros and cons.Implication of advanced glycation end products (Ages) and their receptor (Rage) on myocardial contractile and mitochondrial functions.Glycation & the RAGE axis: targeting signal transduction through DIAPH1.Advanced glycation end products (AGEs) and cardiovascular dysfunction: focus on high molecular weight AGEs.The Effects of Diabetes Induction on the Rat Heart: Differences in Oxidative Stress, Inflammatory Cells, and Fibrosis between Subendocardial and Interstitial Myocardial Areas.Effects of dietary arginine on inflammatory mediator and receptor of advanced glycation endproducts (RAGE) expression in rats with streptozotocin-induced type 2 diabetes.Stopping the primal RAGE reaction in myocardial infarction: capturing adaptive responses to heal the heart?Myocyte membrane and microdomain modifications in diabetes: determinants of ischemic tolerance and cardioprotection.Tiron ameliorates high glucose-induced cardiac myocyte apoptosis by PKCδ-dependent inhibition of osteopontin.Hyperglycemia and liver ischemia reperfusion injury: a role for the advanced glycation endproduct and its receptor pathway.A soluble receptor for advanced glycation end-products inhibits myocardial apoptosis induced by ischemia/reperfusion via the JAK2/STAT3 pathway.Ager Deletion Enhances Ischemic Muscle Inflammation, Angiogenesis, and Blood Flow Recovery in Diabetic Mice.Molecular mechanism of diabetic cardiomyopathy and modulation of microRNA function by synthetic oligonucleotides.Perspectives on RAGE signaling and its role in cardiovascular disease.S100A8/A9 aggravates post-ischemic heart failure through activation of RAGE-dependent NF-κB signaling.

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RAGE and modulation of ischemic injury in the diabetic myocardium

http://www.wikidata.org/entity/Q36756482

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2008 nî lūn-bûn

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2008年の論文

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2008年論文

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2008年論文

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2008年論文

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2008年论文

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2008年论文

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name

RAGE and modulation of ischemic injury in the diabetic myocardium

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RAGE and modulation of ischemic injury in the diabetic myocardium

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RAGE and modulation of ischemic injury in the diabetic myocardium

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RAGE and modulation of ischemic injury in the diabetic myocardium

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RAGE and modulation of ischemic injury in the diabetic myocardium

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Ann Marie Schmidt

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Christopher Strauch

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David R Sell

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Fei Song

http://www.w3.org/2001/XMLSchema#string

Loredana G Bucciarelli

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Michiyo Kaneko

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Radha Ananthakrishnan

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Ravichandran Ramasamy

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Shi Fang Yan

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Vincent M Monnier

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P2860

RAGE mediates a novel proinflammatory axis: a central cell surface receptor for S100/calgranulin polypeptides

Inhibition of norepinephrine-induced cardiac hypertrophy in s100beta transgenic mice.

Reduction of myocardial infarct size by inhibition of inducible nitric oxide synthase

N(epsilon)-(carboxymethyl)lysine adducts of proteins are ligands for receptor for advanced glycation end products that activate cell signaling pathways and modulate gene expression

Vascular and inflammatory stresses mediate atherosclerosis via RAGE and its ligands in apoE-/- mice

Cellular basis for the negative inotropic effects of tumor necrosis factor-alpha in the adult mammalian heart.

Novel cardioprotective role of a small heat-shock protein, Hsp20, against ischemia/reperfusion injury.

Tumor necrosis factor-alpha and myocardial remodeling in progression of heart failure: a current perspective.

Central role of RAGE-dependent neointimal expansion in arterial restenosis.

Glycation products and the pathogenesis of diabetic complications.

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10.2337/DB07-0326

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7

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5433172

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18420491

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2453611

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