An element in the bovine papillomavirus late 3' untranslated region reduces polyadenylated cytoplasmic RNA levels.
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Papillomavirus genome structure, expression, and post-transcriptional regulationA 57-nucleotide upstream early polyadenylation element in human papillomavirus type 16 interacts with hFip1, CstF-64, hnRNP C1/C2, and polypyrimidine tract binding protein.3' end mRNA processing: molecular mechanisms and implications for health and diseaseThe reciprocal regulation between splicing and 3'-end processingGeneralized substitution of isoencoding codons shortens the duration of papillomavirus L1 protein expression in transiently gene-transfected keratinocytes due to cell differentiationMultiparameter RNA and codon optimization: a standardized tool to assess and enhance autologous mammalian gene expressionA downstream polyadenylation element in human papillomavirus type 16 L2 encodes multiple GGG motifs and interacts with hnRNP H.High guanine and cytosine content increases mRNA levels in mammalian cellsWhere do introns come from?Regulation of human papillomavirus type 31 polyadenylation during the differentiation-dependent life cyclePapillomavirus capsid protein expression level depends on the match between codon usage and tRNA availability.Early polyadenylation signals of human papillomavirus type 31 negatively regulate capsid gene expression.Analysis of transcripts from predicted open reading frames of Musca domestica salivary gland hypertrophy virus.A splicing enhancer in the E4 coding region of human papillomavirus type 16 is required for early mRNA splicing and polyadenylation as well as inhibition of premature late gene expression.Gene codon composition determines differentiation-dependent expression of a viral capsid gene in keratinocytes in vitro and in vivo.Position-dependent inhibition of the cleavage step of pre-mRNA 3'-end processing by U1 snRNPSequence of the polypyrimidine tract of the 3'-terminal 3' splicing signal can affect intron-dependent pre-mRNA processing in vivo.Integration of human papillomavirus type 16 DNA into the human genome leads to increased stability of E6 and E7 mRNAs: implications for cervical carcinogenesis.Integrated functional and bioinformatics approach for the identification and experimental verification of RNA signals: application to HIV-1 INS.The Rev protein of human immunodeficiency virus type 1 counteracts the effect of an AU-rich negative element in the human papillomavirus type 1 late 3' untranslated region.Identification of nuclear and cytoplasmic proteins that interact specifically with an AU-rich, cis-acting inhibitory sequence in the 3' untranslated region of human papillomavirus type 1 late mRNAs.A cellular 65-kDa protein recognizes the negative regulatory element of human papillomavirus late mRNAThe 3' untranslated region of the B19 parvovirus capsid protein mRNAs inhibits its own mRNA translation in nonpermissive cells.The human immunodeficiency virus type 1 Rev protein and the Rev-responsive element counteract the effect of an inhibitory 5' splice site in a 3' untranslated regionHierarchy of polyadenylation site usage by bovine papillomavirus in transformed mouse cells.Sequences homologous to 5' splice sites are required for the inhibitory activity of papillomavirus late 3' untranslated regions.Late promoter of human papillomavirus type 8 and its regulation.Regulation of bovine papillomavirus type 1 gene expression by RNA processingHuman papillomavirus type 16 late gene expression is regulated by cellular RNA processing factors in response to epithelial differentiationSplicing and Polyadenylation of Human Papillomavirus Type 16 mRNAs.The inhibitory activity of the AU-rich RNA element in the human papillomavirus type 1 late 3' untranslated region correlates with its affinity for the elav-like HuR protein.Analysis of inhibitory action of modified U1 snRNAs on target gene expression: discrimination of two RNA targets differing by a 1 bp mismatch.Specific inactivation of inhibitory sequences in the 5' end of the human papillomavirus type 16 L1 open reading frame results in production of high levels of L1 protein in human epithelial cells.The human papillomavirus type 31 late 3' untranslated region contains a complex bipartite negative regulatory elementActivity of the human papillomavirus type 16 late negative regulatory element is partly due to four weak consensus 5' splice sites that bind a U1 snRNP-like complexHuman papillomavirus type 31b late gene expression is regulated through protein kinase C-mediated changes in RNA processing.Mutational inactivation of two distinct negative RNA elements in the human papillomavirus type 16 L2 coding region induces production of high levels of L2 in human cells.Polypyrimidine tract binding protein induces human papillomavirus type 16 late gene expression by interfering with splicing inhibitory elements at the major late 5' splice site, SD3632Transcriptional silencer of the human papillomavirus type 8 late promoter interacts alternatively with the viral trans activator E2 or with a cellular factor.The alternative splicing factor hnRNP A1 is up-regulated during virus-infected epithelial cell differentiation and binds the human papillomavirus type 16 late regulatory element.
P2860
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P2860
An element in the bovine papillomavirus late 3' untranslated region reduces polyadenylated cytoplasmic RNA levels.
description
1991 nî lūn-bûn
@nan
1991年の論文
@ja
1991年論文
@yue
1991年論文
@zh-hant
1991年論文
@zh-hk
1991年論文
@zh-mo
1991年論文
@zh-tw
1991年论文
@wuu
1991年论文
@zh
1991年论文
@zh-cn
name
An element in the bovine papil ...... ylated cytoplasmic RNA levels.
@en
type
label
An element in the bovine papil ...... ylated cytoplasmic RNA levels.
@en
prefLabel
An element in the bovine papil ...... ylated cytoplasmic RNA levels.
@en
P2860
P1433
P1476
An element in the bovine papil ...... ylated cytoplasmic RNA levels.
@en
P2093
P2860
P304
P407
P577
1991-11-01T00:00:00Z