Efficient capture of infected neutrophils by dendritic cells in the skin inhibits the early anti-leishmania response.
about
Lights, camera, and action: vertebrate skin sets the stage for immune cell interaction with arthropod-vectored pathogensEscaping Deleterious Immune Response in Their Hosts: Lessons from TrypanosomatidsThe site of the bite: Leishmania interaction with macrophages, neutrophils and the extracellular matrix in the dermisDendritic Cells and Leishmania Infection: Adding Layers of Complexity to a Complex DiseaseThe influence of early neutrophil-Leishmania interactions on the host immune response to infectionMechanisms of cellular invasion by intracellular parasitesReceptor-mediated phagocytosis of Leishmania: implications for intracellular survivalDeterminants for the development of visceral leishmaniasis diseaseLeishmania amazonensis Engages CD36 to Drive Parasitophorous Vacuole MaturationBCG Skin Infection Triggers IL-1R-MyD88-Dependent Migration of EpCAMlow CD11bhigh Skin Dendritic cells to Draining Lymph Node During CD4+ T-Cell PrimingRapid Sequestration of Leishmania mexicana by Neutrophils Contributes to the Development of Chronic LesionNeutrophil recruitment to lymph nodes limits local humoral response to Staphylococcus aureusChronic parasitic infection maintains high frequencies of short-lived Ly6C+CD4+ effector T cells that are required for protection against re-infectionCrosstalk between purinergic receptors and lipid mediators in leishmaniasisNeutrophils: Between host defence, immune modulation, and tissue injuryAppraisal of a Leishmania major strain stably expressing mCherry fluorescent protein for both in vitro and in vivo studies of potential drugs and vaccine against cutaneous leishmaniasisSkin-resident CD4+ T cells protect against Leishmania major by recruiting and activating inflammatory monocytes.Neutrophil Extracellular Traps Reprogram IL-4/GM-CSF-Induced Monocyte Differentiation to Anti-inflammatory Macrophages.A secondary wave of neutrophil infiltration causes necrosis and ulceration in lesions of experimental American cutaneous leishmaniasisCutaneous Manifestations of Human and Murine LeishmaniasisSite-dependent recruitment of inflammatory cells determines the effective dose of Leishmania majorInnate immunity to Leishmania infection: within phagocytes.Deception and manipulation: the arms of leishmania, a successful parasiteBlocking junctional adhesion molecule C enhances dendritic cell migration and boosts the immune responses against Leishmania major.B cells regulate neutrophilia during Mycobacterium tuberculosis infection and BCG vaccination by modulating the interleukin-17 response.Neutrophils have a protective role during early stages of Leishmania amazonensis infection in BALB/c mice.Characterization of the early inflammatory infiltrate at the feeding site of infected sand flies in mice protected from vector-transmitted Leishmania major by exposure to uninfected bites.Exposure to Leishmania braziliensis triggers neutrophil activation and apoptosis.TACI deficiency leads to alternatively activated macrophage phenotype and susceptibility to Leishmania infection.Prison break: pathogens' strategies to egress from host cells.Apoptotic cell clearance of Leishmania major-infected neutrophils by dendritic cells inhibits CD8⁺ T-cell priming in vitro by Mer tyrosine kinase-dependent signalingEvaluation of recombinant Leishmania polyprotein plus glucopyranosyl lipid A stable emulsion vaccines against sand fly-transmitted Leishmania major in C57BL/6 miceThe Nlrp3 inflammasome, IL-1β, and neutrophil recruitment are required for susceptibility to a nonhealing strain of Leishmania major in C57BL/6 mice.Neutrophils mediate immunopathology and negatively regulate protective immune responses during fatal bacterial infection-induced toxic shock.Apoptotic CD8 T-lymphocytes disable macrophage-mediated immunity to Trypanosoma cruzi infection.BAFF-secreting neutrophils drive plasma cell responses during emergency granulopoiesis.Leishmania amazonensis amastigotes trigger neutrophil activation but resist neutrophil microbicidal mechanisms.Inhibition of caspase-8 activity promotes protective Th1- and Th2-mediated immunity to Leishmania major infection.Macrophages clean up: efferocytosis and microbial control.Differential ability of surface and endosomal TLRs to induce CD8 T cell responses in vivo
P2860
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P2860
Efficient capture of infected neutrophils by dendritic cells in the skin inhibits the early anti-leishmania response.
description
2012 nî lūn-bûn
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2012 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2012 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
name
Efficient capture of infected ...... arly anti-leishmania response.
@ast
Efficient capture of infected ...... arly anti-leishmania response.
@en
Efficient capture of infected ...... arly anti-leishmania response.
@nl
type
label
Efficient capture of infected ...... arly anti-leishmania response.
@ast
Efficient capture of infected ...... arly anti-leishmania response.
@en
Efficient capture of infected ...... arly anti-leishmania response.
@nl
prefLabel
Efficient capture of infected ...... arly anti-leishmania response.
@ast
Efficient capture of infected ...... arly anti-leishmania response.
@en
Efficient capture of infected ...... arly anti-leishmania response.
@nl
P2093
P2860
P1433
P1476
Efficient capture of infected ...... arly anti-leishmania response.
@en
P2093
Alain Debrabant
David L Sacks
Flavia L Ribeiro-Gomes
Nathan C Peters
P2860
P304
P356
10.1371/JOURNAL.PPAT.1002536
P577
2012-02-16T00:00:00Z