Tempering the wrath of RAGE: an emerging therapeutic strategy against diabetic complications, neurodegeneration, and inflammation - Wikidata - wikimedia - TriplyDB

Tempering the wrath of RAGE: an emerging therapeutic strategy against diabetic complications, neurodegeneration, and inflammation

Tempering the wrath of RAGE: an emerging therapeutic strategy against diabetic complications, neurodegeneration, and inflammation

http://www.wikidata.org/entity/Q37424029

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Fisetin lowers methylglyoxal dependent protein glycation and limits the complications of diabetes The effects of diabetes on male fertility and epigenetic regulation during spermatogenesis Microglial AGE-albumin is critical in promoting alcohol-induced neurodegeneration in rats and humans RAGE is a nucleic acid receptor that promotes inflammatory responses to DNA Diabetic conditions promote binding of monocytes to vascular smooth muscle cells and their subsequent differentiation Co-expression of BirA with biotin bait achieves in vivo biotinylation of overexpressed stable N-glycosylated sRAGE in transgenic silkworms.LL-37 induced cystitis and the receptor for advanced glycation end-products (RAGE) pathway.Carboxylated N-glycans on RAGE promote S100A12 binding and signaling.Diabetic impairment of C-kit bone marrow stem cells involves the disorders of inflammatory factors, cell adhesion and extracellular matrix molecules Soluble RAGE: therapy and biomarker in unraveling the RAGE axis in chronic disease and aging Diabetes, related conditions, and dementia.Determinants of concentrations of N(ε)-carboxymethyl-lysine and soluble receptor for advanced glycation end products and their associations with risk of pancreatic cancer RAGE regulates immune cell infiltration and angiogenesis in choroidal neovascularization.The role of receptor for advanced glycation end products in airway inflammation in CF and CF related diabetes.Modeling the interaction between quinolinate and the receptor for advanced glycation end products (RAGE): relevance for early neuropathological processes Chronic alcohol administration causes expression of calprotectin and RAGE altering the distribution of zinc ions in mouse testis.Association between the Advanced Glycosylation End Product-Specific Receptor Gene and Cardiovascular Death in Older Men Anti-atherosclerotic effects of sitagliptin in patients with type 2 diabetes mellitus.Effects of RAGE Gene Polymorphisms on the Risk and Progression of Hepatocellular Carcinoma RAGE gene polymorphism and environmental factor in the risk of oral cancer.High-mobility group box-1 induces decreased brain-derived neurotrophic factor-mediated neuroprotection in the diabetic retina.RAGE is a key cellular target for Abeta-induced perturbation in Alzheimer's disease.Adherence to a Mediterranean diet and risk of incident cognitive impairment.The role of inflammatory processes in Alzheimer's disease.HMGB1 in development and diseases of the central nervous system.RAGE regulation and signaling in inflammation and beyond.Unlocking the biology of RAGE in diabetic microvascular complications Critical role of RAGE in lung physiology and tumorigenesis: a potential target of therapeutic intervention?Role of Toll-like receptors in diabetic nephropathy.The pathobiology of diabetic vascular complications--cardiovascular and kidney disease.Aggregation of the Inflammatory S100A8 Precedes Aβ Plaque Formation in Transgenic APP Mice: Positive Feedback for S100A8 and Aβ Productions.The self-association of HMGB1 and its possible role in the binding to DNA and cell membrane receptors.Neurodegeneration and Neuroinflammation in Diabetic Retinopathy: Potential Approaches to Delay Neuronal Loss.Beneficial effects of a novel RAGE inhibitor on early diabetic retinopathy and tactile allodynia.Skin autofluorescence relates to soluble receptor for advanced glycation end-products and albuminuria in diabetes mellitus.Receptor for advanced glycation end-products and World Trade Center particulate induced lung function loss: a case-cohort study and murine model of acute particulate exposure Receptor for Advanced Glycation End Products and its Inflammatory Ligands are Upregulated in Amyotrophic Lateral Sclerosis.RAGE-Mediated Inflammation, Type 2 Diabetes, and Diabetic Vascular Complication.Spontaneous periodontitis development in diabetic rats involves an unrestricted expression of inflammatory cytokines and tissue destructive factors in the absence of major changes in commensal oral microbiota.Expression of high-mobility groups box-1/receptor for advanced glycation end products/osteopontin/early growth response-1 pathway in proliferative vitreoretinal epiretinal membranes.

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Tempering the wrath of RAGE: an emerging therapeutic strategy against diabetic complications, neurodegeneration, and inflammation

http://www.wikidata.org/entity/Q37424029

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bilimsel makale

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scientific article published on January 2009

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Tempering the wrath of RAGE: a ...... degeneration, and inflammation

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Tempering the wrath of RAGE: a ...... degeneration, and inflammation

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Annals of Medicine

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Tempering the wrath of RAGE: a ...... degeneration, and inflammation

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Ann Marie Schmidt

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Ravichandran Ramasamy

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Shi Du Yan

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Shi Fang Yan

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P2860

Receptor for advanced glycation end products (RAGE)-mediated neurite outgrowth and activation of NF-kappaB require the cytoplasmic domain of the receptor but different downstream signaling pathways

RAGE mediates a novel proinflammatory axis: a central cell surface receptor for S100/calgranulin polypeptides

RAGE and amyloid-beta peptide neurotoxicity in Alzheimer's disease

S100 protein translocation in response to extracellular S100 is mediated by receptor for advanced glycation endproducts in human endothelial cells

Loss of pain perception in diabetes is dependent on a receptor of the immunoglobulin superfamily

RAGE, carboxylated glycans and S100A8/A9 play essential roles in colitis-associated carcinogenesis

Neutrophil derived human S100A12 (EN-RAGE) is strongly expressed during chronic active inflammatory bowel disease

Release of high mobility group box 1 by dendritic cells controls T cell activation via the receptor for advanced glycation end products

Release of chromatin protein HMGB1 by necrotic cells triggers inflammation

Requirement of HMGB1 and RAGE for the maturation of human plasmacytoid dendritic cells

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10.1080/07853890902806576

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6

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41

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2009-01-01T00:00:00Z

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19322705

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