The cardiomyocyte molecular clock, regulation of Scn5a, and arrhythmia susceptibility.
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Cardiomyocyte Circadian Oscillations Are Cell-Autonomous, Amplified by β-Adrenergic Signaling, and Synchronized in Cardiac Ventricle TissueComparison of circadian, weekly, and seasonal variations of electrical storms and single events of ventricular fibrillation in patients with Brugada syndrome.Cardiomyocyte-specific BMAL1 plays critical roles in metabolism, signaling, and maintenance of contractile function of the heart.Influence of the cardiomyocyte circadian clock on cardiac physiology and pathophysiology.Light phase-restricted feeding slows basal heart rate to exaggerate the type-3 long QT syndrome phenotype in mice.Circadian clock control of Nox4 and reactive oxygen species in the vasculature.Smooth-muscle BMAL1 participates in blood pressure circadian rhythm regulation.The cardiomyocyte molecular clock regulates the circadian expression of Kcnh2 and contributes to ventricular repolarization.Consequences of Circadian Disruption on Neurologic Health.The end effector of circadian heart rate variation: the sinoatrial node pacemaker cell.Cardiac sodium channel mutations: why so many phenotypes?Cardiac sodium channelopathy associated with SCN5A mutations: electrophysiological, molecular and genetic aspects.Recent advances in circadian rhythms in cardiovascular system.The role of clock genes and circadian rhythm in the development of cardiovascular diseases.Genetic disruption of the cardiomyocyte circadian clock differentially influences insulin-mediated processes in the heart.Bone Morphogenetic Protein 9 and 13 Induce C3H10T1/2 Cell Differentiation to Cardiomyocyte-Like Cells In Vitro.Achilles is a circadian clock-controlled gene that regulates immune function in Drosophila.[Sudden cardiac death : Epidemiology, pathophysiology and risk stratification].Commentary on: "Levofloxacin-Induced QTc Prolongation Depends on the Time of Drug Administration".Levofloxacin-Induced QTc Prolongation Depends on the Time of Drug Administration.Misalignment with the external light environment drives metabolic and cardiac dysfunctionMinding the gaps that link intrinsic circadian clock within the heart to its intrinsic ultradian pacemaker clocks. Focus on "The cardiomyocyte molecular clock, regulation of Scn5a, and arrhythmia susceptibility".CLOCK-BMAL1 regulate the cardiac L-type calcium channel subunit CACNA1C through PI3K-Akt signaling pathway.Coordination of cardiac rhythmic output and circadian metabolic regulation in the heart.Intrinsic muscle clock is necessary for musculoskeletal health.Altered Circadian Timing System-Mediated Non-Dipping Pattern of Blood Pressure and Associated Cardiovascular Disorders in Metabolic and Kidney Diseases.Circadian clock and the onset of cardiovascular events.Reversible lysine acetylation: Another layer of post-translational regulation of the cardiac sodium channel.
P2860
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P2860
The cardiomyocyte molecular clock, regulation of Scn5a, and arrhythmia susceptibility.
description
2013 nî lūn-bûn
@nan
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
2013年论文
@zh
2013年论文
@zh-cn
name
The cardiomyocyte molecular clock, regulation of Scn5a, and arrhythmia susceptibility.
@en
type
label
The cardiomyocyte molecular clock, regulation of Scn5a, and arrhythmia susceptibility.
@en
prefLabel
The cardiomyocyte molecular clock, regulation of Scn5a, and arrhythmia susceptibility.
@en
P2093
P2860
P1476
The cardiomyocyte molecular clock, regulation of Scn5a, and arrhythmia susceptibility.
@en
P2093
Abhijit Patwardhan
Daniel C Bartos
Elizabeth A Schroder
Han-Zhong Feng
Jian-Ping Jin
Karyn A Esser
Mellani Lefta
Xiping Zhang
Yihua Zhao
P2860
P304
P356
10.1152/AJPCELL.00383.2012
P577
2013-01-30T00:00:00Z