The Nlrp3 inflammasome, IL-1β, and neutrophil recruitment are required for susceptibility to a nonhealing strain of Leishmania major in C57BL/6 mice.
about
CD8+ T cell cytotoxicity mediates pathology in the skin by inflammasome activation and IL-1β production.Clash of the Cytokine Titans: counter-regulation of interleukin-1 and type I interferon-mediated inflammatory responses.Immune responses against protozoan parasites: a focus on the emerging role of Nod-like receptors.AIM2 inflammasome is associated with disease severity in tegumentary leishmaniasis caused by Leishmania (V.) braziliensis.Pro- and anti-inflammatory cytokines in cutaneous leishmaniasis: a review.Molecular mechanisms and functions of pyroptosis, inflammatory caspases and inflammasomes in infectious diseases.Immunomodulatory nanoparticles ameliorate disease in the Leishmania (Viannia) panamensis mouse model.Dectin-1 Activation during Leishmania amazonensis Phagocytosis Prompts Syk-Dependent Reactive Oxygen Species Production To Trigger Inflammasome Assembly and Restriction of Parasite Replication.Cutaneous Leishmaniasis Induces a Transmissible Dysbiotic Skin Microbiota that Promotes Skin Inflammation.How inflammasomes inform adaptive immunity.Immune Checkpoint Targets for Host-Directed Therapy to Prevent and Treat Leishmaniasis.Survival Mechanisms Used by Some Leishmania Species to Escape Neutrophil Killing.The NOD2 receptor is crucial for immune responses towards New World Leishmania species.IL-1β production by intermediate monocytes is associated with immunopathology in cutaneous leishmaniasis.An Anti-Inflammatory Role for NLRP10 in Murine Cutaneous Leishmaniasis.Gut Microbes Egested during Bites of Infected Sand Flies Augment Severity of Leishmaniasis via Inflammasome-Derived IL-1β.Mannose receptor high, M2 dermal macrophages mediate nonhealing Leishmania major infection in a Th1 immune environment.How to master the host immune system? Leishmania parasites have the solutions!Leishmania guyanensis parasites block the activation of the inflammasome by inhibiting maturation of IL-1β.
P2860
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P2860
The Nlrp3 inflammasome, IL-1β, and neutrophil recruitment are required for susceptibility to a nonhealing strain of Leishmania major in C57BL/6 mice.
description
2015 nî lūn-bûn
@nan
2015年の論文
@ja
2015年論文
@yue
2015年論文
@zh-hant
2015年論文
@zh-hk
2015年論文
@zh-mo
2015年論文
@zh-tw
2015年论文
@wuu
2015年论文
@zh
2015年论文
@zh-cn
name
The Nlrp3 inflammasome, IL-1β, ...... shmania major in C57BL/6 mice.
@ast
The Nlrp3 inflammasome, IL-1β, ...... shmania major in C57BL/6 mice.
@en
type
label
The Nlrp3 inflammasome, IL-1β, ...... shmania major in C57BL/6 mice.
@ast
The Nlrp3 inflammasome, IL-1β, ...... shmania major in C57BL/6 mice.
@en
prefLabel
The Nlrp3 inflammasome, IL-1β, ...... shmania major in C57BL/6 mice.
@ast
The Nlrp3 inflammasome, IL-1β, ...... shmania major in C57BL/6 mice.
@en
P2093
P2860
P356
P1476
The Nlrp3 inflammasome, IL-1β, ...... shmania major in C57BL/6 mice.
@en
P2093
Audrey Romano
Benjamin P Hurrell
David L Sacks
Flavia Ribeiro-Gomes
Katrin Mayer-Barber
Melanie Charmoy
Nicolas Riteau
Sang Hun Lee
P2860
P304
P356
10.1002/EJI.201546015
P407
P577
2015-12-22T00:00:00Z