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Molecular mechanism of endothelial growth arrest by laminar shear stress.

Molecular mechanism of endothelial growth arrest by laminar shear stress.

http://www.wikidata.org/entity/Q35205740

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Vascular Remodelling and Mesenchymal Transition in Systemic Sclerosis Novel mechanisms of endothelial mechanotransduction Homeodomain interacting protein kinase 2 activation compromises endothelial cell response to laminar flow: protective role of p21(waf1,cip1,sdi1)Pattern formation of vascular smooth muscle cells subject to nonuniform fluid shear stress: role of PDGF-beta receptor and Src Fluid forces control endothelial sprouting.Mechanical strain modulates age-related changes in the proliferation and differentiation of mouse adipose-derived stromal cells.Role of microRNA-23b in flow-regulation of Rb phosphorylation and endothelial cell growth.MicroRNA-19a mediates the suppressive effect of laminar flow on cyclin D1 expression in human umbilical vein endothelial cells.Acute and chronic exposure to shear stress have opposite effects on endothelial permeability to macromolecules Prostaglandin (PG)D(2) and 15-deoxy-Delta(12,14)-PGJ(2), but not PGE(2), mediate shear-induced chondrocyte apoptosis via protein kinase A-dependent regulation of polo-like kinases Shear stress, SIRT1, and vascular homeostasis.MicroRNA-23b regulates cyclin-dependent kinase-activating kinase complex through cyclin H repression to modulate endothelial transcription and growth under flow A biohybrid artificial lung prototype with active mixing of endothelialized microporous hollow fibers.Effects of disturbed flow on vascular endothelium: pathophysiological basis and clinical perspectives Discovery of novel mechanosensitive genes in vivo using mouse carotid artery endothelium exposed to disturbed flow.Endothelial cellular response to altered shear stress.Fluid Mechanics, Arterial Disease, and Gene Expression.Dotted collar placed around carotid artery induces asymmetric neointimal lesion formation in rabbits without intravascular manipulations.Acute venous occlusion enhances matrix metalloprotease activity: Implications on endothelial dysfunction Microfluidic 3D models of cancer.p53 activates ICAM-1 (CD54) expression in an NF-kappaB-independent manner PKCζ mediates disturbed flow-induced endothelial apoptosis via p53 SUMOylation Disturbed flow: p53 SUMOylation in the turnover of endothelial cells Biomechanical activation of vascular endothelium as a determinant of its functional phenotype.Roles of cell confluency and fluid shear in 3-dimensional intracellular forces in endothelial cells KLF2 Is a novel transcriptional regulator of endothelial proinflammatory activation.Fluid shear stress modulates endothelial cell invasion into three-dimensional collagen matrices Cyclic strain-mediated matrix metalloproteinase regulation within the vascular endothelium: a force to be reckoned with.Coexisting proinflammatory and antioxidative endothelial transcription profiles in a disturbed flow region of the adult porcine aorta.Disturbed-flow-mediated vascular reactive oxygen species induce endothelial dysfunction.Endothelial cell respiration is affected by the oxygen tension during shear exposure: role of mitochondrial peroxynitrite.Nitric oxide-dependent stimulation of endothelial cell proliferation by sustained high flow.Effects of disturbed flow on endothelial cells.Regulation of shear-induced nuclear translocation of the Nrf2 transcription factor in endothelial cells.The efficacy of complete surgical excision of keloid and piercing sinus tract on earlobe keloid.Defective fluid shear stress mechanotransduction mediates hereditary hemorrhagic telangiectasia.Microvascular endothelial cells migrate upstream and align against the shear stress field created by impinging flow.ECM-based materials in cardiovascular applications: Inherent healing potential and augmentation of native regenerative processes.Laminar flow downregulates Notch activity to promote lymphatic sprouting.Shear stress and plaque development.

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P2860

Molecular mechanism of endothelial growth arrest by laminar shear stress.

http://www.wikidata.org/entity/Q35205740

description

2000 nî lūn-bûn

@nan

2000 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2000 թվականի օգոստոսին հրատարակված գիտական հոդված

@hy

2000年の論文

@ja

2000年論文

@yue

2000年論文

@zh-hant

2000年論文

@zh-hk

2000年論文

@zh-mo

2000年論文

@zh-tw

2000年论文

@wuu

name

Molecular mechanism of endothelial growth arrest by laminar shear stress.

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Molecular mechanism of endothelial growth arrest by laminar shear stress.

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type

label

Molecular mechanism of endothelial growth arrest by laminar shear stress.

@ast

Molecular mechanism of endothelial growth arrest by laminar shear stress.

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prefLabel

Molecular mechanism of endothelial growth arrest by laminar shear stress.

@ast

Molecular mechanism of endothelial growth arrest by laminar shear stress.

@en

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P1433

Proceedings of the National Academy of Sciences of the United States of America

P1476

Molecular mechanism of endothelial growth arrest by laminar shear stress.

@en

P2093

B P Chen

http://www.w3.org/2001/XMLSchema#string

J Y Shyy

http://www.w3.org/2001/XMLSchema#string

K Lin

http://www.w3.org/2001/XMLSchema#string

P P Hsu

http://www.w3.org/2001/XMLSchema#string

S Chien

http://www.w3.org/2001/XMLSchema#string

S Usami

http://www.w3.org/2001/XMLSchema#string

S Yuan

http://www.w3.org/2001/XMLSchema#string

Y S Li

http://www.w3.org/2001/XMLSchema#string

P2860

GADD45 induction of a G2/M cell cycle checkpoint

Interaction of the p53-regulated protein Gadd45 with proliferating cell nuclear antigen

p53, the cellular gatekeeper for growth and division

Upregulation of superoxide dismutase and nitric oxide synthase mediates the apoptosis-suppressive effects of shear stress on endothelial cells

Tumor suppressor p53 can participate in transcriptional induction of the GADD45 promoter in the absence of direct DNA binding

JNK targets p53 ubiquitination and degradation in nonstressed cells.

Turbulent fluid shear stress induces vascular endothelial cell turnover in vitro

Conformation-dependent phosphorylation of p53.

DNA-damage inducible genes.

The Ras-JNK pathway is involved in shear-induced gene expression

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9385-9389

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scholarly article

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10.1073/PNAS.170282597

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17

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97

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24448412

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10920209

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16873

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