Nitric oxide-dependent stimulation of endothelial cell proliferation by sustained high flow.
about
High shear stress induces atherosclerotic vulnerable plaque formation through angiogenesisBiomechanical regulation of vascular smooth muscle cell functions: from in vitro to in vivo understandingTwo distinct phases of calcium signalling under flow.RhoA and membrane fluidity mediates the spatially polarized Src/FAK activation in response to shear stress.Endothelial cells isolated from caveolin-2 knockout mice display higher proliferation rate and cell cycle progression relative to their wild-type counterpartsEndothelial nitric oxide synthase and superoxide mediate hemodynamic initiation of intracranial aneurysms.Manipulating the microvasculature and its microenvironment.Acute and chronic exposure to shear stress have opposite effects on endothelial permeability to macromoleculesShear stress and the endothelial transport barrier.A microfluidic shear device that accommodates parallel high and low stress zones within the same culturing chamber.Shear stress inhibits apoptosis of ischemic brain microvascular endothelial cellsNitric oxide and TNFα are critical regulators of reversible lymph node vascular remodeling and adaptive immune response.A comparative review of the hemodynamics and pathogenesis of cerebral and abdominal aortic aneurysms: lessons to learn from each other.Endothelial cells express a unique transcriptional profile under very high wall shear stress known to induce expansive arterial remodelingParallel-plate flow chamber and continuous flow circuit to evaluate endothelial progenitor cells under laminar flow shear stress.High wall shear stress and spatial gradients in vascular pathology: a reviewImpaired A2A adenosine receptor/nitric oxide/VEGF signaling pathway in fetal endothelium during late- and early-onset preeclampsia.Differential gene expression by endothelial cells under positive and negative streamwise gradients of high wall shear stress.Molecular alterations associated with aneurysmal remodeling are localized in the high hemodynamic stress region of a created carotid bifurcation.Microvascular endothelial cells migrate upstream and align against the shear stress field created by impinging flow.Effects of wall distensibility in hemodynamic simulations of an arteriovenous fistulaHow do we prevent the vulnerable atherosclerotic plaque from rupturing? Insights from in vivo assessments of plaque, vascular remodeling, and local endothelial shear stress.Intercorrelations of morphology with hemodynamics in intracranial aneurysms in computational fluid dynamics.A robust approach for exploring hemodynamics and thrombus growth associations in abdominal aortic aneurysms.High fluid shear stress and spatial shear stress gradients affect endothelial proliferation, survival, and alignmentPinosylvin induces cell survival, migration and anti-adhesiveness of endothelial cells via nitric oxide production.
P2860
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P2860
Nitric oxide-dependent stimulation of endothelial cell proliferation by sustained high flow.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
2008年论文
@zh
2008年论文
@zh-cn
name
Nitric oxide-dependent stimula ...... ration by sustained high flow.
@en
type
label
Nitric oxide-dependent stimula ...... ration by sustained high flow.
@en
prefLabel
Nitric oxide-dependent stimula ...... ration by sustained high flow.
@en
P2093
P2860
P1476
Nitric oxide-dependent stimula ...... eration by sustained high flow
@en
P2093
Eleni Metaxa
Michael P Szymanski
Rocco A Paluch
Shashikanth R Kaluvala
P2860
P304
P356
10.1152/AJPHEART.01156.2007
P50
P577
2008-06-13T00:00:00Z