Acute Fibrinolysis Shutdown after Injury Occurs Frequently and Increases Mortality: A Multicenter Evaluation of 2,540 Severely Injured Patients. - Wikidata - wikimedia - TriplyDB

Acute Fibrinolysis Shutdown after Injury Occurs Frequently and Increases Mortality: A Multicenter Evaluation of 2,540 Severely Injured Patients.

Acute Fibrinolysis Shutdown after Injury Occurs Frequently and Increases Mortality: A Multicenter Evaluation of 2,540 Severely Injured Patients.

http://www.wikidata.org/entity/Q36807586

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The current understanding of trauma-induced coagulopathy (TIC): a focused review on pathophysiology Advances in the understanding of trauma-induced coagulopathy Tranexamic acid is associated with increased mortality in patients with physiological fibrinolysis.The role of NIGMS P50 sponsored team science in our understanding of multiple organ failure.Relative effects of plasma, fibrinogen concentrate, and factor XIII on ROTEM coagulation profiles in an in vitro model of massive transfusion in trauma.[Uncritical use of tranexamic acid in trauma patients : Do no further harm!]Hemorrhagic blood failure: Oxygen debt, coagulopathy, and endothelial damage.Fibrinolysis in trauma: a review.Fibrinolysis shutdown is associated with a fivefold increase in mortality in trauma patients lacking hypersensitivity to tissue plasminogen activator.Measuring fibrinolysis: from research to routine diagnostic assays.Targeting resuscitation to normalization of coagulating status: Hyper and hypocoagulability after severe injury are both associated with increased mortality.The hyperfibrinolytic phenotype is the most lethal and resource intense presentation of fibrinolysis in massive transfusion patients.Tissue injury suppresses fibrinolysis after hemorrhagic shock in nonhuman primates (rhesus macaque).Thromboelastometry in trauma care: a place in the 2018 Belgian health care system?Goal-directed Hemostatic Resuscitation for Trauma Induced Coagulopathy : Maintaining Homeostasis.Is Coagulopathy an Appropriate Therapeutic Target During Critical Illness Such as Trauma or Sepsis?Harmful or Physiologic: Diagnosing Fibrinolysis Shutdown in a Trauma Cohort With Rotational Thromboelastometry.An increased tendency in fibrinogen activity and its association with a hypo-fibrinolytic state in early stages after injury in patients without acute traumatic coagulopathy (ATC).Mortality after red blood cell transfusions from previously pregnant donors: complexities in the interpretation of large data.Guiding Management in Severe Trauma: Reviewing Factors Predicting Outcome in Vastly Injured Patients.New Advances in the Care of the Hemorrhaging Patient

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Acute Fibrinolysis Shutdown after Injury Occurs Frequently and Increases Mortality: A Multicenter Evaluation of 2,540 Severely Injured Patients.

http://www.wikidata.org/entity/Q36807586

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J.JAMCOLLSURG.2016.01.006

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Journal of the American College of Surgeons

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Acute Fibrinolysis Shutdown af ...... 540 Severely Injured Patients.

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Angela Sauaia

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Bryan A Cotton

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Eduardo Gonzalez

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Ernest E Moore

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Hunter B Moore

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P2860

Plasminogen activator inhibitor (PAI-1) in plasma and platelets.

Early release of high mobility group box nuclear protein 1 after severe trauma in humans: role of injury severity and tissue hypoperfusion.

Fibrinolysis greater than 3% is the critical value for initiation of antifibrinolytic therapy.

A principal component analysis of postinjury viscoelastic assays: clotting factor depletion versus fibrinolysis

Seven haemostatic gene polymorphisms in coronary disease: meta-analysis of 66,155 cases and 91,307 controls.

Hyperfibrinolysis, physiologic fibrinolysis, and fibrinolysis shutdown: the spectrum of postinjury fibrinolysis and relevance to antifibrinolytic therapy

Transfusion of plasma, platelets, and red blood cells in a 1:1:1 vs a 1:1:2 ratio and mortality in patients with severe trauma: the PROPPR randomized clinical trial.

Plasma is the physiologic buffer of tissue plasminogen activator-mediated fibrinolysis: rationale for plasma-first resuscitation after life-threatening hemorrhage.

Early hemostatic responses to trauma identified with hierarchical clustering analysis.

Polymorphisms on PAI-1 and ACE genes in association with fibrinolytic bleeding after on-pump cardiac surgery.

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