Neuropathology and treatment of Alzheimer disease: did we lose the forest for the trees? - Wikidata - wikimedia - TriplyDB

Neuropathology and treatment of Alzheimer disease: did we lose the forest for the trees?

Neuropathology and treatment of Alzheimer disease: did we lose the forest for the trees?

http://www.wikidata.org/entity/Q36817040

about

BAFF controls neural cell survival through BAFF receptor.Alzheimer's disease is not "brain aging": neuropathological, genetic, and epidemiological human studies.Deferiprone reduces amyloid-β and tau phosphorylation levels but not reactive oxygen species generation in hippocampus of rabbits fed a cholesterol-enriched diet.Novel strategies for Alzheimer's disease treatment.Alzheimer disease pathology as a host response A Comparison between Extract Products of Magnolia officinalis on Memory Impairment and Amyloidogenesis in a Transgenic Mouse Model of Alzheimer's Disease.Phosphorylated tau: toxic, protective, or none of the above Does the term 'trophic' actually mean anti-amyloidogenic? The case of NGF.Huprines for Alzheimer's disease drug development.Pathology's new role: defining disease process and protective responses.Amyloid Beta and tau proteins as therapeutic targets for Alzheimer's disease treatment: rethinking the current strategy.Still debating a cause and diagnostic criteria for Alzheimer's disease.The effects of valsartan on cognitive deficits induced by aluminum trichloride and d-galactose in mice.

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Neuropathology and treatment of Alzheimer disease: did we lose the forest for the trees?

http://www.wikidata.org/entity/Q36817040

description

2007 nî lūn-bûn

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2007年の論文

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2007年論文

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2007年論文

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2007年論文

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2007年論文

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2007年論文

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2007年论文

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2007年论文

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2007年论文

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name

Neuropathology and treatment of Alzheimer disease: did we lose the forest for the trees?

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Neuropathology and treatment of Alzheimer disease: did we lose the forest for the trees?

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Neuropathology and treatment of Alzheimer disease: did we lose the forest for the trees?

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Neuropathology and treatment of Alzheimer disease: did we lose the forest for the trees?

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Neuropathology and treatment of Alzheimer disease: did we lose the forest for the trees?

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Neuropathology and treatment of Alzheimer disease: did we lose the forest for the trees?

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Expert Review of Neurotherapeutics: a key contribution to decision making in the treatment of neurologic and neuropsychiatric disorders

P1476

Neuropathology and treatment of Alzheimer disease: did we lose the forest for the trees?

@en

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Hyoung-gon Lee

http://www.w3.org/2001/XMLSchema#string

Rudy J Castellani

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P2860

Alzheimer Disease in the US Population

Amyloid beta binds trimers as well as monomers of the 75-kDa neurotrophin receptor and activates receptor signaling

Amyloid plaque core protein in Alzheimer disease and Down syndrome

Neuropathological stageing of Alzheimer-related changes

The amyloid hypothesis of Alzheimer's disease: progress and problems on the road to therapeutics

Regulation of cell survival by secreted proneurotrophins.

Reduced prevalence of AD in users of NSAIDs and H2 receptor antagonists: the Cache County study

Inflammation and Alzheimer's disease

Glutamate uptake

Deficiency of presenilin-1 inhibits the normal cleavage of amyloid precursor protein

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473-485

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scholarly article

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10.1586/14737175.7.5.473

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5

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7

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Paula I. Moreira

Mark Anthony Smith

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2007-05-01T00:00:00Z

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17492899

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Alzheimer's disease