Matrices of physiologic stiffness potently inactivate idiopathic pulmonary fibrosis fibroblasts.
about
Molecular classification of idiopathic pulmonary fibrosis: personalized medicine, genetics and biomarkersThe extracellular matrix - the under-recognized element in lung disease?Environmental particulate (PM2.5) augments stiffness-induced alveolar epithelial cell mechanoactivation of transforming growth factor betaFibroblast viability and phenotypic changes within glycated stiffened three-dimensional collagen matrices.Prostaglandin E2 inhibits α-smooth muscle actin transcription during myofibroblast differentiation via distinct mechanisms of modulation of serum response factor and myocardin-related transcription factor-AFollowing the path of CCL2 from prostaglandins to periostin in lung fibrosisPathogenesis of idiopathic pulmonary fibrosisHydrogel scaffolds as in vitro models to study fibroblast activation in wound healing and disease.Mechanosignaling through YAP and TAZ drives fibroblast activation and fibrosisThe 2-pore domain potassium channel TREK-1 regulates stretch-induced detachment of alveolar epithelial cellsMatrix, mesenchyme, and mechanotransductionLung extracellular matrix and fibroblast function.Second harmonic generation microscopy reveals altered collagen microstructure in usual interstitial pneumonia versus healthy lung.Why do patients get idiopathic pulmonary fibrosis? Current concepts in the pathogenesis of pulmonary fibrosisPathologic Regulation of Collagen I by an Aberrant Protein Phosphatase 2A/Histone Deacetylase C4/MicroRNA-29 Signal Axis in Idiopathic Pulmonary Fibrosis FibroblastsTAZ activation drives fibroblast spheroid growth, expression of profibrotic paracrine signals, and context-dependent ECM gene expression.Tissue mechanics and fibrosis.Fibroblasts and the ground they walk onMatrix regulation of idiopathic pulmonary fibrosis: the role of enzymes.Newer developments in idiopathic pulmonary fibrosis in the era of anti-fibrotic medications.TAZ contributes to pulmonary fibrosis by activating profibrotic functions of lung fibroblasts.Exosomes in immunoregulation of chronic lung diseases.Plasma Surfactant Protein-D, Matrix Metalloproteinase-7, and Osteopontin Index Distinguishes Idiopathic Pulmonary Fibrosis from Other Idiopathic Interstitial Pneumonias.Regulation of tissue fibrosis by the biomechanical environment.Matrix stiffness regulates migration of human lung fibroblasts.The Processes and Mechanisms of Cardiac and Pulmonary Fibrosis.Extracellular matrix type modulates cell migration on mechanical gradients.Lysyl oxidases regulate fibrillar collagen remodelling in idiopathic pulmonary fibrosis.Mechanosensing and fibrosis.Matrix biomechanics and dynamics in pulmonary fibrosis.Stem Cell Differentiation is Regulated by Extracellular Matrix Mechanics.Inhibition of PHGDH Attenuates Bleomycin-induced Pulmonary Fibrosis.Stretch-induced Activation of Transforming Growth Factor-β1 in Pulmonary Fibrosis.Extracellular matrix in lung development, homeostasis and disease.Idiopathic pulmonary fibrosis: Epithelial-mesenchymal interactions and emerging therapeutic targets.TGF beta inhibits HGF, FGF7, and FGF10 expression in normal and IPF lung fibroblasts
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P2860
Matrices of physiologic stiffness potently inactivate idiopathic pulmonary fibrosis fibroblasts.
description
2013 nî lūn-bûn
@nan
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
2013年论文
@zh
2013年论文
@zh-cn
name
Matrices of physiologic stiffn ...... ulmonary fibrosis fibroblasts.
@en
type
label
Matrices of physiologic stiffn ...... ulmonary fibrosis fibroblasts.
@en
prefLabel
Matrices of physiologic stiffn ...... ulmonary fibrosis fibroblasts.
@en
P2093
P2860
P356
P1476
Matrices of physiologic stiffn ...... ulmonary fibrosis fibroblasts.
@en
P2093
Aleksandar Marinković
Daniel J Tschumperlin
P2860
P304
P356
10.1165/RCMB.2012-0335OC
P577
2013-04-01T00:00:00Z