The hyperfibrinolytic phenotype is the most lethal and resource intense presentation of fibrinolysis in massive transfusion patients.

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The hyperfibrinolytic phenotype is the most lethal and resource intense presentation of fibrinolysis in massive transfusion patients.

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2017 nî lūn-bûn

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2017年の論文

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年学术文章

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The hyperfibrinolytic phenotyp ...... massive transfusion patients.

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The hyperfibrinolytic phenotyp ...... massive transfusion patients.

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label

The hyperfibrinolytic phenotyp ...... massive transfusion patients.

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The hyperfibrinolytic phenotyp ...... massive transfusion patients.

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The hyperfibrinolytic phenotyp ...... massive transfusion patients.

@en

The hyperfibrinolytic phenotyp ...... massive transfusion patients.

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P1476

The hyperfibrinolytic phenotyp ...... massive transfusion patients.

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P2093

Bryan A Cotton

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Charles E Wade

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Eileen Bulger

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Erin E Fox

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John R Taylor

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Karen Brasel

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Kenji Inaba

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Martin A Schreiber

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PROPPR Study Group

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Sandro Rizoli

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P2860

Acute renal failure - definition, outcome measures, animal models, fluid therapy and information technology needs: the Second International Consensus Conference of the Acute Dialysis Quality Initiative (ADQI) Group

Activated thrombin-activatable fibrinolysis inhibitor (TAFIa) levels are decreased in patients with trauma-induced coagulopathy.

Fibrinolysis greater than 3% is the critical value for initiation of antifibrinolytic therapy.

The prospective, observational, multicenter, major trauma transfusion (PROMMTT) study: comparative effectiveness of a time-varying treatment with competing risks

Coagulation abnormalities in the trauma patient: the role of point-of-care thromboelastography.

Hyperfibrinolysis, physiologic fibrinolysis, and fibrinolysis shutdown: the spectrum of postinjury fibrinolysis and relevance to antifibrinolytic therapy

Transfusion of plasma, platelets, and red blood cells in a 1:1:1 vs a 1:1:2 ratio and mortality in patients with severe trauma: the PROPPR randomized clinical trial.

Overwhelming tPA release, not PAI-1 degradation, is responsible for hyperfibrinolysis in severely injured trauma patients.

Acute Fibrinolysis Shutdown after Injury Occurs Frequently and Increases Mortality: A Multicenter Evaluation of 2,540 Severely Injured Patients.

The ratio of blood products transfused affects mortality in patients receiving massive transfusions at a combat support hospital.

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25-30

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scholarly article

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10.1097/TA.0000000000001699

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P478

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John B Holcomb

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2017-09-13T00:00:00Z

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28914713

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5739990

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